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Publikováno v:
J Vis Exp
Single-cell methodologies have revolutionized the analysis of the transcriptomes of specific cell types. However, they often require species-specific genetic "toolkits," such as promoters driving tissue-specific expression of fluorescent proteins. Fu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::262c87662a8e7606de245ac6c2a2e817
https://europepmc.org/articles/PMC9976942/
https://europepmc.org/articles/PMC9976942/
Autor:
A Adhemir Valera, S Alberto Manchego, C Nieves Sandoval, R Antonio Herrera, H Shirley Palacios, S Gina Castro
Publikováno v:
Revista de Investigaciones Veterinarias del Perú. 32:e20016
El objetivo del presente estudio fue la caracterización molecular del segmento 4 del virus de la tilapia de lago (TiLV) detectado en tilapias de cultivo de los departamentos de Piura (Costa) y San Martín (Selva) en un brote ocurrido en 2017-2018. E
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7394c40fd53687c812e8edbaf8e0bfcb
https://doi.org/10.15381/rivep.v32i2.20016
https://doi.org/10.15381/rivep.v32i2.20016
Autor:
Edward Vuong, Jintao Luo, David H. A. Fitch, Karin Kiontke, R. Antonio Herrera, Douglas S. Portman, Erich M. Schwarz
Publikováno v:
Dev Cell
Summary Biological roles for most long non-coding RNAs (lncRNAs) remain mysterious. Here, using forward genetics, we identify lep-5, a lncRNA acting in the C. elegans heterochronic (developmental timing) pathway. Loss of lep-5 delays hypodermal matur
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::67bbd5718852843e15893315ccaa226c
https://doi.org/10.1016/j.devcel.2019.03.003
https://doi.org/10.1016/j.devcel.2019.03.003
Publikováno v:
Viral Immunology. 21:141-152
Interferon-gamma (IFN-gamma) has potent antiviral activity in neurons which is affected by the production of nitric oxide (NO). This study examines the interactions between cannabinoid receptor-1 (CB(1)), IFNgamma-induced pathways, and inhibition of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6275d86fb10430dc6a8b308ff9958ffd
https://doi.org/10.1089/vim.2007.0109
https://doi.org/10.1089/vim.2007.0109
Publikováno v:
Development (Cambridge, England). 143(5)
The heterochronic genes lin-28, let-7 and lin-41 regulate fundamental developmental transitions in animals, such as stemness versus differentiation and juvenile versus adult states. We identify a new heterochronic gene, lep-2, in Caenorhabditis elega
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::acdc4a26a45e7abaf66642112a7781f2
https://pubmed.ncbi.nlm.nih.gov/26811380
https://pubmed.ncbi.nlm.nih.gov/26811380
Autor:
Park, Reyna1, Herrera, R. Antonio1
Publikováno v:
Southeastern Biology. Jan-Dec2024, Vol. 71 Issue 1-4, p116-117. 2p.
Publikováno v:
Southeastern Biology. Jan-Dec2024, Vol. 71 Issue 1-4, p115-116. 2p.
Autor:
Yajun Tang, R. Antonio Herrera, Xiangyang Liu, Isabelle Richard, Jer R. Kuszak, Rebecca K. Zoltoski, L.A. Novak, Nalin M. Kumar
Publikováno v:
Investigative ophthalmologyvisual science. 48(6)
Gap junctions consist of two hexameric structures of connexin (Cx) molecules called connexons, which are located in the plasma membranes of two neighboring cells. So far, more than 20 connexin genes have been identified in vertebrates.1,2 Three conne
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ada7c269aec3478260d2893ca14cfc64
https://pubmed.ncbi.nlm.nih.gov/17525200
https://pubmed.ncbi.nlm.nih.gov/17525200
Publikováno v:
Otolaryngology–Head and Neck Surgery. 131
Problem: Mutations in the gap junction beta-2 (GJB2) gene, which encodes the connexin 26 protein, account for 50% of all the patients with autosomal recessive nonsyndromic hearing loss. Connexins are proteins that oligomerize to form a gap junction c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::17d7b23c3cf2b4a227ffce51cacf060b
https://doi.org/10.1016/j.otohns.2004.06.138
https://doi.org/10.1016/j.otohns.2004.06.138