Zobrazeno 1 - 10
of 37
pro vyhledávání: '"R V Thakker"'
Autor:
K E Lines, M Stevenson, R Mihai, I V Grigorieva, O A Shariq, K U Gaynor, J Jeyabalan, M Javid, R V Thakker
Publikováno v:
Endocrine Oncology, Vol 1, Iss 1, Pp 23-32 (2023)
Hypoxia, a primary stimulus for angiogenesis, is important for tumour proliferation and survival. The effects of hypoxia on parathyroid tumour cells, which may also be important for parathyroid autotransplantation in patients, are, however, unknown.
Externí odkaz:
https://doaj.org/article/761cd33860c04fa7b2afbaa8733011ab
Publikováno v:
Endocrine Connections, Vol 6, Iss 4, Pp 232-242 (2017)
Multiple endocrine neoplasia type 1 (MEN1) is an autosomal dominant disorder characterised by occurrence of parathyroid tumours and neuroendocrine tumours (NETs) of the pancreatic islets and anterior pituitary. The MEN1 gene, encoding menin, is a tum
Externí odkaz:
https://doaj.org/article/f4f4891489d34467b28c78b9be8c5bb5
Autor:
R V Thakker
Publikováno v:
Indian Journal of Endocrinology and Metabolism, Vol 16, Iss 8, Pp 213-216 (2012)
The calcium-sensing receptor (CaSR) is a 1,078 amino acid G protein-coupled receptor (GPCR), which is predominantly expressed in the parathyroids and kidney. The CaSR allows regulation of parathyroid hormone (PTH) secretion and renal tubular calcium
Externí odkaz:
https://doaj.org/article/fd88634c7cf8480ea6bfc5e8e8d5f481
Autor:
R V Thakker
Publikováno v:
Indian Journal of Endocrinology and Metabolism, Vol 16, Iss 8, Pp 272-274 (2012)
Multiple endocrine neoplasia type 1 (MEN1) is characterized by the occurrence of parathyroid, pancreatic islet and anterior pituitary tumors. Some patients may also develop carcinoid tumors, adrenocortical tumors, facial angiofibromas, collagenomas,
Externí odkaz:
https://doaj.org/article/1aa95664f0324288b2a9d089397f97b9
Autor:
Katherine U. Gaynor, Mahsa Javid, Radu Mihai, Kate E Lines, Jeshmi Jeyabalan, O A Shariq, Mark Stevenson, R. V. Thakker, Irina V. Grigorieva
Publikováno v:
Endocrine Oncology. 1:23-32
Hypoxia, a primary stimulus for angiogenesis, is important for tumour proliferation and survival. The effects of hypoxia on parathyroid tumour cells, which may also be important for parathyroid autotransplantation in patients, are, however, unknown.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::42000f1807d6330ead2061ec656b6f38
https://doi.org/10.1530/eo-21-0014
https://doi.org/10.1530/eo-21-0014
Autor:
C. Legendre, D. Cohen, Y. Delmas, T. Feldkamp, D. Fouque, R. Furman, O. Gaber, L. Greenbaum, T. Goodship, H. Haller, M. Herthelius, M. Hourmant, C. Licht, B. Moulin, N. Sheerin, A. Trivelli, C. L. Bedrosian, C. Loirat, S. Babu, T. Jungraithmayr, Y. Lebranchu, M. Riedl, A. O. Gaber, C. Bedrosian, P. Muus, K. Douglas, G. Remuzzi, A. Kourouklaris, K. Ioannou, I. Athanasiou, K. Demetriou, A. Panagidou, M. Zavros, N. Y. Rodriguez C, M. Blasco, C. Arcal, L. F. Quintana, S. Rodriguez de Cordoba, J. M. Campistol, N. Bachmann, T. Eisenberger, C. Decker, H. J. Bolz, C. Bergmann, F. Pesce, S. N. Cox, G. Serino, G. De Palma, F. P. Sallustio, F. Schena, M. Falchi, M. Pieri, C. Stefanou, A. Zaravinos, K. Erguler, G. Lapathitis, H. Dweep, C. Sticht, N. Anastasiadou, I. Zouvani, K. Voskarides, N. Gretz, C. C. Deltas, A. Ruiz, O. Bonny, F. Sallustio, C. Curci, S. Cox, E. Kemter, S. Sklenak, B. Aigner, R. Wanke, T. M. Kitzler, J. L. Moskowitz, S. E. Piret, K. Lhotta, A. Tashman, E. Velez, R. V. Thakker, P. Kotanko, J. Leierer, M. Rudnicki, P. Perco, C. Koppelstaetter, G. Mayer, M. J. N. Sa, S. Alves, H. Storey, F. Flinter, P. J. Willems, F. Carvalho, J. Oliveira, M. Arsali, L. Papazachariou, P. Demosthenous, A. Lazarou, M. Hadjigavriel, C. Stavrou, L. Yioukkas, C. Deltas, A. Pierides, M. Kkolou, H. R. Toka, S. Dibartolo, B. Lanske, E. M. Brown, M. R. Pollak, A. Familiari, B. Zavan, S. Sanna Cherchi, A. Fabris, R. Cristofaro, G. Gambaro, A. D'Angelo, F. Anglani, H. Toka, D. Mount, M. Pollak, G. Curhan, G. Sengoge, T. Bajari, A. Kupczok, A. von Haeseler, M. Schuster, W. Pfaller, P. Jennings, A. Weltermann, S. Blake, G. Sunder-Plassmann, A. Kerti, R. Csohany, L. Wagner, E. Javorszky, E. Maka, T. Tulassay, K. Tory, J. Kingswood, N. Nikolskaya, J. Mbundi, S. Jozwiak, E. Belousova, M. Frost, R. Kuperman, M. Bebin, B. Korf, R. Flamini, M. Kohrman, S. Sparagana, J. Wu, T. Brechenmacher, K. Stein, J. Bissler, D. Franz, B. Zonnenberg, W. Cheung, J. Wang, D. Lam, K. Budde, L. Ivanitskiy, E. Sowershaewa, T. Krasnova, L. Samokhodskaya, M. Safarikova, R. Jana, S. Jitka, L. Obeidova, M. Kohoutova, V. Tesar, H. Evrengul, P. Ertan, E. Serdaroglu, S. Yuksel, S. Mir, E. Yang n Ergon, A. Berdeli, A. Zawada, K. Rogacev, B. Rotter, P. Winter, D. Fliser, G. Heine, S. Bataille, V. Moal, Y. Berland, L. Daniel, C. Rosado, E. Bueno, P. Fraile, C. Lucas, P. Garcoa-Cosmes, J. M. Tabernero, R. Gonzalez, P. Garcia-Cosmes, M. Silska-Dittmar, K. Zaorska, A. Malke, A. Musielak, D. Ostalska-Nowicka, J. Zachwieja, V. K d r, E. Uz, A. Yigit, A. Altuntas, B. Yigit, S. Inal, M. Sezer, R. Yilmaz, B. Visciano, C. Porto, E. Acampora, R. Russo, E. Riccio, I. Capuano, G. Parenti, A. Pisani, S. Feriozzi, A. Perrin, M. West, K. Nicholls, J. Torras, M. Cybulla, M. Conti, A. Angioi, M. Floris, P. Melis, A. M. Asunis, D. Piras, A. Pani, D. Warnock, A. Guasch, C. Thomas, C. Wanner, R. Campbell, B. Vujkovac, I. Okur, G. Biberoglu, F. Ezgu, L. Tumer, A. Hasanoglu, Z. Bicik, Y. Akin, M. Mumcuoglu, T. Ecder, C. Paliouras, G. Mattas, N. Papagiannis, G. Ntetskas, F. Lamprianou, N. Karvouniaris, P. Alivanis
Publikováno v:
Nephrology Dialysis Transplantation. 29:iii339-iii350
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0f93d37001a0d21f29b49c8e5eca855d
https://doi.org/10.1093/ndt/gfu162
https://doi.org/10.1093/ndt/gfu162
Autor:
G V, Walls, M, Stevenson, K E, Lines, P J, Newey, A A C, Reed, M R, Bowl, J, Jeyabalan, B, Harding, K J, Bradley, S, Manek, J, Chen, P, Wang, B O, Williams, B T, Teh, R V, Thakker
Publikováno v:
Oncogene
The hyperparathyroidism-jaw tumour (HPT-JT) syndrome is an autosomal dominant disorder characterized by occurrence of parathyroid tumours, often atypical adenomas and carcinomas, ossifying jaw fibromas, renal tumours and uterine benign and malignant
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::1c2b1a863cb9cae7272fde1ad624affa
https://pubmed.ncbi.nlm.nih.gov/28288139
https://pubmed.ncbi.nlm.nih.gov/28288139
Autor:
Pierre J. Verroust, Anthony G.W. Norden, Erik Ilsø Christensen, R. V. Thakker, Maria João Saraiva, Christian Jacobsen, Søren K. Moestrup, Mónica Mendes Sousa, Thomas E. Willnow
The kidney is a major organ for uptake of the thyroid hormone thyroxine (T(4)) and its conversion to the active form, triiodothyronine. In the plasma, one of the T(4) carriers is transthyretin (TTR). In the present study we observed that TTR, the tra
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::345b9e7b1865b042392f8fed9ff8f72c
https://doi.org/10.1074/jbc.m002886200
https://doi.org/10.1074/jbc.m002886200
Autor:
R. V. Thakker
Publikováno v:
Epstein's Inborn Errors of Development: The Molecular Basis of Clinical Disorders of Morphogenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::13cece69ad52dc989f0f09fab89567e3
https://doi.org/10.1093/med/9780199934522.003.0131
https://doi.org/10.1093/med/9780199934522.003.0131
Publikováno v:
Molecular and Cellular Endocrinology
Pituitary neoplasias can occur as part of a complex inherited disorder, or more commonly as sporadic (non-familial) disease. Studies of the molecular and genetic mechanisms causing such pituitary tumours have identified dysregulation of >35 genes, wi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::400b3dbc66454ac00af2def6e793271e
https://pubmed.ncbi.nlm.nih.gov/26320859
https://pubmed.ncbi.nlm.nih.gov/26320859