Zobrazeno 1 - 10
of 16
pro vyhledávání: '"R T, Urbanus"'
Autor:
A R T Brand Kanters, N C Roozendaal, N M J Parr, G Pasterkamp, R T Urbanus, S J A Korporaal, Gert J de Borst
Publikováno v:
PLoS ONE, Vol 19, Iss 6, p e0304800 (2024)
BackgroundDespite Antiplatelet therapy (APT), cardiovascular patients undergoing revascularisation remain at high risk for thrombotic events. Individual response to APT varies substantially, resulting in insufficient protection from thrombotic events
Externí odkaz:
https://doaj.org/article/ab6c025d5d3b48ba959723ad87afa67a
Autor:
G-J de Borst, M. Frans, Mark Roest, Peter Paul Wisman, Martin Teraa, Jasper A. Remijn, T. C. Van Holten, P. G. De Groot, R. T. Urbanus, T.C. Leunissen, Marianne C. Verhaar
Publikováno v:
Thrombosis Research. 141:S1-S19
24th Biennial International Congress on Thrombosis – EMLTD Congress 2016 4–7 May 2016, Istanbul, Turkey
Publikováno v:
Nederlands tijdschrift voor geneeskunde. 161
Platelet aggregation inhibitors, also known as antiplatelet therapy (APT), are prescribed for the prevention of secondary cardiovascular events (CVE) after endovascular revascularization procedures.- Platelet aggregation inhibitors are not equally ef
Autor:
P. G. De Groot, C. Ağar, R. T. Urbanus, Matthias Mörgelin, Mercedes Valls Seron, J. A. Marquart, Heiko Herwald, R. H. W. M. Derksen, Joost C. M. Meijers, G. M. A. Van Os, Per Åkesson
Publikováno v:
Journal of thrombosis and haemostasis, 9(12), 2447-2456. Wiley-Blackwell
The antiphospholipid syndrome (APS) is characterized by the persistent presence of anti-β(2) -glycoprotein I (β(2) -GPI) autoantibodies. β(2) -GPI can exist in two conformations. In plasma it is a circular protein, whereas it adopts a fish-hook co
Autor:
Maarten T. T. Pennings, Guadalupe Montiel-Manzano, Silvia S. Pierangeli, R. T. Urbanus, Zurina Romay-Penabad, P. DeGroot
Publikováno v:
Journal of Thrombosis and Haemostasis. 5:P-W
Publikováno v:
International journal of laboratory hematology. 34(3)
The antiphospholipid syndrome is an autoimmune disease that manifests clinically as recurrent thrombotic complications or foetal losses and serologically with elevated levels of antiphospholipid antibodies in the plasmas of these patients. The term '
Publikováno v:
Hamostaseologie. 30(3)
The antiphospholipid syndrome (APS) is a non-inflammatory autoimmune disease characterized by the presence of antiphospholipid antibodies (aPL) in the plasma of patients with vascular thrombosis, recurrent complications of pregnancy, or both (1, 2).
Autor:
M T T, Pennings, R H W M, Derksen, M, van Lummel, J, Adelmeijer, K, VanHoorelbeke, R T, Urbanus, T, Lisman, P G, de Groot
Publikováno v:
Journal of thrombosis and haemostasis : JTH. 5(2)
The major antigen implicated in the antiphospholipid syndrome is beta2-glycoprotein I (beta2GPI). Dimerized beta2GPI binds to apolipoprotein E receptor 2' (apoER2') on platelets and increases platelet adhesion to collagen under conditions of flow.To
Autor:
P. G. De Groot, Maarten T. T. Pennings, Peter J. Lenting, M. Van Lummel, R. T. Urbanus, R. H. W. M. Derksen, Roland A. Romijn
Publikováno v:
Journal of thrombosis and haemostasis : JTH. 4(8)
The antiphospholipid syndrome (APS) is a non-inflammatory autoimmune disease characterized by arterial and/or venous thrombosis and/or pregnancy morbidity in the presence of autoantibodies that recognize beta2-glycoprotein I (beta2GPI) bound to phosp
Autor:
Ronald H. W. M. Derksen, Philip G. de Groot, Bianca C H Lutters, Niels Kaldenhoven, Maarten T.T. Pennings, R. T. Urbanus, Menno van Lummel
Publikováno v:
The Journal of biological chemistry. 280(44)
The antiphospholipid syndrome is caused by autoantibodies directed against beta(2)-glycoprotein I (beta(2)GPI). Dimerization of beta(2)GPI results in an increased platelet deposition to collagen. We found that apolipoprotein E receptor 2' (apoER2'),