Zobrazeno 1 - 10
of 24
pro vyhledávání: '"R N Eisenman"'
Autor:
R N, Eisenman
Publikováno v:
Harvey lectures. 96
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::d02bea527b361a521423404270e465b2
https://pubmed.ncbi.nlm.nih.gov/12200869
https://pubmed.ncbi.nlm.nih.gov/12200869
Autor:
A, Orian, R N, Eisenman
Publikováno v:
Science's STKE : signal transduction knowledge environment. 2001(88)
Although transforming growth factor-beta (TGF-beta) can affect cell cycle arrest, not much molecular detail is known about how TGF-beta-dependent arrest is mediated. Two recent papers shed some light on how this is accomplished. Orian and Eisenman di
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::e5d191de717bce2fdd9e5492877cca95
https://pubmed.ncbi.nlm.nih.gov/11752658
https://pubmed.ncbi.nlm.nih.gov/11752658
Publikováno v:
Cell growthdifferentiation : the molecular biology journal of the American Association for Cancer Research. 12(9)
The homeodomain protein TG-interacting factor (TGIF) represses transcription by histone deacetylase-dependent and -independent means. Heterozygous mutations in human TGIF result in holoprosencephaly, a severe genetic disorder affecting craniofacial d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::e7715e93da536effa3bdcc4201140048
https://pubmed.ncbi.nlm.nih.gov/11571228
https://pubmed.ncbi.nlm.nih.gov/11571228
Autor:
R N, Eisenman
Publikováno v:
Genesdevelopment. 15(16)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c2d4aa748b8d0645406a4f37ffbd0dd2
https://pubmed.ncbi.nlm.nih.gov/11511533
https://pubmed.ncbi.nlm.nih.gov/11511533
Autor:
K, Brubaker, S M, Cowley, K, Huang, L, Loo, G S, Yochum, D E, Ayer, R N, Eisenman, I, Radhakrishnan
Publikováno v:
Cell. 103(4)
Gene-specific targeting of the Sin3 corepressor complex by DNA-bound repressors is an important mechanism of gene silencing in eukaryotes. The Sin3 corepressor specifically associates with a diverse group of transcriptional repressors, including memb
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::b57c4d53ea980f966133c0d074616cf5
https://pubmed.ncbi.nlm.nih.gov/11106735
https://pubmed.ncbi.nlm.nih.gov/11106735
Publikováno v:
Cell growthdifferentiation : the molecular biology journal of the American Association for Cancer Research. 10(12)
The four members of the MAD family are bHLHZip proteins that heterodimerize with MAX and act as transcriptional repressors. The switch from MYC-MAX complexes to MAD-MAX complexes has been postulated to couple cell-cycle arrest with differentiation. T
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::8eaad7f1c559a3708895aac3a8db5ea3
https://pubmed.ncbi.nlm.nih.gov/10616903
https://pubmed.ncbi.nlm.nih.gov/10616903
Publikováno v:
International journal of cancer. 82(6)
Medulloblastomas (MBs) are the most frequent malignant brain tumors in children. The molecular pathogenesis of these tumors is still poorly understood. Microsatellite and restriction-fragment-length polymorphism studies have revealed allelic loss of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::498c9547360ea5083e8b7ce6d83f4499
https://pubmed.ncbi.nlm.nih.gov/10446446
https://pubmed.ncbi.nlm.nih.gov/10446446
Publikováno v:
Current topics in microbiology and immunology. 224
We have identified a novel Max-binding protein, Mnt, which belongs to neither the Myc nor the Mad families (Hurlin et al. 1997). Mnt interacts with Max in vivo and functions as a transcriptional repressor of reporter genes containing promoter-proxima
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::387bce06be7f540b6ad9bb8cfc6c292d
https://pubmed.ncbi.nlm.nih.gov/9308234
https://pubmed.ncbi.nlm.nih.gov/9308234
Publikováno v:
Cell growthdifferentiation : the molecular biology journal of the American Association for Cancer Research. 6(6)
Mad is a bHLH/Zip protein that, as a heterodimer with Max, can repress Myc-induced transcriptional transactivation. Expression of Mad is induced upon terminal differentiation of several cell types, where it has been postulated to down-regulate Myc-in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::a887e44a66bd62b0234b1fc6826cc0cf
https://pubmed.ncbi.nlm.nih.gov/7669717
https://pubmed.ncbi.nlm.nih.gov/7669717
Autor:
E M, Blackwood, R N, Eisenman
Publikováno v:
Methods in enzymology. 254
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::df1b5e324f7c4d1f0f38bc4912b4b65a
https://pubmed.ncbi.nlm.nih.gov/8531689
https://pubmed.ncbi.nlm.nih.gov/8531689