Zobrazeno 1 - 10
of 12
pro vyhledávání: '"R M, Tate"'
Publikováno v:
American Review of Respiratory Disease. 140:294-301
Despite extensive investigation, the pathogenesis of the adult respiratory distress syndrome (ARDS) remains uncertain. As yet, there is no clear explanation of why some patients at risk for ARDS develop the syndrome, whereas others do not. Neutrophil
Autor:
R. M. Tate, J. E. Repine
Publikováno v:
Chest. 83:48S-50
Autor:
R M, Tate, J E, Repine
Publikováno v:
The American review of respiratory disease. 128(3)
Publikováno v:
The American review of respiratory disease. 125(4)
Acute edematous lung injury is associated with a marked increase in the number of granulocytes in the alveoli and microvasculature of the lung. Phorbol myristate acetate (PMA) causes granulocytes to adhere, aggregate, and release oxygen radicals and
Publikováno v:
The American review of respiratory disease. 127(5)
A good model of the adult respiratory distress syndrome (ARDS) is intravenously injected phorbol myristate acetate (PMA), which causes pulmonary sequestration of neutrophils and a neutrophil-dependent acute edematous lung injury in rabbits. In the pr
Autor:
J E, Repine, R M, Tate
Publikováno v:
The Physiologist. 26(3)
Autor:
R M, Tate, T L, Petty
Publikováno v:
Advances in internal medicine. 29
Publikováno v:
The American review of respiratory disease. 126(5)
Oxygen radicals have been implicated in the pathogenesis of permeability pulmonary edema. To determine directly if O2 radicals can cause increased alveolar-capillary membrane (ACM) permeability and low-pressure permeability edema, we chemically produ
Publikováno v:
The American review of respiratory disease. 124(2)
Increased concentrations of angiotensin converting enzyme (ACE) were found in lung lavages from rabbits exposed to hyperoxia for 72 h and the concentrations of ACE were correlated with ratios of extravascular lung water to body weight (r = 0.69, p le
Autor:
S A, Shoemaker, J E, Heffner, E M, Canham, R M, Tate, H G, Morris, I F, McMurtry, J E, Repine
Publikováno v:
The American review of respiratory disease. 129(1)
The potential contributions of bacterial-platelet interactions to the development of acute edematous lung injury, such as that seen in the adult respiratory distress syndrome (ARDS), remains unknown. We found that the addition of Staphylococcus aureu