Zobrazeno 1 - 10
of 127
pro vyhledávání: '"R J Olds"'
Autor:
ALBERS, ANN C.
Publikováno v:
Journal of Allied Health, 1975 Jul 01. 4(3), 49-50.
Externí odkaz:
https://www.jstor.org/stable/45441850
Autor:
D. K. Banerjee
Publikováno v:
Journal of Medical Microbiology. 24:284-284
Autor:
David Neil Cooper, J. Emmerich, Sl Thein, M. Boisclair, D W Perry, R. J. Olds, M. Aiach, V. Chowdhury, Morris A. Blajchman, David A. Lane
Publikováno v:
Scopus-Elsevier
Autor:
Helen Ireland, J. M. Freyssinet, David A. Lane, R J Olds, Howard R. Morris, L. Grunebaum, E. Thompson, S. L. Thein, I. D. Walker, I Blench
Publikováno v:
British Journal of Haematology. 79:70-74
A female with recurrent thrombosis was found to have a functional abnormality of antithrombin, with a ratio of functional to immunological activity in plasma of approximately 50%. Crossed immunoelectrophoresis in the presence of heparin was normal, i
Autor:
R. Caso, D. A. Lane, E. A. Thompson, R. J. Olds, S. L. Thein, M. Panico, I. Blench, H. R. Morris, J. M. Freyssinet, M. Aiach, F. Rodeghiero and, G. Finazzi
Publikováno v:
British Journal of Haematology. 77:87-92
Antithrombin (AT) Vicenza has been previously identified as a functionally abnormal antithrombin associated with familial thrombosis (Finazzi et al, 1985). It binds normally to heparin, but loses its affinity following interaction with thrombin: it i
Publikováno v:
Blood. 76:2182-2186
Type 1 antithrombin III (ATIII) deficiency, which is the commonest form of inherited ATIII defect, is characterized by a quantitative reduction in both immunologically and functionally detectable protein. This condition is associated with a high inci
Publikováno v:
Thrombosis and haemostasis. 86(4)
We report the identification of a new mutation resulting in type I antithrombin (AT) deficiency and the mechanism by which the deficiency arose. The single base substitution of G to A at nucleotide 2709 was identified in a proband with a family histo
Publikováno v:
British journal of haematology. 109(3)
Autor:
T A, Bayston, A, Tripodi, P M, Mannucci, E, Thompson, H, Ireland, A C, Fitches, L, Hananeia, R J, Olds, D A, Lane
Publikováno v:
Blood. 93(12)
We have investigated the basis of antithrombin deficiency in an asymptomatic individual (and family) with borderline levels (approximately 70% antigen and activity) of antithrombin. Direct sequencing of amplified DNA showed a mutation in codon 135, A
Publikováno v:
Blood. 92(12)
Most secretory proteins, including antithrombin (AT), are synthesized with a signal peptide, which is cleaved before the mature protein is exported from the cell. The signal peptide is important in the process whereby nascent protein is recognized as