Zobrazeno 1 - 10
of 14
pro vyhledávání: '"R C, Hard"'
Publikováno v:
Clinical and Experimental Immunology. 100:352-358
SUMMARY Rather than central tolerance, the perinatal inoculation of related F1 hybrid spleen cells into inbred mice may result in host-versus-graft (HVG) reactions manifested as transient autoimmunity, or as a lethal immunodeficiency syndrome. RFM/(T
Publikováno v:
Archives of pathologylaboratory medicine. 115(9)
Spontaneous chimerism is rare, but it has been observed in infants who have received intrauterine transfusions, or in those infants who have severe combined immunodeficiency disease. The site of maternal cell engraftment has not been identified in th
Publikováno v:
Infection and Immunity. 41:570-577
Host versus graft disease is the fatal syndrome of altered immunity that follows the perinatal inoculation of related F1 hybrid spleen cells to susceptible strains of inbred mice. The allogenic reaction results in severe depletion of T-lymphocytes, b
Publikováno v:
American Journal of Reproductive Immunology. 1:240-245
Antisera to some simian retroviruses were previously shown to react with human fetal cells. Conversely, it is demonstrated here that an antiserum to syncytialtro-phoblast plasma membrane (TrPM antiserum) possesses complement-dependent cytotoxic activ
Publikováno v:
Journal of Histochemistry & Cytochemistry. 37:909-912
We describe an immunocytochemical assay for cells forming antibody to glucose oxidase (GO). The method is specific in that only cells containing intracytoplasmic antibody capable of binding the immunogen (GO binding cells; GOBC) are stained. The meth
Publikováno v:
The Journal of Immunology. 131:2623-2629
Host-vs-graft (HVG) syndrome is the fatal disease of altered immunity that may be induced in susceptible strains of inbred mice by the perinatal inoculation of semiallogenic spleen cells. The allogenic HVG reaction causes severe depletion of both the
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 131(6)
Host-vs-graft (HVG) syndrome is the fatal disease of altered immunity that may be induced in susceptible strains of inbred mice by the perinatal inoculation of semiallogenic spleen cells. The allogenic HVG reaction causes severe depletion of both the
Autor:
R C, Hard
Publikováno v:
Clinical and experimental immunology. 22(2)
Host versus graft (HVG) syndrome may be induced in parental strain mice by perinatal inoculations of F1 hybrid spleen cells. The principal manifestations of the disease include thrombocytopaenia, intravascular fibrin deposits, intestinal haemorrhage,
Publikováno v:
Cancer detection and prevention. 7(4)
It appears that the development and type of lymphoid neoplasm in mice depends on the ratio of T cell subsets and the repeated stimulation of the lymphoid systems as well as a genetic predisposition. Mice with HVG disease developed lymphomas in an ave
Autor:
R C, Hard
Publikováno v:
Immunology. 39(4)
RFM mice perinatally inoculated with (T6 X RFM)F1 spleen cells develop raidply progressive host-versus-graft (HVG) disease. They are usually dead by 30 days with hyperimmunoglobulinaemia, immune complexes, plasmacytosis and marked T-cell deficiency.