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Akademický článek
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Publikováno v:
Journal of Comparative Pathology. 154:80
Publikováno v:
Food and Chemical Toxicology. 28:197-203
To investigate the relationship between vitamin A status and 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) lethality, the influence of TCDD on tissue and serum vitamin A levels was determined in the most TCDD-susceptible (Long-Evans) and the most TCDD-r
Autor:
S. Mahiout, R. Pohjanvirta
Publikováno v:
Toxicology Letters. 238:S308
Autor:
Riitta Maijala, E.-R. Venäläinen, L. Vahteristo, R. Pohjanvirta, E. Lindfors, M. Eskola, T. Lyytikäinen
Publikováno v:
Food additives and contaminants. 20(5)
Although the average cadmium intake in Finland is about 10 microg day(-1), some risk groups can be identified. This study assessed cadmium intake from the consumption of moose meat, liver and kidneys by moose hunters. Consumption data from a postal q
Publikováno v:
Biochemical pharmacology. 62(12)
The aryl hydrocarbon receptor (AHR) mediates toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and regulates expression of several genes such as CYP1A1. Little is known about what regulates expression of the AHR itself. We tested the ability of
Akademický článek
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Publikováno v:
Advances in experimental medicine and biology. 467
Polychlorinated dioxins (PCDD) are widespread environmental contaminants. The most potent and the general model compound for dioxins is 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD). Our laboratory has developed a new model for studies of dioxin toxicit
Publikováno v:
Pharmacology, biochemistry, and behavior. 62(4)
Interactions of diet and diet-induced obesity, and the characteristic wasting syndrome caused by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) were studied in TCDD-resistant Han/Wistar and TCDD-sensitive Long-Evans rats. The rats were made obese by feed
Autor:
A M, Partanen, S, Alaluusua, P J, Miettinen, I, Thesleff, J, Tuomisto, R, Pohjanvirta, P L, Lukinmaa
Publikováno v:
Laboratory investigation; a journal of technical methods and pathology. 78(12)
We have previously shown that dioxins at prevailing levels in mothers' milk may cause mineralization defects in the developing teeth of their children. Developmental dental defects have also been reported in rhesus macaques and rats experimentally ex