Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Quinesha A. Williams"'
Autor:
Bo Liu, Heng Zeng, Han Su, Quinesha A. Williams, Jessie Besanson, Yingjie Chen, Jian‐Xiong Chen
Publikováno v:
Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, Vol 13, Iss 16 (2024)
Background Endothelial prolyl hydroxylase‐2 (PHD2) is essential for pulmonary remodeling and hypertension. In the present study, we investigated the role of endothelial PHD2 in angiotensin II–mediated arterial stiffness, pericyte recruitment, and
Externí odkaz:
https://doaj.org/article/9162e368d9a64a469bdf6a5a2833ab7c
Autor:
Xiaochen He, Quinesha A. Williams, Aubrey C. Cantrell, Jessie Besanson, Heng Zeng, Jian-Xiong Chen
Publikováno v:
International Journal of Molecular Sciences, Vol 25, Iss 4, p 2433 (2024)
Hypertension is the key contributor to pathological cardiac hypertrophy. Growing evidence indicates that glucose metabolism plays an essential role in cardiac hypertrophy. TP53-induced glycolysis and apoptosis regulator (TIGAR) has been shown to regu
Externí odkaz:
https://doaj.org/article/637f66e6f6834ff98bb15be04ff60b87
Publikováno v:
Cells, Vol 12, Iss 7, p 1060 (2023)
The induction of hypoxia tolerance has emerged as a novel therapeutic strategy for the treatment of ischemic diseases. The disruption of hypoxic signaling by hyperglycemia has been shown to contribute to diabetic cardiomyopathy. In this study, we exp
Externí odkaz:
https://doaj.org/article/d5abf02186db48c792f6027249ef7fa9
Publikováno v:
Physiological Reports, Vol 10, Iss 8, Pp n/a-n/a (2022)
Abstract Angiotensin II (Ang‐II) is one of the major contributors to the progression of renal fibrosis, inflammation, glomerular injury, and chronic kidney disease. Emerging evidence suggests that renal glycolysis plays an important role in renal f
Externí odkaz:
https://doaj.org/article/6eb662de8baf43abbbcaba403fb10c43
Autor:
Xiaochen He, Aubrey C Cantrell, Quinesha A Williams, Wei Gu, Yingjie Chen, Jian-Xiong Chen, Heng Zeng
Publikováno v:
bioRxiv
Coronary microvascular dysfunction (CMD) has been shown to contribute to cardiac hypertrophy and heart failure with preserved ejection fraction. At this point, there are no proven treatments for CMD. We have shown that histone acetylation may play a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c9e7324f98adeb1297e5d78a373550f8
https://europepmc.org/articles/PMC9934706/
https://europepmc.org/articles/PMC9934706/
Publikováno v:
Journal of cellular physiology. 237(8)
Hypertension is an important risk factor in the pathogenesis of diastolic dysfunction. Growing evidence indicates that glucose metabolism plays an essential role in diastolic dysfunction. TP53-induced glycolysis and apoptosis regulator (TIGAR) has be
Publikováno v:
Biophysical Journal. 120:305a
Autor:
Brittany Notice, Isabelle Petit-Hӓrtlein, Quinesha A. Williams, Victoria Gonzalez, Franck Fieschi, Thomas C. Leeper, Annelise Vermot, Sarah Waldron, Susan M.E. Smith
Publikováno v:
Biophysical Journal. 120:306a