Zobrazeno 1 - 10
of 25
pro vyhledávání: '"Quan-Yun Wang"'
Autor:
Hua Fang, Hua-Feng Li, Miao Yang, Ru-Rong Wang, Quan-Yun Wang, Peng-Cheng Zheng, Fang-Xiang Zhang, Jian-Ping Zhang
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 165, Iss , Pp 114874- (2023)
Externí odkaz:
https://doaj.org/article/17bf36c425a34ca58de1c2d9104b6437
Autor:
Hua Fang, Hua-Feng Li, Qin Pan, Hon-Ling Jin, Miao Yang, Ru-Rong Wang, Quan-Yun Wang, Jian-Ping Zhang
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 9 (2021)
Spinal cord ischemia-reperfusion (SCIR) injury is a serious complication of open surgical and endovascular aortic procedures. MicroRNA-132-3p (miR-132-3p) has been reported to be involved in the progression of various diseases, but its role in SCIR i
Externí odkaz:
https://doaj.org/article/87626f3266eb4823866d0314702b7329
Autor:
Hua Fang, Hua-Feng Li, Miao Yang, Ru-Rong Wang, Quan-Yun Wang, Peng-Cheng Zheng, Fang-Xiang Zhang, Jian-Ping Zhang
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 113, Iss , Pp - (2019)
Objective: Hypoxic-ischemic brain damage (HIBD) is a major cause of acute mortality and chronic neurological morbidity in infants and children. Dexmedetomidine (DEX) is an effective choice in HIBD treatment. Recent findings have revealed that microRN
Externí odkaz:
https://doaj.org/article/0064a627a0fe45efb525168099dadfec
Autor:
Ru-Rong Wang, Qin Pan, Hon-Ling Jin, Hua Fang, Miao Yang, Jian-Ping Zhang, Hua-Feng Li, Quan-Yun Wang
Publikováno v:
Journal of Neurochemistry. 156:106-120
Cell death after spinal cord ischemia/reperfusion (I/R) can occur through necrosis, apoptosis, and autophagy, resulting in changes to the immune environment. However, the molecular mechanism of this immune regulation is not clear. Accumulating eviden
Autor:
Hua Fang, Miao Yang, Quan-Yun Wang, Ren Liao, Fang-Xiang Zhang, Ru-Rong Wang, Jian-Ping Zhang, Peng-Cheng Zheng, Hua-Feng Li
Publikováno v:
Cell Cycle
NF-κB is a core transcription factor, the activation of which can lead to hypoxic-ischemic brain damage (HIBD), while RCAN1 plays a protective role in HIBD. However, the relationship between NF-κB and RCAN1 in HIBD remains unclear. This study aimed
Autor:
Qin Pan, Quan-Yun Wang, Hua-Feng Li, Fang-Xiang Zhang, Ru-Rong Wang, Miao Yang, Jian-Ping Zhang, Hua Fang
Publikováno v:
The American journal of pathology. 191(3)
Long noncoding RNAs play critical roles in cellular homeostasis, and long noncoding RNA H19 (H19) is implicated in several pathologic conditions. The putative role of H19 in the pathogenesis and progression of hypoxic-ischemic brain damage (HIBD) is
Autor:
Fang-Xiang Zhang, Quan-Yun Wang, Jian-Ping Zhang, Hua-Feng Li, Ren Liao, Hua Fang, Peng-Cheng Zheng, Ru-Rong Wang, Miao Yang
Publikováno v:
Journal of Clinical Laboratory Analysis
Background We conducted a randomized clinical trial to determine whether adjunctive lidocaine diminishes the incidence of adverse effects in pediatric patients sedated with ketamine. Methods This case‐control study involved 586 consecutive pediatri
Autor:
Ren Liao, Hua Fang, Ru-Rong Wang, Hua-Feng Li, Miao Yang, Jian-Ping Zhang, Quan-Yun Wang, Peng-Cheng Zheng, Fang-Xiang Zhang
Publikováno v:
Journal of cellular physiology. 235(3)
Long noncoding RNAs have been documented to be protective against ischemia/reperfusion (I/R) injury. However, few research works have focused on the protective effects of PRR34-AS1 on I/R injury after total knee arthroplasty (TKA). The objective of t
Autor:
Jian-Ping Zhang, Quan-Yun Wang, Hua Fang, Ru-Rong Wang, Miao Yang, Fang-Xiang Zhang, Jian-Yong Yan, Hua-Feng Li, Ming-Hai He
Publikováno v:
Brain research bulletin. 152
Hypoxic-ischemic brain damage (HIBD) is a common neurological disorder. Emerging reports reveal that long non-coding RNAs and microRNAs (miRs) are implicated in the progress of HIBD. In this study we tried to ascertain whether lncRNA MALAT1, with the
Autor:
Jian-Ping Zhang, Hua-Feng Li, Ru-Rong Wang, Hua Fang, Fang-Xiang Zhang, Miao Yang, Peng-Cheng Zheng, Quan-Yun Wang
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 113, Iss, Pp-(2019)
Objective Hypoxic-ischemic brain damage (HIBD) is a major cause of acute mortality and chronic neurological morbidity in infants and children. Dexmedetomidine (DEX) is an effective choice in HIBD treatment. Recent findings have revealed that microRNA