Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Qiaoya Ma"'
Publikováno v:
Molecular Medicine, Vol 27, Iss 1, Pp 1-13 (2021)
Abstract Background Alzheimer’s disease (AD), the most common form of dementia, is caused by the degeneration of the central nervous system (CNS). A previous study reported that signal transducer and activator of transcription 3 (STAT3) is activate
Externí odkaz:
https://doaj.org/article/6c3b6354346f48fd8f7bda8a110db5d2
Publikováno v:
Image Analysis and Stereology, Vol 36, Iss 2, Pp 111-120 (2017)
Studies have provided qualitative evidence of de-myelination and re-myelination in aged brain white matter. However, there have been no quantitative evidences of degeneration and regeneration of myelin sheaths in white matter. The present study was d
Externí odkaz:
https://doaj.org/article/a7f791cc79f042a2a7156a45b92211f8
Publikováno v:
Acta neurobiologiae experimentalis. 82(3)
Alzheimer’s disease (AD) is a chronic and progressive neurodegenerative disease that is the most common cause of dementia in the elderly. Aβ1‑42 is significantly associated with memory deficits and it can increase the level of acetylcholine, pro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0160255c34256e31443b568ee4bfdbc3
https://pubmed.ncbi.nlm.nih.gov/36214721
https://pubmed.ncbi.nlm.nih.gov/36214721
Publikováno v:
Molecular Medicine
Molecular Medicine, Vol 27, Iss 1, Pp 1-13 (2021)
Molecular Medicine, Vol 27, Iss 1, Pp 1-13 (2021)
Background Alzheimer’s disease (AD), the most common form of dementia, is caused by the degeneration of the central nervous system (CNS). A previous study reported that signal transducer and activator of transcription 3 (STAT3) is activated during
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9e41792f8d473c070bc8652830232499
http://europepmc.org/articles/PMC8626961
http://europepmc.org/articles/PMC8626961
Publikováno v:
Cellular and Molecular Biology. 65:54-58
Alzheimer's disease (AD) is a high-incidence neurodegenerative disease with complex and diverse pathogenesis. With aging of the population and continuous improvement of living standards, the incidence of AD is on the increase. Therefore, there is nee
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e2c2fc7e53110e2bdf36ad7df6402f2d
https://doi.org/10.14715/cmb/2019.65.5.9
https://doi.org/10.14715/cmb/2019.65.5.9
Publikováno v:
Cellular and Molecular Biology. 65:63-68
To investigate the expressions and roles of semaphorin3A (Sema3A) and vascular endothelial growth factor 165 (VEGF165) in cultured rat cortical neurons and vascular endothelial cells after oxygen glucose deprivation (OGD) stimulation. Cultured cortic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::89783ff2f996b5cab149bf3692a9a335
https://doi.org/10.14715/cmb/2019.65.4.10
https://doi.org/10.14715/cmb/2019.65.4.10
Publikováno v:
Genes to cells : devoted to molecularcellular mechanismsREFERENCES. 25(6)
Microglial inflammation is identified as a key process associated with Parkinson's disease (PD) pathogenesis. Our previous study showed that miR-29c-3p (miR-29c) exhibited anti-inflammatory properties in PD animal and neuronal models. However, the sp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f6a0b01063df346c6c9cfb59f181ecdf
https://pubmed.ncbi.nlm.nih.gov/32160394
https://pubmed.ncbi.nlm.nih.gov/32160394
Publikováno v:
Cellular and Molecular Biology. 64:119-124
Pyridoxine is a water- soluble pyridine derivative. The effect of pyridoxine in cell models of Alzheimer's disease (AD), and the potential mechanisms involved, are not fully understood. In this study, the anti-AD effects of pyridoxine were studied in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0af8fcc4d6d9d8f3c49951c59f3d33d5
https://doi.org/10.14715/cmb/2018.64.10.19
https://doi.org/10.14715/cmb/2018.64.10.19
Publikováno v:
Cellular and Molecular Biology. 64:64-69
To investigate the expression changes and roles of Semaphorin3A (Sema3A) and Neuropilin-1 (Nrp1) in cultured rat cortical neurons after oxygen glucose deprivation (OGD). Cultured cortical neurons of newborn SD rats were divided randomly into control
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a9cc58b81af020b6c88297fb026a669f
https://doi.org/10.14715/cmb/2018.64.9.10
https://doi.org/10.14715/cmb/2018.64.9.10
Publikováno v:
Yonsei Medical Journal
Purpose Ischemic brain injury results in high mortality and serious neurologic morbidity. Here, we explored the role of SNHG15 in modulating neuronal damage and microglial inflammation after ischemia stroke. Materials and methods The hypoxia/ischemia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::98555263bb105541495ce7cf0ead9def
https://doi.org/10.3349/ymj.2021.62.4.325
https://doi.org/10.3349/ymj.2021.62.4.325