Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Qian-Qian Geng"'
Autor:
Yan-Song Lin, Hui Yang, Xiao-Yi Li, Li-Qing Wu, Jin-Guo Xu, Ai-Min Yang, Zai-Rong Gao, Yong Ding, Ying-Qiang Zhang, Kai Chen, Zhuan-Zhuan Mu, Jian-Min Jia, Na Niu, Di Sun, Xin Zhang, Shao-Qiang Zhang, Qian-Qian Geng, Ya-Jing Zhang, Fang-Ni Chen, Bao-Xia He
Publikováno v:
European journal of nuclear medicine and molecular imaging. 49(12)
Purpose Thyroid hormone withdrawal (THW) inevitably induced hypothyroidism in patients with differentiated thyroid cancer (DTC), we aimed to evaluate the safety and efficacy of a novel recombinant human thyroid-stimulating hormone (rhTSH, ZGrhTSH) as
Autor:
Qian-Qian, Geng, Yin-Liu, Wang, Guo-Xiang, Niu, Nan-Nan, Wang, Muqier, Hasi, Ang, Li, Jian-Hui, Huang
Publikováno v:
Ying yong sheng tai xue bao = The journal of applied ecology. 32(8)
Increasing atmospheric nitrogen (N) deposition greatly affects species diversity, productivity, and stability of ecosystems. It is thus of the great importance to understand how grassland N pools respond to the increased atmospheric N deposition. Thi
Publikováno v:
Zhonghua zhong liu za zhi [Chinese journal of oncology]. 35(6)
To discuss the expression and clinical significance of VEGF-C and nm23-H1 in stage II and III colorectal carcinomas.SP immunohistochemical staining was employed to determine the expression of vascular endothelial growth factor-C (VEGF-C) and nm23-H1
Autor:
Shaomeng Wang, Dan Feng Dong, Yin Ying Wu, Nan Zheng Chen, En Xiao Li, Jie Wang, Qian Qian Geng
Publikováno v:
International journal of oncology. 43(6)
The tumor suppressor gene p53 is often inactivated in breast cancer cells due to gene mutation or overexpression of its repressors (such as murine double minute 2 and murine double minute X). Inhibitors of murine double minute 2 (MDM2) and murine dou
Publikováno v:
Journal of Clinical Oncology. 31:e22096-e22096
e22096 Background: The MDM2 inhibitor which disrupted the MDM2-p53 interaction made little effort on the activation of p53 for breast cancer treatments, due to MDMX over expression. Previously a small molecule inhibitor targeting at MDM2 and MDMX had