Zobrazeno 1 - 10
of 56
pro vyhledávání: '"Punnee Butthep"'
Autor:
Chanita Sanyear, Buraporn Chiawtada, Punnee Butthep, Saovaros Svasti, Suthat Fucharoen, Patarabutr Masaratana
Publikováno v:
PeerJ, Vol 9, p e11367 (2021)
Background Hepcidin controls iron homeostasis by inducing the degradation of the iron efflux protein, ferroportin (FPN1), and subsequently reducing serum iron levels. Hepcidin expression is influenced by multiple factors, including iron stores, ineff
Externí odkaz:
https://doaj.org/article/7fc2ed387e8a4311bda038d2f5801fd8
Autor:
Chanita Sanyear, Punnee Butthep, Wiraya Eamsaard, Suthat Fucharoen, Saovaros Svasti, Patarabutr Masaratana
Publikováno v:
PeerJ, Vol 8, p e8802 (2020)
Background Iron overload is one of common complications of β-thalassemia. Systemic iron homeostasis is regulated by iron-regulatory hormone, hepcidin, which inhibits intestinal iron absorption and iron recycling by reticuloendothelial system. In add
Externí odkaz:
https://doaj.org/article/07f1e3fe9d514b2da97b38ffd27a3f36
Autor:
Sirichan Chunhakan, Punnee Butthep, Sutee Yoksan, Kanchana Tangnararatchakit, Ampaiwan Chuansumrit
Publikováno v:
International Journal of Vascular Medicine, Vol 2015 (2015)
The vascular leakage was shown by the increment of hematocrit (Hct), dengue viral infected monocyte, monocyte status, and cytokines production in patients infected with dengue virus. Dengue viral antigens were demonstrated in monocytes (CD14+) from p
Externí odkaz:
https://doaj.org/article/ba71cd5b29564177849f1b73b06a9257
Publikováno v:
Journal of Clinical Laboratory Analysis. 36
The high concentrated thrombin time (hcTT), a thrombin time modified by increasing the thrombin concentration, is a possible alternative assay to activated partial thromboplastin time (aPTT) in unfractionated heparin (UFH) monitoring. This study aime
Autor:
Patarabutr Masaratana, Suthat Fucharoen, Buraporn Chiawtada, Chanita Sanyear, Punnee Butthep, Saovaros Svasti
Publikováno v:
PeerJ
PeerJ, Vol 9, p e11367 (2021)
PeerJ, Vol 9, p e11367 (2021)
Background Hepcidin controls iron homeostasis by inducing the degradation of the iron efflux protein, ferroportin (FPN1), and subsequently reducing serum iron levels. Hepcidin expression is influenced by multiple factors, including iron stores, ineff
Autor:
Suthat Fucharoen, Punnee Butthep, Saovaros Svasti, Chanita Sanyear, Patarabutr Masaratana, Wiraya Eamsaard
Publikováno v:
PeerJ, Vol 8, p e8802 (2020)
PeerJ
PeerJ
Background Iron overload is one of common complications of β-thalassemia. Systemic iron homeostasis is regulated by iron-regulatory hormone, hepcidin, which inhibits intestinal iron absorption and iron recycling by reticuloendothelial system. In add
Publikováno v:
Hemoglobin. 41(4-6)
Thalassemias and glucose-6-phosphate dehydrogenase (G6PD) deficiency are the most common inherited blood disorders. They are distributed among populations living in malaria endemic regions resulting in survival advantage from severe malaria disease.
Autor:
Atthapol Srisuddee, Wasun Chantratita, Pawinee Kupatawintu, Punnee Butthep, Mayuree Kengkate, Oytip Nathalang
Publikováno v:
Journal of Clinical Laboratory Analysis. 29:94-99
Background We aimed to compare HPA-1 to HPA-6 and HPA-15 genotyping results obtained by a simple-probe real-time polymerase chain reaction (PCR) technique with the multiplex PCR technique. Methods Five hundred DNA samples from the Thai National Stem
Autor:
Futrakul, Narisa1 fmednft@md.chula.ac.th, Punnee Butthep1, Suthiluk Patumraj1, Prasong Siriviriyakul1, Prasit Futrakul1
Publikováno v:
Clinical Hemorheology & Microcirculation. 2006, Vol. 34 Issue 1-2, p265-271. 7p. 2 Diagrams, 1 Chart.
Autor:
Pornpan Sirankapracha, Chanita Sanyear, Ramaneeya Nithipongvanich, Punnee Butthep, Suthat Fucharoen, Pranee Winichagoon, Saovaros Svasti
Publikováno v:
International Journal of Experimental Pathology. 94:336-342
β-thalassaemia is a hereditary anaemia resulting from the absence or reduction in β-globin chain production. Heart complications related to iron overload are the most serious cause of death in these patients. In this report cardiac pathology of β-