Zobrazeno 1 - 10
of 42
pro vyhledávání: '"Prakash babu, Phanithi"'
Publikováno v:
Heliyon, Vol 8, Iss 12, Pp e12278- (2022)
Aim: Autophagy is a catabolic process, which plays a pivotal role in neuronal homeostases. Sirtuin-1 (Sirt1, Silent information regulator family protein 1) is a protein deacetylase that is activated by nicotinamide adenine dinucleotide (NAD+), is als
Externí odkaz:
https://doaj.org/article/d1a62377eec5492f97312a8b0a0dc5d6
Publikováno v:
In Journal of Clinical Neuroscience February 2020 72:402-411
Autor:
Shankaraiah Pagilla, Bhavani Anagani Kanaka Durga, Sumathi Vodnala, Ramakrishna Kamutam, Anwita Mudiraj, Prakash Babu Phanithi, Prabhakar Chetti
Publikováno v:
Journal of the Chinese Chemical Society. 69:968-978
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::06bd0a2ed894727fb9fd192e23f85df1
https://doi.org/10.1002/jccs.202200058
https://doi.org/10.1002/jccs.202200058
Autor:
Kishore Madhamanchi, Pradeep Madhamanchi, Sita Jayalakshmi, Manas Panigrahi, Anuja Patil, Prakash Babu Phanithi
Publikováno v:
Molecular Neurobiology.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::728a8baacf53b97f37cb60feb53472b7
https://doi.org/10.1007/s12035-023-03361-4
https://doi.org/10.1007/s12035-023-03361-4
Autor:
Venkata Prasuja, Nakka, Raghu, Gogada, Praveen Kumar, Simhadri, Mohammed Abdul, Qadeer, Prakash Babu, Phanithi
Publikováno v:
Brain Research Bulletin. 182:1-11
Stroke leads to disturbance in the physiology of the ER (Endoplasmic Reticulum) that triggers UPR (Unfolded Protein Response) pathways aimed to compensate neuronal cell damage. However, sustained UPR causes stressful conditions in the ER lumen formin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4c841cb921d5d04fd89ddaee1f93480b
https://doi.org/10.1016/j.brainresbull.2022.02.003
https://doi.org/10.1016/j.brainresbull.2022.02.003
Autor:
Teertam, Sireesh Kumar1, Prakash Babu, Phanithi1 prakash@uohyd.ac.in
Publikováno v:
Scientific Reports. 3/18/2021, Vol. 11 Issue 1, p1-14. 14p.
Autor:
Kishore Madhamanchi, Pradeep Madhamanchi, Sita Jayalakshmi, Manas Panigrahi, Anuja Patil, Prakash Babu Phanithi
Publikováno v:
Cell Stress Chaperones
Epileptic seizures occur due to an imbalance between excitatory and inhibitory neurosignals. The excitotoxic insults promote the accumulation of reactive oxygen species (ROS), unfolded proteins (UFP) aggregation, and sometimes even cell death. The ep
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0ba2d3134b83210f3443ac3cf7927bbf
https://europepmc.org/articles/PMC9672265/
https://europepmc.org/articles/PMC9672265/
Distinct expression and function of breast cancer metastasis suppressor 1 in mutant P53 glioblastoma
Publikováno v:
Cellular oncology (Dordrecht). 45(6)
Glioblastoma (GBM) is the most malignant subtype of astrocytic tumors with the worst prognosis in all its progressive forms. Breast cancer metastasis suppressor 1 (BRMS1) is a metastasis suppressor gene that controls malignancy in multiple tumors. As
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::db2b747a5b98caf061e042e7690bbdea
https://pubmed.ncbi.nlm.nih.gov/36284039
https://pubmed.ncbi.nlm.nih.gov/36284039
Autor:
Parimala Narne, Prakash Babu Phanithi
Publikováno v:
Cellular and Molecular Neurobiology.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c4090528ca5ff74de6f07f4144f6cbfe
https://doi.org/10.1007/s10571-022-01287-4
https://doi.org/10.1007/s10571-022-01287-4
Autor:
Prabhakar Eeka, Prakash Babu Phanithi
Publikováno v:
Journal of molecular neuroscience : MN. 72(12)
Knockdown studies have shown lymphotoxin-α (Lt-α) as a critical molecule for Experimental cerebral malaria (ECM) pathogenesis. We investigated the role of lymphotoxin-α in regulating active caspase-3 and calpain1. T cell infiltration into the brai
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f1fa627d0a11a8b868f68e8fcb0ce5ff
https://pubmed.ncbi.nlm.nih.gov/36469197
https://pubmed.ncbi.nlm.nih.gov/36469197