Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Prajakta S. Palkar"'
Autor:
Prajakta S. Palkar
Publikováno v:
2019 3rd International conference on Electronics, Communication and Aerospace Technology (ICECA).
Noise pollution ranked second among the environmental stressors for public health impact. In silence zones such as hospitals and school area adults and children are in significant number. In many silence zones sound level is not maintained as per the
Autor:
Wen-Chi L. Chang, Bokai Zhu, Jeffrey M. Peters, Michael G. Borland, Frank J. Gonzalez, Jennie L. Williams, Lance R. Kramer, Prajakta S. Palkar, Jennifer E. Foreman, Margie L. Clapper
Publikováno v:
Molecular Carcinogenesis. 50:884-900
This study critically examined the role of PPARb/d in colon cancer models. Expression of PPARb/d mRNA and protein was lower and expression of CYCLIN D1 protein higher in human colon adenocarcinomas compared to matched nontransformed tissue. Similar r
Publikováno v:
Toxicology and Applied Pharmacology. 230:338-345
Administration of a low priming dose of 2-butoxyethanol (BE, 500 mg/kg, p.o.) 7 days prior to a larger LD(90) dose (1500 mg BE/kg, p.o.) offers protection against the lethal dose-induced hemolysis and death in female Sprague Dawley rats because of pr
Publikováno v:
Toxicology and Applied Pharmacology. 225:102-112
Protection against a high dose of a toxicant by prior exposure to another toxicant is called heteroprotection. Our objective was to establish a heteroprotection model in RBCs. Female Sprague Dawley rats treated with an LD90 dose of 2-butoxyethanol (B
Autor:
Prajakta S. Palkar, Moiz Mumtaz, Sathanandam S. Anand, John R. Latendresse, Harihara M. Mehendale, Binu K. Philip
Publikováno v:
Toxicology and Applied Pharmacology. 216:108-121
Protection offered by pre-exposure priming with a small dose of a toxicant against the toxic and lethal effects of a subsequently administered high dose of the same toxicant is autoprotection. Although autoprotection has been extensively studied with
Autor:
Harihara M. Mehendale, Moiz Mumtaz, Sathanandam S. Anand, Binu K. Philip, Prajakta S. Palkar, John R. Latendresse
Publikováno v:
Toxicology and Applied Pharmacology. 213:267-281
The aims of the present study were to characterize the subchronic toxicity of chloroform by measuring tissue injury, repair, and distribution of chloroform and to assess the reasons for the development of tolerance to subchronic chloroform toxicity.
Autor:
Jennifer E, Foreman, Wen-Chi L, Chang, Prajakta S, Palkar, Bokai, Zhu, Michael G, Borland, Jennie L, Williams, Lance R, Kramer, Margie L, Clapper, Frank J, Gonzalez, Jeffrey M, Peters
Publikováno v:
Molecular carcinogenesis. 50(11)
This study critically examined the role of PPARβ/δ in colon cancer models. Expression of PPARβ/δ mRNA and protein was lower and expression of CYCLIN D1 protein higher in human colon adenocarcinomas compared to matched non-transformed tissue. Simi
Autor:
Gary H. Perdew, Rolf Müller, Michael G. Borland, Shantu Amin, Jeffrey M. Peters, Iain A. Murray, Prajakta S. Palkar, Arun Sharma, Ugir Hossain Sk, Cherie R. Anderson, Christina H. Ferry, Frank J. Gonzalez, Simone Naruhn, Christina Lee
The availability of high-affinity agonists for peroxisome proliferator-activated receptor-β/δ (PPARβ/δ) has led to significant advances in our understanding of the functional role of PPARβ/δ. In this study, a new PPARβ/δ antagonist, 4-chloro-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::71d8294050ce5f0f90fd1a42f2f3dba0
https://europepmc.org/articles/PMC2939490/
https://europepmc.org/articles/PMC2939490/
Autor:
John L. Butenhoff, Prajakta S. Palkar, Jeffrey M. Peters, Boo Hyon Kang, Jennifer E. Foreman, David J. Ehresman, Frank J. Gonzalez, Cherie R. Anderson, Shu Ching Chang
Publikováno v:
Toxicological sciences : an official journal of the Society of Toxicology. 110(1)
Perfluorobutyrate (PFBA) is a short chain perfluoroalkyl carboxylate that is structurally similar to perfluorooctanoate. Administration of PFBA can cause peroxisome proliferation, induction of peroxisomal fatty acid oxidation and hepatomegaly, sugges
Publikováno v:
Current Protocols in Toxicology
Many hepatotoxicants like acetaminophen, chloroform, carbon tetrachloride, halothane, and thioacetamide cause hepatotoxicity through covalent binding of their reactive metabolites to proteins. The covalent binding to proteins may lead to dysfunction