Zobrazeno 1 - 10
of 29
pro vyhledávání: '"Prabhat R, Napit"'
Autor:
Karen P. Briski, Prabhat R. Napit, Abdulrahman Alhamyani, Jérôme Leprince, A.S.M. Hasan Mahmood
Publikováno v:
ASN Neuro, Vol 15 (2023)
Central endozepinergic signaling is implicated in glucose homeostasis. Ventromedial hypothalamic nucleus (VMN) metabolic monitoring governs glucose counter-regulation. VMN glucose-stimulatory nitric oxide (NO) and glucose-inhibitory γ-aminobutyric a
Externí odkaz:
https://doaj.org/article/0a33517431af4e798a870ea4a71f61ed
Publikováno v:
AIMS Neuroscience, Vol 8, Iss 4, Pp 510-525 (2021)
Gamma-aminobutyric acid (GABA) acts on ventromedial hypothalamic targets to suppress counter-regulatory hormone release, thereby lowering blood glucose. Maladaptive up-regulation of GABA signaling is implicated in impaired counter-regulatory outflow
Externí odkaz:
https://doaj.org/article/e5c981b3b0f343cda58add9656d98cc8
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-11 (2021)
Abstract Astrocyte glycogen, the primary energy reserve in brain, undergoes continuous remodeling by glucose passage through the glycogen shunt prior to conversion to the oxidizable energy fuel l-lactate. Glucogenic amino acids (GAAs) are a potential
Externí odkaz:
https://doaj.org/article/e4b858a45e394e799d3b44841f5da804
Autor:
K.P. Briski, Prabhat R. Napit, Md. Haider Ali, A.A. Alshamrani, A.R. Alhamyani, Khaggeswar Bheemanapally, Mostafa M.H. Ibrahim
Publikováno v:
Endocrine and Metabolic Science, Vol 3, Iss , Pp 100087- (2021)
Ventromedial hypothalamic nucleus (VMN) glycogen metabolism affects local glucoregulatory signaling. The hindbrain metabolic-sensitive catecholamine (CA) neurotransmitter norepinephrine controls VMN glycogen phosphorylase (GP)-muscle (GPmm) and -brai
Externí odkaz:
https://doaj.org/article/e34d43bf1a9f4ea4884d90a552a7c10d
Autor:
Sagor Chandra Roy, Prabhat R. Napit, MadhuBabu Pasula, Khaggeswar Bheemanapally, Karen P. Briski
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 324:R20-R34
Astrocytes store glycogen as energy and promote neurometabolic stability through supply of oxidizable l-lactate. Whether lactate regulates ventromedial hypothalamic nucleus (VMN) glucostatic function as a metabolic volume transmitter is unknown. Curr
Publikováno v:
ASN Neuro, Vol 12 (2020)
Norepinephrine (NE) directly regulates ventromedial hypothalamic nucleus (VMN) glucoregulatory neurons and also controls glycogen-derived fuel provision to those cells. VMN nitric oxide (NO) and γ-aminobutyric acid (GABA) neurons and astrocytes expr
Externí odkaz:
https://doaj.org/article/f97949197887498eb70a29ee5a25cb5a
Publikováno v:
Pharmaceutics, Vol 13, Iss 7, p 1075 (2021)
Despite the apparent advantages for long-term treatment and local therapies against intestinal diseases, the oral delivery of nucleic acids has been challenging due to unfavorable physiological conditions for their stability. In this study, a novel n
Externí odkaz:
https://doaj.org/article/f300fd23b8534282b8cb02b79c794f04
Autor:
Madhu Babu Pasula, Prabhat R. Napit, Abdulrahman Alhamyani, Sagor C. Roy, Paul W. Sylvester, Khaggeswar Bheemanapally, Karen P. Briski
Publikováno v:
Neurochemical Research. 48:404-417
Autor:
Prabhat R. Napit, Abdulrahman Alhamyani, Khaggeswar Bheemanapally, Paul W. Sylvester, Karen P. Briski
Publikováno v:
Neuroglia
Neuroglia; Volume 3; Issue 4; Pages: 144-157
Neuroglia; Volume 3; Issue 4; Pages: 144-157
Astrocyte glycogen is a critical metabolic variable that affects hypothalamic control of glucostasis. Glucocorticoid hormones regulate peripheral glycogen, but their impact on hypothalamic glycogen is not known. A hypothalamic astrocyte primary cultu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c168f7e6880d8040fca37b154fa58423
https://europepmc.org/articles/PMC9850496/
https://europepmc.org/articles/PMC9850496/
Publikováno v:
AIMS Neuroscience
AIMS Neuroscience, Vol 8, Iss 4, Pp 510-525 (2021)
AIMS Neuroscience, Vol 8, Iss 4, Pp 510-525 (2021)
Gamma-aminobutyric acid (GABA) acts on ventromedial hypothalamic targets to suppress counter-regulatory hormone release, thereby lowering blood glucose. Maladaptive up-regulation of GABA signaling is implicated in impaired counter-regulatory outflow