Sustained mucosal colonization and fecal metabolic dysfunction by Bacteroides associates with fecal microbial transplant failure in ulcerative colitis patients.

Autor: Zhang B; Department of Medicine, Division of Gastrointestinal and Liver Diseases, Keck School of Medicine, University of Southern California, Los Angeles, CA, 90033, USA., Magnaye KM; Division of Gastroenterology, Department of Medicine, University of California San Francisco, San Francisco, CA, 94143, USA.; The Benioff Center for Microbiome Medicine, University of California San Francisco, San Francisco, CA, 94143, USA., Stryker E; Division of Gastroenterology, Department of Medicine, University of California San Francisco, San Francisco, CA, 94143, USA., Moltzau-Anderson J; Division of Gastroenterology, Department of Medicine, University of California San Francisco, San Francisco, CA, 94143, USA.; The Benioff Center for Microbiome Medicine, University of California San Francisco, San Francisco, CA, 94143, USA., Porsche CE; Division of Gastroenterology, Department of Medicine, University of California San Francisco, San Francisco, CA, 94143, USA., Hertz S; Department of Infectious Diseases, Aalborg University Hospital, Aalborg, Denmark., McCauley KE; Division of Gastroenterology, Department of Medicine, University of California San Francisco, San Francisco, CA, 94143, USA., Smith BJ; The Gladstone Institutes, Data Science and Biotechnology, San Francisco, CA, 94158, USA., Zydek M; Division of Gastroenterology, Department of Medicine, University of California San Francisco, San Francisco, CA, 94143, USA., Pollard KS; The Gladstone Institutes, Data Science and Biotechnology, San Francisco, CA, 94158, USA.; Department of Epidemiology and Biostatistics, University of California San Francisco, San Francisco, CA, 94158, USA.; Chan Zuckerberg Biohub, San Francisco, CA University of California, San Francisco, CA, 94158, USA., Ma A; Division of Gastroenterology, Department of Medicine, University of California San Francisco, San Francisco, CA, 94143, USA., El-Nachef N; Division of Gastroenterology, Department of Medicine, University of California San Francisco, San Francisco, CA, 94143, USA.; Division of Gastroenterology, Henry Ford Health System, Detroit, MI, 48208, USA., Lynch SV; Division of Gastroenterology, Department of Medicine, University of California San Francisco, San Francisco, CA, 94143, USA. susan.lynch@ucsf.edu.; The Benioff Center for Microbiome Medicine, University of California San Francisco, San Francisco, CA, 94143, USA. susan.lynch@ucsf.edu.

Publikováno v: Scientific reports [Sci Rep] 2024 Aug 09; Vol. 14 (1), pp. 18558. Date of Electronic Publication: 2024 Aug 09.

Precocious infant fecal microbiome promotes enterocyte barrier dysfuction, altered neuroendocrine signaling and associates with increased childhood obesity risk.

Autor: Yong GJM; Division of Gastroenterology, Department of Medicine, University of California, San Francisco, CA, USA.; Asian Microbiome Library Pte Ltd, Singapore and Singapore Institute of Food and Biotechnology Innovation, Singapore, Singapore., Porsche CE; Division of Gastroenterology, Department of Medicine, University of California, San Francisco, CA, USA., Sitarik AR; Department of Public Health Sciences, Henry Ford Health System, Detroit, MI, USA., Fujimura KE; Division of Gastroenterology, Department of Medicine, University of California, San Francisco, CA, USA.; Genetic Disease Laboratory, California Department of Public Health, San Francisco, CA, USA., McCauley K; Division of Gastroenterology, Department of Medicine, University of California, San Francisco, CA, USA., Nguyen DT; Division of Gastroenterology, Department of Medicine, University of California, San Francisco, CA, USA.; Department of Molecular Microbiology and Immunology, Johns Hopkins Bloomberg School of Public Health, Baltimore, MD, USA., Levin AM; Department of Public Health Sciences, Henry Ford Health System, Detroit, MI, USA., Woodcroft KJ; Department of Public Health Sciences, Henry Ford Health System, Detroit, MI, USA., Ownby DR; Division of Allergy and Clinical Immunology, Department of Pediatrics, Augusta University, Augusta, GA, USA., Rundle AG; Department of Epidemiology, Mailman School of Public Health, Columbia University, New York, NY, USA., Johnson CC; Department of Public Health Sciences, Henry Ford Health System, Detroit, MI, USA., Cassidy-Bushrow A; Department of Public Health Sciences, Henry Ford Health System, Detroit, MI, USA., Lynch SV; Division of Gastroenterology, Department of Medicine, University of California, San Francisco, CA, USA.

Publikováno v: Gut microbes [Gut Microbes] 2024 Jan-Dec; Vol. 16 (1), pp. 2290661. Date of Electronic Publication: 2023 Dec 20.

Acute Aerobic Exercise Remodels the Adipose Tissue Progenitor Cell Phenotype in Obese Adults.

Autor: Ludzki AC; Substrate Metabolism Laboratory, School of Kinesiology, University of Michigan, Ann Arbor, MI, United States., Krueger EM; Substrate Metabolism Laboratory, School of Kinesiology, University of Michigan, Ann Arbor, MI, United States., Baldwin TC; Substrate Metabolism Laboratory, School of Kinesiology, University of Michigan, Ann Arbor, MI, United States., Schleh MW; Substrate Metabolism Laboratory, School of Kinesiology, University of Michigan, Ann Arbor, MI, United States., Porsche CE; Department of Pediatrics, Medical School, University of Michigan, Ann Arbor, MI, United States.; Graduate Program in Immunology, Medical School, University of Michigan, Ann Arbor, MI, United States., Ryan BJ; Substrate Metabolism Laboratory, School of Kinesiology, University of Michigan, Ann Arbor, MI, United States., Muir LA; Department of Computational Medicine and Bioinformatics, Medical School, University of Michigan, Ann Arbor, MI, United States., Singer K; Department of Pediatrics, Medical School, University of Michigan, Ann Arbor, MI, United States.; Graduate Program in Immunology, Medical School, University of Michigan, Ann Arbor, MI, United States.; Department of Molecular and Integrative Physiology, Medical School, University of Michigan, Ann Arbor, MI, United States., Lumeng CN; Department of Pediatrics, Medical School, University of Michigan, Ann Arbor, MI, United States.; Graduate Program in Immunology, Medical School, University of Michigan, Ann Arbor, MI, United States.; Department of Molecular and Integrative Physiology, Medical School, University of Michigan, Ann Arbor, MI, United States., Horowitz JF; Substrate Metabolism Laboratory, School of Kinesiology, University of Michigan, Ann Arbor, MI, United States.

Publikováno v: Frontiers in physiology [Front Physiol] 2020 Jul 28; Vol. 11, pp. 903. Date of Electronic Publication: 2020 Jul 28 (Print Publication: 2020).

Adipose tissue dendritic cell signals are required to maintain T cell homeostasis and obesity-induced expansion.

Autor: Porsche CE; Graduate Program in Immunology, University of Michigan Medical School, Ann Arbor, MI, USA., Delproposto JB; Department of Pediatrics and Communicable Diseases, University of Michigan Medical School, Ann Arbor, MI, USA., Patrick E; College of Literature Sciences and Arts, University of Michigan, Ann Arbor, USA., Zamarron BF; Graduate Program in Immunology, University of Michigan Medical School, Ann Arbor, MI, USA., Lumeng CN; Graduate Program in Immunology, University of Michigan Medical School, Ann Arbor, MI, USA; Department of Pediatrics and Communicable Diseases, University of Michigan Medical School, Ann Arbor, MI, USA. Electronic address: clumeng@umich.edu.

Publikováno v: Molecular and cellular endocrinology [Mol Cell Endocrinol] 2020 Apr 05; Vol. 505, pp. 110740. Date of Electronic Publication: 2020 Jan 24.

Adipose Tissue Dendritic Cells Are Independent Contributors to Obesity-Induced Inflammation and Insulin Resistance.

Autor: Cho KW; Department of Pediatrics and Communicable Diseases, University of Michigan Medical School, Ann Arbor, MI 48109.; Soonchunhyang Institute of Medi-bio Science, Soonchunhyang University, Cheonan-si, Chungcheongnam-do, 31151, Korea., Zamarron BF; Department of Pediatrics and Communicable Diseases, University of Michigan Medical School, Ann Arbor, MI 48109.; Graduate Program in Immunology, University of Michigan Medical School, Ann Arbor, MI 48109., Muir LA; Department of Pediatrics and Communicable Diseases, University of Michigan Medical School, Ann Arbor, MI 48109., Singer K; Department of Pediatrics and Communicable Diseases, University of Michigan Medical School, Ann Arbor, MI 48109.; Graduate Program in Immunology, University of Michigan Medical School, Ann Arbor, MI 48109., Porsche CE; Department of Pediatrics and Communicable Diseases, University of Michigan Medical School, Ann Arbor, MI 48109.; Graduate Program in Immunology, University of Michigan Medical School, Ann Arbor, MI 48109., DelProposto JB; Department of Pediatrics and Communicable Diseases, University of Michigan Medical School, Ann Arbor, MI 48109., Geletka L; Department of Pediatrics and Communicable Diseases, University of Michigan Medical School, Ann Arbor, MI 48109., Meyer KA; Department of Surgery, University of Michigan Medical School, Ann Arbor, MI 48109., O'Rourke RW; Department of Surgery, University of Michigan Medical School, Ann Arbor, MI 48109.; Department of Surgery, Ann Arbor Veteran's Administration Hospital, Ann Arbor, MI 48109; and., Lumeng CN; Department of Pediatrics and Communicable Diseases, University of Michigan Medical School, Ann Arbor, MI 48109; clumeng@umich.edu.; Graduate Program in Immunology, University of Michigan Medical School, Ann Arbor, MI 48109.; Molecular and Integrative Physiology, University of Michigan Medical School, Ann Arbor, MI 48109.

Publikováno v: Journal of immunology (Baltimore, Md. : 1950) [J Immunol] 2016 Nov 01; Vol. 197 (9), pp. 3650-3661. Date of Electronic Publication: 2016 Sep 28.