Zobrazeno 1 - 2
of 2
pro vyhledávání: '"Popoli P. 6"'
Autor:
Moresco R.M. 1, Lavazza T. 2, Belloli S. 1, Lecchi M. 5, Pezzola A. 6, Todde S. 1, Matarrese M. 1, Carpinelli A. 1, Turolla E. 1, Zimarino V. 3, Popoli P. 6, Malgaroli A. 2, 4, Fazio F. 1
Publikováno v:
European journal of nuclear medicine and molecular imaging
35 (2008): 704–715.
info:cnr-pdr/source/autori:Moresco R.M. 1, Lavazza T. 2, Belloli S. 1, Lecchi M. 5, Pezzola A. 6, Todde S. 1, Matarrese M. 1, Carpinelli A. 1, Turolla E. 1, Zimarino V. 3, Popoli P. 6, Malgaroli A. 2, 4 and Fazio F. 1/titolo:Quinolinic acid induced neurodegeneration in the striatum: a combined in vivo and in vitro analysis of receptor changes and microglia activation./doi:/rivista:European journal of nuclear medicine and molecular imaging (Print)/anno:2008/pagina_da:704/pagina_a:715/intervallo_pagine:704–715/volume:35
35 (2008): 704–715.
info:cnr-pdr/source/autori:Moresco R.M. 1, Lavazza T. 2, Belloli S. 1, Lecchi M. 5, Pezzola A. 6, Todde S. 1, Matarrese M. 1, Carpinelli A. 1, Turolla E. 1, Zimarino V. 3, Popoli P. 6, Malgaroli A. 2, 4 and Fazio F. 1/titolo:Quinolinic acid induced neurodegeneration in the striatum: a combined in vivo and in vitro analysis of receptor changes and microglia activation./doi:/rivista:European journal of nuclear medicine and molecular imaging (Print)/anno:2008/pagina_da:704/pagina_a:715/intervallo_pagine:704–715/volume:35
Purpose. Huntingtons disease (HD) is a progressive neurodegenerative disorder, which is characterised by prominent neuronal cell loss in the basal ganglia with motor and cognitive disturbances. One of the most well-studied pharmacological models of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=cnr_________::c62a383a19f10ce917fee4d56888b177
https://publications.cnr.it/doc/10364
https://publications.cnr.it/doc/10364
Autor:
De Luca G. 1, Russo M.T. 1, Degan P. 2, Tiveron C. 3, Zijno A. 1, Meccia E. 1, Ventura I. 1, Mattei E. 4, Nakabeppu Y. 5, Crescenzi M. 1, Pepponi R. 6, Pezzola A. 6, Popoli P. 6, Bignami M. 1.
Publikováno v:
PLOS genetics 4(11) (2008): 1–14.
info:cnr-pdr/source/autori:De Luca G. 1#; Russo M.T. 1#; Degan P. 2; Tiveron C. 3; Zijno A. 1; Meccia E. 1; Ventura I. 1; Mattei E. 4; Nakabeppu Y. 5; Crescenzi M. 1; Pepponi R. 6; Pezzola A. 6; Popoli P. 6; Bignami M. 1./titolo:A role for oxidized DNA precursors in Huntington's disease-like striatal neurodegeneration/doi:/rivista:PLOS genetics/anno:2008/pagina_da:1/pagina_a:14/intervallo_pagine:1–14/volume:4(11)
info:cnr-pdr/source/autori:De Luca G. 1#; Russo M.T. 1#; Degan P. 2; Tiveron C. 3; Zijno A. 1; Meccia E. 1; Ventura I. 1; Mattei E. 4; Nakabeppu Y. 5; Crescenzi M. 1; Pepponi R. 6; Pezzola A. 6; Popoli P. 6; Bignami M. 1./titolo:A role for oxidized DNA precursors in Huntington's disease-like striatal neurodegeneration/doi:/rivista:PLOS genetics/anno:2008/pagina_da:1/pagina_a:14/intervallo_pagine:1–14/volume:4(11)
Several human neurodegenerative disorders are characterized by the accumulation of 8-oxo-7,8-dihydroguanine (8-oxodG) in the DNA of affected neurons. This can occur either through direct oxidation of DNA guanine or via incorporation of the oxidized n
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=cnr_________::6d32a7d77da287d3c8c40253faa14cf2
https://publications.cnr.it/doc/4888
https://publications.cnr.it/doc/4888