Zobrazeno 1 - 10
of 137
pro vyhledávání: '"Podkalicka, P."'
Autor:
Olga Mucha, Paulina Podkalicka, Monika Żukowska, Ewelina Pośpiech, Józef Dulak, Agnieszka Łoboda
Publikováno v:
Scientific Reports, Vol 13, Iss 1, Pp 1-17 (2023)
Abstract Skeletal muscle regeneration relies on the reciprocal interaction between many types of cells. Regenerative capacity may be altered in different disorders. In our study, we investigated whether the deletion of miR-378a (miR-378) affects musc
Externí odkaz:
https://doaj.org/article/bb84564b4ad84804a5000fbf158dda0d
Autor:
Olga Mucha, Małgorzata Myszka, Paulina Podkalicka, Bianka Świderska, Agata Malinowska, Józef Dulak, Agnieszka Łoboda
Publikováno v:
Biomolecules, Vol 13, Iss 11, p 1648 (2023)
Mdx mice with a spontaneous mutation in exon 23 of the Dmd gene represent the most common model to investigate the pathophysiology of Duchenne muscular dystrophy (DMD). The disease, caused by the lack of functional dystrophin, is characterized by irr
Externí odkaz:
https://doaj.org/article/b5a527238d524efe87466f14c2ea7a38
Autor:
Paulina Podkalicka, Olga Mucha, Katarzyna Kaziród, Krzysztof Szade, Jacek Stępniewski, Liudmyla Ivanishchuk, Hirofumi Hirao, Ewelina Pośpiech, Alicja Józkowicz, Jerzy W. Kupiec-Weglinski, Józef Dulak, Agnieszka Łoboda
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-19 (2022)
Abstract Although Duchenne muscular dystrophy (DMD) primarily affects muscle tissues, the alterations to systemic metabolism manifested in DMD patients contribute to the severe phenotype of this fatal disorder. We propose that microRNA-378a (miR-378)
Externí odkaz:
https://doaj.org/article/31f8767de0804f9ea3cdfb34755154d9
Autor:
Olga Mucha, Paulina Podkalicka, Katarzyna Kaziród, Emilia Samborowska, Józef Dulak, Agnieszka Łoboda
Publikováno v:
Skeletal Muscle, Vol 11, Iss 1, Pp 1-16 (2021)
Abstract Background Duchenne muscular dystrophy (DMD) is an incurable disease, caused by the mutations in the DMD gene, encoding dystrophin, an actin-binding cytoskeletal protein. Lack of functional dystrophin results in muscle weakness, degeneration
Externí odkaz:
https://doaj.org/article/60466cde0c564a7bb16d8020398b93f5
Autor:
Alexa Färber, Aneta Podkalicka
Publikováno v:
Culture Unbound: Journal of Current Cultural Research, Vol 11, Iss 3-4, Pp 421-442 (2020)
Concepts of thrift and dwelling are central to how societies live together. Thrift refers to a complex and morally-loaded set of economic practices that people engage with out of necessity, choice, or both. Whilst home-making or dwelling refers to so
Externí odkaz:
https://doaj.org/article/3aae8d9088e94519b50d8ec57176876c
Autor:
Iwona Bronisz-Budzyńska, Katarzyna Chwalenia, Olga Mucha, Paulina Podkalicka, Karolina-Bukowska-Strakova, Alicja Józkowicz, Agnieszka Łoboda, Magdalena Kozakowska, Józef Dulak
Publikováno v:
Skeletal Muscle, Vol 9, Iss 1, Pp 1-17 (2019)
Abstract Duchenne muscular dystrophy (DMD) is a genetic disease evoked by a mutation in the dystrophin gene. It is associated with progressive muscle degeneration and increased inflammation. Up to this date, mainly anti-inflammatory treatment is avai
Externí odkaz:
https://doaj.org/article/175c5c5bbe5442d28ac094fd7588e154
Akademický článek
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Autor:
Melissa Dewulf, Darius Vasco Köster, Bidisha Sinha, Christine Viaris de Lesegno, Valérie Chambon, Anne Bigot, Mona Bensalah, Elisa Negroni, Nicolas Tardif, Joanna Podkalicka, Ludger Johannes, Pierre Nassoy, Gillian Butler-Browne, Christophe Lamaze, Cedric M. Blouin
Publikováno v:
Nature Communications, Vol 10, Iss 1, Pp 1-13 (2019)
Caveolae are mechanosensors and mutations of their coat proteins are implicated in muscle disorders, but molecular mechanisms are unclear. Here, the authors show that caveolae can regulate IL6/STAT3 signaling in muscle cells under stress, and that dy
Externí odkaz:
https://doaj.org/article/af0a410ef64d4e98b3b78ef974859a6d
Autor:
Olga Mucha, Katarzyna Kaziród, Paulina Podkalicka, Kinga Rusin, Józef Dulak, Agnieszka Łoboda
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 1, p 470 (2021)
Dysregulation of autophagy may contribute to the progression of various muscle diseases, including Duchenne muscular dystrophy (DMD). Heme oxygenase-1 (HO-1, encoded by Hmox1), a heme-degrading enzyme, may alleviate symptoms of DMD, inter alia, throu
Externí odkaz:
https://doaj.org/article/89f848ba6b4d4c1e811f4e40656ca3b0
Akademický článek
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K zobrazení výsledku je třeba se přihlásit.
K zobrazení výsledku je třeba se přihlásit.