Overexpression of Malat1 drives metastasis through inflammatory reprogramming of the tumor microenvironment.

Autor: Martinez-Terroba E; Department of Molecular, Cellular, and Developmental Biology, Yale University, New Haven, CT 06511, USA., Plasek-Hegde LM; Department of Molecular, Cellular, and Developmental Biology, Yale University, New Haven, CT 06511, USA., Chiotakakos I; Department of Molecular, Cellular, and Developmental Biology, Yale University, New Haven, CT 06511, USA., Li V; Department of Molecular, Cell, and Developmental Biology, University of California, Los Angeles, CA 90095, USA., de Miguel FJ; Yale Cancer Center, Yale University, New Haven, CT 06511, USA., Robles-Oteiza C; Departments of Pathology and Internal Medicine (Section of Medical Oncology), Yale School of Medicine, New Haven, CT 06511, USA., Tyagi A; Yale Center for Genome Analysis, Yale University, New Haven, CT 06516, USA., Politi K; Yale Cancer Center, Yale University, New Haven, CT 06511, USA.; Departments of Pathology and Internal Medicine (Section of Medical Oncology), Yale School of Medicine, New Haven, CT 06511, USA., Zamudio JR; Department of Molecular, Cell, and Developmental Biology, University of California, Los Angeles, CA 90095, USA., Dimitrova N; Department of Molecular, Cellular, and Developmental Biology, Yale University, New Haven, CT 06511, USA.; Yale Cancer Center, Yale University, New Haven, CT 06511, USA.

Publikováno v: Science immunology [Sci Immunol] 2024 Jun 14; Vol. 9 (96), pp. eadh5462. Date of Electronic Publication: 2024 Jun 14.