Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Pinggui Gong"'
Publikováno v:
HeadneckREFERENCES. 44(10)
This study explored whether laryngeal carcinoma could be divided into different subtypes based on molecular differences using a molecular subtype-prediction model.We extracted data from the Cancer Genome Atlas and Gene Expression Omnibus databases an
BackgroundThe phosphatase and tensin homolog gene(PTEN) is a crucial cancersuppressor gene in nasopharyngeal carcinoma(NPC),which encodes the protein i-ncluding lipid phosphatase and protein phosphatase.p-P38 plays a vital role in the development pro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e06547c298f376c84677185038fd68ad
https://doi.org/10.21203/rs.3.rs-34333/v1
https://doi.org/10.21203/rs.3.rs-34333/v1
Publikováno v:
Biochemical and biophysical research communications. 514(1)
Objective Early malignant transformation of nasopharyngeal carcinoma(NPC) is associated with Epstein-Barr virus(EBV) infection and telomerase activation. The EBV latent membrane protein 1(LMP1) regulates expression of various genes by triggering NF-
Autor:
Junjun Zhang, Shusheng Gong, Pinggui Gong, Tengfei Qu, Aihui Yan, Ke Liu, Tonghui Ding, Yunyi Liu, Le Yang, Daishi Chen, Shiming Yang, Hong Peng
Publikováno v:
Acta oto-laryngologica. 138(1)
To investigate whether there are more quantitative pre-synaptic ribbons formed in the cochlear region corresponding to middle-frequency in cochlea of mice.Counts of pre-synaptic ribbons were performed using immunostaining and laser confocal microscop
Publikováno v:
Journal of Translational Medicine
Background The receptor for activated C kinase 1 (RACK1) is involved in various cancers, but its roles in nasopharyngeal carcinoma (NPC) have not yet been fully elucidated. Methods Initially, RACK1 expression was analyzed by immunohistochemistry in N
Autor:
Yuxiang Chen, Xiaoming Lyu, Jinbang Li, Kunping Liu, Qiang Jiang, Kaitai Yao, Longmei Cai, Jianguo Wang, Juan Lu, Hong Peng, Pinggui Gong, Xin Li
Publikováno v:
Oncotarget
Phosphatase and tensin homolog (PTEN) is a major tumor suppressor and usually silenced via the deletion, insertion and mutation. We previously discovered its inactivation via aberrant CpG island methylation. Here, we provide further evidence that EBV