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Publikováno v:
Neurobiology of aging 40, 98-102 (2016). doi:10.1016/j.neurobiolaging.2016.01.008
Soluble forms of oligomeric amyloid beta (AβO) are involved in the loss of synaptic plasticity and memory, especially in early phases of Alzheimer's disease. Stimulation of dopamine D1/D5 receptors (D1R/D5R) is known to increase surface expression o