Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Pichili V B, Reddy"'
Autor:
Venkata Subba Rao Atluri, Sudheesh P Kanthikeel, Pichili V B Reddy, Adriana Yndart, Madhavan P N Nair
Publikováno v:
PLoS ONE, Vol 8, Iss 4, p e61399 (2013)
HIV-associated neurocognitive disorders (HAND) is characterized by development of cognitive, behavioral and motor abnormalities, and occur in approximately 50% of HIV infected individuals. Our current understanding of HAND emanates mainly from HIV-1
Externí odkaz:
https://doaj.org/article/31f1b9f73f3a42eb9c22e0e3c9f47dcf
Autor:
Pichili V. B. Reddy, Kevin M. Curtis, Kakulavarapu V. Rama Rao, Xiaoying Tong, Michael D. Norenberg, Arumugam R. Jayakumar
Publikováno v:
Glia. 58:1490-1499
Manganese in excess is neurotoxic and causes CNS injury resembling that of Parkinson's disease. In brain, astrocytes predominantly take up and accumulate manganese and are thus vulnerable to its toxicity. Manganese was shown to induce cell swelling i
Publikováno v:
The American Journal of Pathology. 176:1400-1408
Brain edema and the associated increase in intracranial pressure are potentially lethal complications of acute liver failure (ALF). Astrocyte swelling (cytotoxic edema) represents a significant component of the brain edema in ALF, and elevated blood
Publikováno v:
Journal of Neuroscience Research. 87:2677-2685
Ammonia is the principal neurotoxin implicated in the pathogenesis of hepatic encephalopathy, and astrocytes are the neural cells predominantly affected in this condition. Astrocyte swelling (cytotoxic edema) represents a critical component of the br
Autor:
Pichili V. B. Reddy, Michael D. Norenberg, Ramugounder Ramakrishnan, Arumugam R. Jayakumar, Bliss Forbush, Mitsuaki Moriyama, Mingli Liu
Publikováno v:
Journal of Biological Chemistry. 283:33874-33882
Brain edema and the consequent increase in intracranial pressure and brain herniation are major complications of acute liver failure (fulminant hepatic failure) and a major cause of death in this condition. Ammonia has been strongly implicated as an
Publikováno v:
Laboratory Investigation. 88:816-830
Copper is an essential element and an integral component of various enzymes. However, excess copper is neurotoxic and has been implicated in the pathogenesis of Wilson's disease, Alzheimer's disease, prion conditions, and other disorders. Although me
Publikováno v:
Journal of neurotrauma. 28(3)
The development of cytotoxic brain edema resulting in increased intracranial pressure is a major cause of death occurring in the early phase of traumatic brain injury (TBI). Such edema predominantly develops as a consequence of astrocyte swelling. We
Autor:
Kakulavarapu V Rama, Rao, Arumugam R, Jayakumar, Pichili V B, Reddy, Xiaoying, Tong, Kevin M, Curtis, M D, Norenberg
Publikováno v:
Glia. 58(12)
Manganese in excess is neurotoxic and causes CNS injury resembling that of Parkinson's disease. In brain, astrocytes predominantly take up and accumulate manganese and are thus vulnerable to its toxicity. Manganese was shown to induce cell swelling i
Autor:
K. V. Rama Rao, Pichili V. B. Reddy, Kiran S. Panickar, Mitsuaki Moriyama, Michael D. Norenberg, Arumugam R. Jayakumar
Publikováno v:
Journal of neuropathology and experimental neurology. 67(5)
Brain edema and associated increased intracranial pressure are major consequences of traumatic brain injury that account for most early deaths after traumatic brain injury. An important component of brain edema after traumatic brain injury is astrocy
Autor:
Anne P. Sinke, Pichili V. B. Reddy, Arumugam R. Jayakumar, Kiran S. Panickar, Michael D. Norenberg, Mitsuaki Moriyama
Publikováno v:
Journal of Neurochemistry, 106, 2302-11
Journal of Neurochemistry, 106, 6, pp. 2302-11
Journal of Neurochemistry, 106, 6, pp. 2302-11
Item does not contain fulltext Astrocyte swelling and brain edema are major neuropathological findings in the acute form of hepatic encephalopathy (fulminant hepatic failure), and substantial evidence supports the view that elevated brain ammonia lev
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7083887b9e39566058c803c13694c918
http://hdl.handle.net/2066/70884
http://hdl.handle.net/2066/70884