Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Phoebe S. Lee"'
Autor:
Phoebe S. Lee, Jun Fang, Lea Jessop, Timothy Myers, Preethi Raj, Nan Hu, Chaoyu Wang, Philip R. Taylor, Jianjun Wang, Javed Khan, Maria Jasin, Stephen J. Chanock
Publikováno v:
Breast Cancer: Basic and Clinical Research, Vol 2014, Iss 8, Pp 135-144 (2014)
Externí odkaz:
https://doaj.org/article/aaada8e992454f288108a2429929f0a2
Autor:
Phoebe S. Lee, Jun Fang, Lea Jessop, Timothy Myers, Preethi Raj, Nan Hu, Chaoyu Wang, Philip R. Taylor, Jianjun Wang, Javed Khan, Maria Jasin, Stephen J. Chanock
Publikováno v:
Breast Cancer: Basic and Clinical Research, Vol 8 (2014)
Common genetic variants mapping to two distinct regions of RADS1B , a paralog of RADS1 , have been associated with breast cancer risk in genome-wide association studies (GWAS). RADS1B is a plausible candidate gene because of its established role in t
Externí odkaz:
https://doaj.org/article/0ea540d79eef40f18fbba5bfe7f1d86f
Autor:
Timothy G. Myers, Phoebe S. Lee, Lea Jessop, Stephen J. Chanock, Jun Fang, Chaoyu Wang, Javed Khan, Nan Hu, Jianjun Wang, Maria Jasin, Preethi S. Raj, Philip R. Taylor
Publikováno v:
Breast Cancer : Basic and Clinical Research
Breast Cancer: Basic and Clinical Research, Vol 2014, Iss 8, Pp 135-144 (2014)
Breast Cancer: Basic and Clinical Research, Vol 2014, Iss 8, Pp 135-144 (2014)
Common genetic variants mapping to two distinct regions of RADS1B, a paralog of RADS1, have been associated with breast cancer risk in genome-wide association studies (GWAS). RADS1B is a plausible candidate gene because of its established role in the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7638d6c6b42feac9d995d808216ab104
http://europepmc.org/articles/PMC4213955
http://europepmc.org/articles/PMC4213955
Autor:
Phoebe S. Lee, Thomas D. Petes
Publikováno v:
Proceedings of the National Academy of Sciences. 107:7383-7388
In a previous study, we mapped spontaneous mitotic reciprocal crossovers (RCOs) in a 120-kb interval of chromosome V of Saccharomyces cerevisiae . About three-quarters of the crossovers were associated with gene conversion tracts. About 40% of these
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::92e12fadf7affe6916c1b47b9b44a83f
https://doi.org/10.1073/pnas.1001940107
https://doi.org/10.1073/pnas.1001940107
Autor:
Regina G. Ziegler, Gilles Thomas, Rebecca D. Jackson, Louise A. Brinton, Stephen J. Chanock, Phoebe S. Lee, Christine D. Berg, Jolanta Lissowska, Amy Hutchinson, Charles Kooperberg, Yi-Ping Fu, Kevin B. Jacobs, Jonine D. Figueroa, Ross L. Prentice, Heather Spencer Feigelson, Catherine A. McCarty, Zhaoming Wang, Joseph F. Fraumeni, Peter Kraft, Meredith Yeager, Nilanjan Chatterjee, Marie Josephe Horner, Margaret A. Tucker, David J. Hunter, Ludmila Prokunina-Olsson, Michael J. Thun, Saundra S. Buys, Robert N. Hoover, Montserrat Garcia-Closas, Rowan T. Chlebowski, Ryan Diver, Jesus Gonzalez-Bosquet, Susan E. Hankinson, Beata Peplonska
Publikováno v:
Human genetics. 131(3)
In the National Cancer Institute Cancer Genetic Markers of Susceptibility (CGEMS) genome-wide association study of breast cancer, a single nucleotide polymorphism (SNP) marker, rs999737, in the 14q24.1 interval, was associated with breast cancer risk
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8d19e0b2a816558b3f5fca347b3ae78d
https://pubmed.ncbi.nlm.nih.gov/21959381
https://pubmed.ncbi.nlm.nih.gov/21959381
Autor:
Phoebe S. Lee, Malgorzata Gawel, Margaret Dominska, Patricia W. Greenwell, Thomas D. Petes, Monica L. Hamilton
Publikováno v:
PLoS Genetics
PLoS Genetics, Vol 5, Iss 3, p e1000410 (2009)
PLoS Genetics, Vol 5, Iss 3, p e1000410 (2009)
Homologous recombination is an important mechanism for the repair of DNA damage in mitotically dividing cells. Mitotic crossovers between homologues with heterozygous alleles can produce two homozygous daughter cells (loss of heterozygosity), whereas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::05a78291ffcff35b7f8ac50dd791d4dd
https://doi.org/10.1371/journal.pgen.1000410
https://doi.org/10.1371/journal.pgen.1000410