Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Philip Zisman"'
Autor:
Nissim Arish, Pazit Y Cohen, Regina Golan-Gerstl, Zvi Fridlender, Mark Richter Dayan, Philip Zisman, Raphael Breuer, Shulamit B Wallach-Dayan
Publikováno v:
PLoS ONE, Vol 10, Iss 5, p e0126730 (2015)
High doses of bleomycin administered to patients with lymphomas and other tumors lead to significant lung toxicity in general, and to apoptosis of epithelial cells, in particular. Apoptosis of alveolar epithelium is an important step in the pathogene
Externí odkaz:
https://doaj.org/article/564f91e5276a42b3bc512159f78de243
Publikováno v:
Mediators of Inflammation, Vol 2015 (2015)
Lung fibrosis is characterized by abnormal accumulation of fibroblasts in the interstitium of the alveolar space. Two populations of myofibroblasts, distinguished by Thy1 expression, are detected in human and murine lungs. Accumulation of Thy1-negati
Externí odkaz:
https://doaj.org/article/0e4fd347295c43db80524e8cf1432bb1
Autor:
Philip Zisman, Mark Richter Dayan, Jenya Konikov, Liron Elkayam, Shulamit B. Wallach-Dayan, Nissim Arish, Regina Golan-Gerstl, Raphael Breuer
Publikováno v:
Journal of Autoimmunity. 59:67-76
Immune cells, particularly those expressing the ligand of the Fas-death receptor (FasL), e.g. cytotoxic T cells, induce apoptosis in ‘undesirable’ self- and non-self-cells, including lung fibroblasts, thus providing a means of immune surveillance
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ea9ea83419867e29e4304dccba7e4d8d
https://doi.org/10.1016/j.jaut.2015.02.006
https://doi.org/10.1016/j.jaut.2015.02.006
Autor:
Ronald H. Goldstein, Wellington V. Cardoso, Philip Zisman, Shulamit B. Wallach-Dayan, Regina Golan-Gerstl, Raphael Breuer
Publikováno v:
American journal of respiratory cell and molecular biology. 47(3)
A prominent feature of fibrotic tissue in general and of lungs in particular is fibroblast proliferation and accumulation. In patients overcoming fibrosis, apoptosis limits this excessive cell growth. We have previously shown resistance to Fas-induce
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::11b35a51ee4539ee539d0a7572d514a4
https://pubmed.ncbi.nlm.nih.gov/22582174
https://pubmed.ncbi.nlm.nih.gov/22582174
Autor:
S.Z. Ben Sasson, Michal Baniyash, Nina Fainstein, Ilan Vaknin, Tamir Ben-Hur, Philip Zisman, Ofira Einstein
Publikováno v:
Molecular and cellular neurosciences. 39(3)
Intravenous neural precursor cell (NPCs) injection attenuates experimental autoimmune encephalomyelitis by reducing autoreactive T cell encephalitogenicity in lymph nodes in vivo. Here we examined NPC-lymphocyte interactions in vitro. NPCs inhibited
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a411d89dfeb916bc80b8affae30149c1
https://pubmed.ncbi.nlm.nih.gov/18692138
https://pubmed.ncbi.nlm.nih.gov/18692138
Autor:
Zvi G. Fridlender, Mark Richter Dayan, Pazit Y. Cohen, Philip Zisman, Nissim Arish, Shulamit B. Wallach-Dayan, Regina Golan-Gerstl, Raphael Breuer
Publikováno v:
PLoS ONE, Vol 10, Iss 5, p e0126730 (2015)
PLoS ONE
PLoS ONE
High doses of bleomycin administered to patients with lymphomas and other tumors lead to significant lung toxicity in general, and to apoptosis of epithelial cells, in particular. Apoptosis of alveolar epithelium is an important step in the pathogene
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::042ecf0ef10999d84ee7325fc2eefb5a
https://doi.org/10.1371/journal.pone.0126730
https://doi.org/10.1371/journal.pone.0126730
Autor:
Tal Lerer, Hannah Cohen, Yuval Dor, Miri Stolovich-Rain, Oded Meyuhas, Igor Ruvinsky, Tomer Nir, Nitzan Sharon, Philip Zisman
Publikováno v:
Genesdevelopment. 19(18)
The regulated phosphorylation of ribosomal protein (rp) S6 has attracted much attention since its discovery in 1974, yet its physiological role has remained obscure. To directly address this issue, we have established viable and fertile knock-in mice
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::df84a5df37c00259c558ddf28671f35f
https://pubmed.ncbi.nlm.nih.gov/16166381
https://pubmed.ncbi.nlm.nih.gov/16166381