HFE gene variants and iron induced O2 radical generation in idiopathic pulmonary fibrosis

Autor: Sangiuolo F. (1), Puxeddu E. (1), Pezzuto G. (2), Cavalli F. (3), Longo G. (1), Comandini A. (2), Di Pierro D. (4), Pallante M. (1), Sergiacomi G. (5), Simonetti G. (5), Zompatori M. (6), Orlandi A. (7), Magrini A. (1), Amicosante M. (1), Mariani F. (8), Losi M. (2), Fraboni D. (9), Bisetti A. (10), Saltini C. (1, 2, 3)

Publikováno v: European respiratory journal (Online) 0 (2015): 1–8. doi:10.1183/09031936.00104814
info:cnr-pdr/source/autori:Sangiuolo F. (1); Puxeddu E. (1); Pezzuto G. (2); Cavalli F. (3); Longo G. (1); Comandini A. (2); Di Pierro D. (4); Pallante M. (1); Sergiacomi G. (5); Simonetti G. (5); Zompatori M. (6); Orlandi A. (7); Magrini A. (1); Amicosante M. (1); Mariani F. (8); Losi M. (2); Fraboni D. (9); Bisetti A. (10); Saltini C. (1, 2, 3)/titolo:HFE gene variants and iron induced O2 radical generation in idiopathic pulmonary fibrosis/doi:10.1183%2F09031936.00104814/rivista:European respiratory journal (Online)/anno:2015/pagina_da:1/pagina_a:8/intervallo_pagine:1–8/volume:0

In idiopathic pulmonary fibrosis (IPF), lung accumulation of excessive extracellular iron and macrophage haemosiderin may suggest disordered iron homeostasis leading to recurring microscopic injury and fibrosing damage. The current study population c

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http://www.cnr.it/prodotto/i/307233