Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Petter Järemo"'
Publikováno v:
Annals of Medicine, Vol 55, Iss 1, Pp 1102-1110 (2023)
AbstractBackground/objective Type 2 diabetes is a major risk factor for atherosclerotic disease. It is well agreed that the reactivity of diabetic platelets is increased but how platelet reactivity regulates is unknown. In our laboratory, density sep
Externí odkaz:
https://doaj.org/article/41e07d64a34149d68999bc3385bf390a
Publikováno v:
Thrombosis research. 195
Background Strong agonist provocation in vitro creates small procoagulant platelets characterized by down-regulated fibrinogen receptors as judged from surface αIIbβ3 activation specific antibody (PAC-1). They further show increased surface Annexin
Autor:
Vesna Jelic, Petter Järemo, Homira Behbahani, Bengt Winblad, Alenka Jejcic, Magnus Oweling, Tasmin Shahnaz
Publikováno v:
Current Alzheimer research. 16(9)
Background: Alzheimer’s Disease (AD) features the accumulation of β-amyloid in erythrocytes. The subsequent red cell damage may well affect their oxygen-carrying capabilities. 2,3- diphosphoglycerate (2,3-DPG) binds to the hemoglobin thereby promo
Autor:
Petter Järemo, Bengt Winblad, Magnus Oweling, Tasmin Shahnaz, Homira Behbahani, Vesna Jelic, Alenka Jejcic
Publikováno v:
Current Alzheimer research. 16(11)
Introduction: We recently showed that Amyloid Beta (Aβ)40 accumulates in erythrocytes and possibly causes cell damage as evidenced by an increased number of assumed injured low-density (kg/L) erythrocytes. Furthermore, we have suggested a separation
Autor:
Vesna Jelic, Rudolf Rigler, Petter Järemo, Jüri Jarvet, Nenad Bogdanovic, Lars Terenius, Astrid Gräslund, Vladana Vukojević, Ann Tiiman
Publikováno v:
Journal of Alzheimer's Disease
Background: Biomarkers are central to current research on molecular mechanisms underlying Alzheimer’s disease (AD). Their further development is of paramount importance for understanding pathophysiological processes that eventually lead to disease
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b4073252a03baf5d129d62fcafaf92b1
http://hdl.handle.net/10852/74201
http://hdl.handle.net/10852/74201
Autor:
Vesna Jelic, Magnus Oweling, Tasmin Shahnaz, Micha Milovanovic, Staffan Nilsson, Bengt Winblad, Homira Behbahani, Petter Järemo
Publikováno v:
Platelets. 28:182-186
Previous work indicates that erythrocytes (RBCs) accumulate β-amyloid X-40 (Aβ40) in individuals with Alzheimer disease (AD) and to a lesser extent in healthy elderly. The toxin damages RBCs and increases their mean corpuscular volume (MCV). Furthe
Autor:
Staffan Nilsson, Micha Milovanovic, Bengt Winblad, Claes Post, Petter Järemo, Tomas L. Lindahl, Kristoffer Eriksson
Publikováno v:
Clinical Biochemistry. 47:51-53
Introduction Alzheimer's disease (AD) is a progressive form of dementia characterized by an increase in the toxic substance β-amyloid in the brain. Platelets display a substantial heterogeneity with respect to density. They further contain a substan
Autor:
Homira Behbahani, Jan Kehr, Vesna Jelic, Walid Tajeddinn, Takashi Yoshitake, Micha Milovanovic, Dag Aarsland, Angel Cedazo-Minguez, Tasmin Shahnaz, Seyed-Mohammad Fereshtehnejad, Kina Höglund, Petter Järemo, Bengt Winblad, Mohammed Seed Ahmed
Publikováno v:
Journal of Alzheimer's disease : JAD. 53(2)
Introduction Serotonin (5-HT) is involved in the pathology of Alzheimer's disease (AD). Objective We aimed to measure 5-HT level in platelets in AD and explore its association with cerebrospinal fluid (CSF), AD biomarkers (amyloid-β 1-42 (Aβ42), to
Publikováno v:
Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association. 25(8)
Rationale According to literature, the inflammatory response and platelets are associated with coronary heart disease mortality. In this study, we examine if similar relationships exist after acute cerebral infarctions. Design Between 2005 and 2007,
Autor:
Petter Järemo, Staffan Nilsson
Publikováno v:
BMC Family Practice
Background: Chest pain assumed to be of non-coronary origin (NCCP) may be linked to enhanced mortality due to coronary heart disease (CHD). The aim of this study was to follow NCCP patients, as defined in primary care, with respect to mortality and l
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::24b95618859852f85123809ad8bc742c
http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-133110
http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-133110