Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Peter S. Rowe"'
Autor:
Kyle P. Jansson, Jordan Kuluva, Shiqin Zhang, Taylor Swanson, Yan Zhang, Kurt A. Zimmerman, Timothy A. Fields, Darren P. Wallace, Peter S. Rowe, Jason R. Stubbs
Publikováno v:
Physiological Reports, Vol 12, Iss 17, Pp n/a-n/a (2024)
Abstract Osteopontin (OPN) is a multi‐functional glycoprotein that coordinates the innate immune response, prevents nanocrystal formation in renal tubule fluid, and is a biomarker for kidney injury. OPN expression is markedly increased in cystic ep
Externí odkaz:
https://doaj.org/article/15be34ec754f420ba0668b95d8c7b8af
Autor:
Katherine I. Swenson-Fields, Christopher J. Ward, Micaila E. Lopez, Shaneann Fross, Anna L. Heimes Dillon, James D. Meisenheimer, Adib J. Rabbani, Emily Wedlock, Malay K. Basu, Kyle P. Jansson, Peter S. Rowe, Jason R. Stubbs, Darren P. Wallace, Michael P. Vitek, Timothy A. Fields
Publikováno v:
Frontiers in Molecular Biosciences, Vol 9 (2022)
We and others have previously shown that the presence of renal innate immune cells can promote polycystic kidney disease (PKD) progression. In this study, we examined the influence of the inflammasome, a key part of the innate immune system, on PKD.
Externí odkaz:
https://doaj.org/article/9c08d54c8b8b492abf160be7de40df31
Autor:
Jason R. Stubbs, Shiqin Zhang, Kyle P. Jansson, Timothy A. Fields, Joseph Boulanger, Shiguang Liu, Peter S. Rowe
Publikováno v:
Kidney360
Nephron loss dramatically increases tubular phosphate to concentrations that exceed supersaturation. Osteopontin (OPN) is a matricellular protein that enhances mineral solubility in solution; however, the role of OPN in maintaining urinary phosphate
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2d5e5a8a05ec13c14f7536184ec7329f
https://doi.org/10.34067/kid.0007352021
https://doi.org/10.34067/kid.0007352021
Autor:
Benjamin Salmon, Claire Bardet, Mayssam Khaddam, Jiar Naji, Benjamin R Coyac, Brigitte Baroukh, Franck Letourneur, Julie Lesieur, Franck Decup, Dominique Le Denmat, Antonino Nicoletti, Anne Poliard, Peter S Rowe, Eric Huet, Sibylle Opsahl Vital, Agnès Linglart, Marc D McKee, Catherine Chaussain
Publikováno v:
PLoS ONE, Vol 8, Iss 2, p e56749 (2013)
Mutations in PHEX (phosphate-regulating gene with homologies to endopeptidases on the X-chromosome) cause X-linked familial hypophosphatemic rickets (XLH), a disorder having severe bone and tooth dentin mineralization defects. The absence of function
Externí odkaz:
https://doaj.org/article/820187aed9344615a2c61876bf69651a