Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Peta L. S. Reeves"'
Autor:
Anthony W. Purcell, Casper G. Schalkwijk, Rochelle Ayala, David Briskey, Jean L.J.M. Scheijen, Josephine M. Forbes, Raymond J. Steptoe, Kai Lin Giam, Sherman Leung, Domenica A. McCarthy, Brooke E. Harcourt, Micheal S. Ward, Peta L. S. Reeves, Amelia K. Fotheringham, Pouya Faridi, Danielle J. Borg, Nadine L. Dudek
Publikováno v:
Metabolites
Volume 11
Issue 7
Metabolites, Vol 11, Iss 426, p 426 (2021)
Metabolites, 11(7):426. Multidisciplinary Digital Publishing Institute (MDPI)
Volume 11
Issue 7
Metabolites, Vol 11, Iss 426, p 426 (2021)
Metabolites, 11(7):426. Multidisciplinary Digital Publishing Institute (MDPI)
Mechanisms by which advanced glycation end products (AGEs) contribute to type 1 diabetes (T1D) pathogenesis are poorly understood. Since life-long pharmacotherapy with alagebrium chloride (ALT) slows progression to experimental T1D, we hypothesized t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::56a32c3a88149b4349c0a51a2c8471c0
http://europepmc.org/articles/PMC8305727
http://europepmc.org/articles/PMC8305727
Autor:
Peta L. S. Reeves, Emma E. Hamilton-Williams, F. Susan Wong, Raymond J. Steptoe, Rajeev Rudraraju
Publikováno v:
European Journal of Immunology. 47:1550-1561
Type 1 diabetes (T1D) results from autoimmune destruction of insulin-producing pancreatic β cells. Therapies need to incorporate strategies to overcome the genetic defects that impair induction or maintenance of peripheral T-cell tolerance and contr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::41f5e87053a3c2f626044b16a08ad967
https://doi.org/10.1002/eji.201747089
https://doi.org/10.1002/eji.201747089
Autor:
Emma E. Hamilton-Williams, Xiao Liu, Rajeev Rudraraju, Peta L. S. Reeves, Raymond J. Steptoe, F. Susan Wong
Type 1 diabetes (T1D) results from T-cell-mediated autoimmune destruction of pancreatic β cells. Effector T-cell responses emerge early in disease development and expand as disease progresses. Following β-cell destruction, a long-lived T-cell memor
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fa441fc80289d98734b757b6a54ea653
https://orca.cardiff.ac.uk/id/eprint/105269/1/SteptoeICBmerged_1496663775.pdf
https://orca.cardiff.ac.uk/id/eprint/105269/1/SteptoeICBmerged_1496663775.pdf
Autor:
Peta L S, Reeves, Rajeev, Rudraraju, F Susan, Wong, Emma E, Hamilton-Williams, Raymond J, Steptoe
Publikováno v:
European journal of immunology. 47(9)
Type 1 diabetes (T1D) results from autoimmune destruction of insulin-producing pancreatic β cells. Therapies need to incorporate strategies to overcome the genetic defects that impair induction or maintenance of peripheral T-cell tolerance and contr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::481da8df2f4d00174a175dfce834521c
https://pubmed.ncbi.nlm.nih.gov/28665492
https://pubmed.ncbi.nlm.nih.gov/28665492
Publikováno v:
Journal of autoimmunity. 72
Reestablishment of immune tolerance to the insulin-producing beta cells is the desired goal for type 1 diabetes (T1D) treatment and prevention. Immune tolerance to multiple islet antigens is defective in individuals with T1D, but the mechanisms invol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f55d25132faf79223bcb280ef22b58e3
https://pubmed.ncbi.nlm.nih.gov/27255733
https://pubmed.ncbi.nlm.nih.gov/27255733