Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Perla Ugalde-Muñiz"'
Autor:
Roxana Noriega‐Navarro, Ricardo Jesús Martínez‐Tapia, Rubén González‐Rivera, Alicia Ochoa‐Sánchez, Julio César Abarca‐Magaña, Lucía Landa‐Navarro, Verónica Rodríguez‐Mata, Perla Ugalde‐Muñiz, Armando Pérez‐Torres, Abraham Landa, Luz Navarro
Publikováno v:
Brain and Behavior, Vol 13, Iss 6, Pp n/a-n/a (2023)
Abstract Introduction Traumatic brain injury (TBI) is a public health concern with limited treatment options because it causes a cascade of side effects that are the leading cause of hospital death. Thioredoxin is an enzyme with neuroprotective prope
Externí odkaz:
https://doaj.org/article/bb1927bcc15c4930b7d5ec94a5a59b5b
Autor:
Perla Ugalde-Muñiz, María Guadalupe Hernández-Luna, Stephany García-Velasco, Rafael Lugo-Huitrón, Jimena Murcia-Ramírez, Ricardo Jesus Martínez-Tapia, Roxana Noriega-Navarro, Luz Navarro
Publikováno v:
Frontiers in Neuroscience, Vol 16 (2022)
The proinflammatory state, which may be induced by sleep deprivation, seems to be a determining factor in the development of neurodegenerative processes. Investigations of mechanisms that help to mitigate the inflammatory effects of sleep disorders a
Externí odkaz:
https://doaj.org/article/c7eb50253295415c8cdddf41ee6ba426
Autor:
Ricardo Jesus Martinez-Tapia, Francisco Estrada-Rojo, Teresita Guadalupe Lopez-Aceves, Veronica Rodríguez-Mata, Armando Perez-Torres, Antonio Barajas-Martinez, Stephany Garcia-Velasco, Perla Ugalde-Muñiz, Luz Navarro
Publikováno v:
Frontiers in Neuroscience, Vol 14 (2020)
Traumatic brain injury (TBI) induces two types of brain damage: primary and secondary. Damage initiates a series of pathophysiological processes, such as metabolic crisis, excitotoxicity with oxidative stress-induced damage, and neuroinflammation. Th
Externí odkaz:
https://doaj.org/article/56b4d20a60a1496fa7bdc63ef8586c9b
Autor:
null Roxana Noriega‐Navarro, null Ricardo Jesús Martínez‐Tapia, null Rubén González‐Rivera, null Alicia Ochoa‐Sánchez, null Julio César Abarca‐Magaña, null Lucía Landa‐Navarro, null Verónica Rodríguez‐Mata, null Perla Ugalde‐Muñiz, null Armando Pérez‐Torres, null Abraham Landa, null Luz Navarro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1a95aed18bb66beb93fb72542838ad56
https://doi.org/10.1002/brb3.3031/v2/response1
https://doi.org/10.1002/brb3.3031/v2/response1
Publikováno v:
Challenges in Parkinson's Disease
The etiology of Parkinson’s disease (PD) is complex and involves numerous risk factors as environmental and hereditary. Nevertheless, recent studies have established that systemic inflammation and neuroinflammation are both present in the prodromal
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::64f3cbab35d0d022583e57ace455b807
https://doi.org/10.5772/62955
https://doi.org/10.5772/62955
Autor:
Ismael Torres, Paul Carrillo-Mora, Perla Ugalde-Muñiz, José Pedraza-Chaverri, E. Ortiz-Islas, Mónica Adriana Torres-Ramos, Enrique Pinzón, Rafael Lugo-Huitrón, Benjamín Pineda, Tonali Blanco-Ayala, V. Pérez-De La Cruz, Perla D. Maldonado, Tessy Lopez, Abel Santamaría, Esperanza Garcia, Daniela Silva-Adaya
Publikováno v:
Neurotoxicology and Teratology. 33:538-547
Kynurenic acid (KYNA) is an endogenous metabolite of the kynurenine pathway for tryptophan degradation and an antagonist of both N-methyl-D-aspartate (NMDA) and alpha-7 nicotinic acetylcholine (α7nACh) receptors. KYNA has also been shown to scavenge
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3d64e2c2858bb7f837bbbe6715ab883f
https://doi.org/10.1016/j.ntt.2011.07.002
https://doi.org/10.1016/j.ntt.2011.07.002
Autor:
Anabel Jiménez-Anguiano, Rafael Lugo Huitrón, Dinora F. González-Esquivel, Camilo Ríos, Verónica Pérez de la Cruz, José Pedraza-Chaverri, Perla Ugalde-Muñiz, Jazmin Reyes Ocampo, Benjamín Pineda
Publikováno v:
Oxidative Medicine and Cellular Longevity
Oxidative Medicine and Cellular Longevity, Vol 2014 (2014)
Oxidative Medicine and Cellular Longevity, Vol 2014 (2014)
The kynurenine pathway (KP) is the main route of tryptophan degradation whose final product is NAD+. The metabolism of tryptophan can be altered in ageing and with neurodegenerative process, leading to decreased biosynthesis of nicotinamide. This fac
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ac8d198ad450a5fd826b9d896e38f695
https://pubmed.ncbi.nlm.nih.gov/24693337
https://pubmed.ncbi.nlm.nih.gov/24693337
Autor:
Rafael Lugo-Huitrón, José Pedraza-Chaverri, Perla Ugalde Muñiz, Camilo Ríos, Verónica Pérez de la Cruz, Benjamin Pineda
Publikováno v:
Oxidative Medicine and Cellular Longevity, Vol 2013 (2013)
Oxidative Medicine and Cellular Longevity
Oxidative Medicine and Cellular Longevity
Quinolinic acid (QUIN), a neuroactive metabolite of the kynurenine pathway, is normally presented in nanomolar concentrations in human brain and cerebrospinal fluid (CSF) and is often implicated in the pathogenesis of a variety of human neurological
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c60d007a23c848b2ccfd0aacccee7c28
https://doaj.org/article/4cace8a902fd40f5a152d92bc06ffca5
https://doaj.org/article/4cace8a902fd40f5a152d92bc06ffca5
Autor:
Perla D. Maldonado, Gustavo Ignacio Vazquez-Cervantes, Syed F. Ali, Carlos A. Silva-Islas, Verónica Pérez de la Cruz, Rafael Lugo-Huitrón, Ramón Lecona-Vargas, Mónica Adriana Torres-Ramos, Abel Santamaría, Tonali Blanco-Ayala, Perla Ugalde-Muñiz, Teresa I. Fortoul
Publikováno v:
Neurochemistry international. 61(2)
Selenium (Se) is a crucial element exerting antioxidant and neuroprotective effects in different toxic models. It has been suggested that Se acts through selenoproteins, of which thioredoxin reductase (TrxR) is relevant for reduction of harmful hydro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7d80a5f9b5afebccf9dd74170bafdf16
https://pubmed.ncbi.nlm.nih.gov/22579569
https://pubmed.ncbi.nlm.nih.gov/22579569