Zobrazeno 1 - 10
of 161
pro vyhledávání: '"Pericleous C"'
Autor:
Lang, M, Leung, K-Y, Greene, N, Malone, K, Saginc Giannoustas, G, Randi, A, Kiprianos, A, Maughan, R, Pericleous, C, Mason, J
Objectives: The disease-modifying anti-rheumatic drug methotrexate (MTX) is recognized to reduce cardiovascular risk in patients with systemic inflammatory diseases. However, the molecular basis for these cardioprotective effects remains incompletely
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______1032::e58b948acd59355aff2117e890d0b52d
http://hdl.handle.net/10044/1/105037
http://hdl.handle.net/10044/1/105037
Akademický článek
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Autor:
Paschalaki, K, Rossios, C, Pericleous, C, MacLeod, M, Rothery, S, Donaldson, G, Wedzicha, J, Gorgoulis, V, Randi, A, Barnes, P
Cellular senescence contributes to the pathophysiology of chronic obstructive pulmonary disease (COPD) and cardiovascular disease. Using endothelial-colony-forming-cells (ECFC), we have demonstrated accelerated senescence in smokers and COPD patients
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______1032::71ae361afdb64dfcc7886a43a1ceffad
http://hdl.handle.net/10044/1/93096
http://hdl.handle.net/10044/1/93096
Publikováno v:
Lupus
Objectives This study aims to inhibit antiphospholipid syndrome (APS) serum derived IgA anti-beta-2-glycoprotein I (aβ2GPI) binding using Domain I (DI). Methods Serum from 13 APS patients was tested for IgA aβ2GPI and Anti-Domain I. Whole IgA was p
Autor:
Ioannou, Y. *, Romay Penabad, Z., Pericleous, C., Giles, I., Papalardo, E., Vargas, G., Shilagard, T., Latchman, D.S., Isenberg, D.A., Rahman, A., Pierangeli, S.
Publikováno v:
In Journal of Thrombosis and Haemostasis May 2009 7(5):833-842
Autor:
Khawaja, AA, Chong, DLW, Sahota, J, Mikolasch, TA, Pericleous, C, Ripoll, VM, Booth, HL, Khan, S, Rodriguez-Justo, M, Giles, IP, Porter, JC
Neutrophilic inflammation correlates with mortality in fibrotic interstitial lung disease (ILD) particularly in the most severe form, idiopathic pulmonary fibrosis (IPF), although the underlying mechanisms remain unclear. Neutrophil function is modul
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=core_ac_uk__::3303535d2098d85e665c69bbda246014
https://openaccess.sgul.ac.uk/id/eprint/112329/9/fimmu-11-02190.pdf
https://openaccess.sgul.ac.uk/id/eprint/112329/9/fimmu-11-02190.pdf
Akademický článek
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Akademický článek
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Objectives This study aims to inhibit antiphospholipid syndrome (APS) serum derived IgA anti-beta-2-glycoprotein I (aβ2GPI) binding using Domain I (DI). Methods Serum from 13 APS patients was tested for IgA aβ2GPI and Anti-Domain I. Whole IgA was p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______1032::fe2e8cfce66f59edeb66514d564764a7
http://hdl.handle.net/10044/1/71956
http://hdl.handle.net/10044/1/71956
Autor:
McDonnell, TCR, Willis, R, Pericleous, C, Ripoll, VM, Giles, IP, Isenberg, DA, Brasier, AR, Gonzalez, EB, Papalardo, E, Romay-Penabad, Z, Jamaluddin, M, Ioannou, V, Rahman, A
APS is an autoimmune disease in which antiphospholipid antibodies (aPL) cause vascular thrombosis and pregnancy morbidity. In patients with APS, aPL exert pathogenic actions by binding serum beta-2-glycoprotein I (β2GPI) via its N-terminal domain I
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______1032::cb80073f75f0d022b32794de185e1a16
http://hdl.handle.net/10044/1/68244
http://hdl.handle.net/10044/1/68244