Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Peptide YY-3"'
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-10 (2024)
Abstract Fetal alcohol spectrum disorders (FASD) are a severe developmental condition resulting from exposure to alcohol during pregnancy. The aim of this study was to examine the concentrations of hormones involved in appetite regulation—ghrelin,
Externí odkaz:
https://doaj.org/article/051185c6805741d8b82ddf53292d98ee
Akademický článek
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Publikováno v:
Drug Development and Industrial Pharmacy. 45:1101-1110
Peptide YY(3–36) (PYY(3–36)) is an endogenous appetite suppressing peptide. The present research was to perform pharmacokinetic/pharmacodynamic (PK/PD) analysis for predicting the concentration- an...
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::bafb7d577544ce0f04a6f2942f14aca9
https://doi.org/10.1080/03639045.2019.1593443
https://doi.org/10.1080/03639045.2019.1593443
Publikováno v:
Gastroenterology. 160:S-74
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9c7783432cda1960a1248b45ca7031ea
https://doi.org/10.1016/s0016-5085(21)00923-9
https://doi.org/10.1016/s0016-5085(21)00923-9
Autor:
Kimberly P. Kinzig, Ulrika Smedh, Ellen E. Ladenheim, Susan Knipp, Karen A. Scott, Timothy H. Moran
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 288:R384-R388
Peptide YY3–36 [PYY(3–36)], a gastrointestinal peptide that is released into the circulation in response to ingesting a meal, has recently been suggested to play a role in controlling food intake. PYY(3–36) has been reported to inhibit food int
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::732038fefc148708d11fa0a76ff0423c
https://doi.org/10.1152/ajpregu.00535.2004
https://doi.org/10.1152/ajpregu.00535.2004
Autor:
Niamh M. Martin, A Sajedi, Stephen R. Bloom, Caroline J. Small, Michael Patterson, Mohammad A. Ghatei
Publikováno v:
International Journal of Obesity. 28:886-893
The role of the melanocortin system in the feeding effects of peripheral peptide YY(3-36) (PYY(3-36)) and ghrelin was investigated using the agouti (A(y)/a) mouse as a model of abnormal melanocortin signalling. Furthermore, we examined whether the ec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a07cdaba6c1fd39bbeaca5823deb9322
https://doi.org/10.1038/sj.ijo.0802646
https://doi.org/10.1038/sj.ijo.0802646
Publikováno v:
Peptides. 27(12)
We used a conditioned taste aversion test to assess whether PYY(3-36) reduces food intake by producing malaise. Two-hour IV infusion of PYY(3-36) (8, 15, and 30 pmol/kg/min) at dark onset in non-food-deprived rats produced a dose-dependent inhibition
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bb3f8dea978e61d7e760c45bb901d25d
https://pubmed.ncbi.nlm.nih.gov/16962209
https://pubmed.ncbi.nlm.nih.gov/16962209
Autor:
Joseph R. Reeve, Prasanth K. Chelikani, Roger D. Reidelberger, Alvin C. Haver, David A. Keire
Publikováno v:
American journal of physiology. Regulatory, integrative and comparative physiology. 290(2)
The gut hormone peptide YY(3-36) [PYY(3-36)] decreases food intake when administered by intravenous infusion to lean and obese humans and rats. Whether chronic administration of PYY(3-36) produces a sustained reduction in food intake and adiposity is
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c8219bf91d0cbdc1b93f34b894d794c9
https://pubmed.ncbi.nlm.nih.gov/16210414
https://pubmed.ncbi.nlm.nih.gov/16210414
Publikováno v:
Endocrinology. 145(11)
The gut-derived hormones peptide YY[3-36] (PYY[3-36]) and ghrelin are believed to influence similar hypothalamic circuits, albeit with opposing actions on energy balance. Thus, we carried out a series of studies to evaluate the interaction of these h
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5487cba55fcf54fa4e0a40576228c7d5
https://pubmed.ncbi.nlm.nih.gov/15284202
https://pubmed.ncbi.nlm.nih.gov/15284202