Hypoxia sensing in resident cardiac macrophages regulates monocyte fate specification following ischemic heart injury.

Autor: Kadyrov FF; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Koenig AL; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Amrute JM; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Dun H; Division of Cardiothoracic Surgery, Department of Surgery, Washington University School of Medicine, Saint Louis, MO, USA., Li W; Division of Cardiothoracic Surgery, Department of Surgery, Washington University School of Medicine, Saint Louis, MO, USA., Weinheimer CJ; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Nigro JM; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Kovacs A; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Bredemeyer AL; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Yang S; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Das S; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Penna VR; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Parvathaneni A; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Lai L; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Hartmann N; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA.; German Center for Cardiovascular Research (DZHK), Partner Site Heidelberg, Heidelberg, Germany.; Department of Cardiology, Internal Medicine III, Heidelberg University Hospital, Heidelberg, Germany., Kopecky BJ; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA., Kreisel D; Division of Cardiothoracic Surgery, Department of Surgery, Washington University School of Medicine, Saint Louis, MO, USA.; Department of Pathology and Immunology, Washington University School of Medicine, Saint Louis, MO, USA., Lavine KJ; Center for Cardiovascular Research, Division of Cardiology, Department of Medicine, Washington University School of Medicine, Saint Louis, MO, USA. klavine@wustl.edu.; Department of Pathology and Immunology, Washington University School of Medicine, Saint Louis, MO, USA. klavine@wustl.edu.; Department of Developmental Biology, Washington University School of Medicine, Saint Louis, MO, USA. klavine@wustl.edu.

Publikováno v: Nature cardiovascular research [Nat Cardiovasc Res] 2024 Nov; Vol. 3 (11), pp. 1337-1355. Date of Electronic Publication: 2024 Oct 21.

NFĸB signaling drives myocardial injury via CCR2+ macrophages in a preclinical model of arrhythmogenic cardiomyopathy.

Autor: Chelko SP; Department of Biomedical Sciences, Florida State University College of Medicine, Tallahassee, Florida, USA.; Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA., Penna VR; Department of Medicine, Washington University, St. Louis, Missouri, USA., Engel M; Department of Biomedical Sciences, Florida State University College of Medicine, Tallahassee, Florida, USA., Shiel EA; Department of Biomedical Sciences, Florida State University College of Medicine, Tallahassee, Florida, USA., Centner AM; Department of Biomedical Sciences, Florida State University College of Medicine, Tallahassee, Florida, USA., Farra W; Department of Biomedical Sciences, Florida State University College of Medicine, Tallahassee, Florida, USA., Cannon EN; Department of Biomedical Sciences, Florida State University College of Medicine, Tallahassee, Florida, USA., Landim-Vieira M; Department of Biomedical Sciences, Florida State University College of Medicine, Tallahassee, Florida, USA., Schaible N; Departments of Pathology and Emergency Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts, USA., Lavine K; Department of Medicine, Washington University, St. Louis, Missouri, USA., Saffitz JE; Departments of Pathology and Emergency Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts, USA.

Publikováno v: The Journal of clinical investigation [J Clin Invest] 2024 Apr 02; Vol. 134 (10). Date of Electronic Publication: 2024 Apr 02.

A Murine Herpesvirus Closely Related to Ubiquitous Human Herpesviruses Causes T-Cell Depletion.

Autor: Patel SJ; Division of Rheumatology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA., Zhao G; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, Missouri, USA., Penna VR; Division of Rheumatology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA., Park E; Division of Rheumatology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA., Lauron EJ; Division of Rheumatology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA., Harvey IB; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, Missouri, USA., Beatty WL; Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, Missouri, USA., Plougastel-Douglas B; Division of Rheumatology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA., Poursine-Laurent J; Howard Hughes Medical Institute, St. Louis, Missouri, USA., Fremont DH; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, Missouri, USA.; Department of Biochemistry and Molecular Biophysics, Washington University School of Medicine, St. Louis, Missouri, USA., Wang D; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, Missouri, USA.; Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, Missouri, USA., Yokoyama WM; Division of Rheumatology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA yokoyama@dom.wustl.edu.; Howard Hughes Medical Institute, St. Louis, Missouri, USA.

Publikováno v: Journal of virology [J Virol] 2017 Apr 13; Vol. 91 (9). Date of Electronic Publication: 2017 Apr 13 (Print Publication: 2017).