Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Pengran Du"'
Autor:
Yuqian Chang, Pan Kang, Tingting Cui, Weinan Guo, Weigang Zhang, Pengran Du, Xiuli Yi, Sen Guo, Tianwen Gao, Chunying Li, Shuli Li
Publikováno v:
Journal of Translational Medicine, Vol 21, Iss 1, Pp 1-19 (2023)
Abstract Background The activation of CD8+ T cells and their trafficking to the skin through JAK-STAT signaling play a central role in the development of vitiligo. Thus, targeting this key disease pathway with innovative drugs is an effective strateg
Externí odkaz:
https://doaj.org/article/965a199f1d41414ca6d1b92f08574ed9
Autor:
Pan Kang, Jianru Chen, Weigang Zhang, Ningning Guo, Xiuli Yi, Tingting Cui, Jiaxi Chen, Yuqi Yang, Yinghan Wang, Pengran Du, Zhubiao Ye, Baizhang Li, Chunying Li, Shuli Li
Publikováno v:
Cell Death Discovery, Vol 8, Iss 1, Pp 1-9 (2022)
Abstract Vitiligo is a cutaneous depigmenting autoimmune disease caused by the extensive destruction of epidermal melanocytes. Convincing data has defined a critical role for oxidative stress in the pathogenesis of vitiligo. Oxeiptosis is a caspase-i
Externí odkaz:
https://doaj.org/article/677ea584fdac4da19ce4825f4fb70742
Autor:
Zhubiao Ye, Jianru Chen, Pengran Du, Qingrong Ni, Baizhang Li, Zhe Zhang, Qi Wang, Tingting Cui, Xiuli Yi, Chunying Li, Shuli Li
Publikováno v:
Frontiers in Immunology, Vol 13 (2022)
Vitiligo is a depigmented skin disorder caused by a variety of factors, including autoimmune, metabolic disturbance or their combined effect, etc. Non-targeted metabolomic analyses have denoted that dysregulated fatty acids metabolic pathways are inv
Externí odkaz:
https://doaj.org/article/2b1569d5140748b49737836ba010587d
Autor:
Jianru Chen, Weinan Guo, Pengran Du, Tingting Cui, Yuqi Yang, Yinghan Wang, Pan Kang, Zhe Zhang, Qi Wang, Zhubiao Ye, Ling Liu, Zhe Jian, Tianwen Gao, Huijie Bian, Shuli Li, Chunying Li
Publikováno v:
The Journal of Pathology. 260:84-96
Autor:
Zhubiao Ye, Jianru Chen, Pengran Du, Qingrong Ni, Baizhang Li, Zhe Zhang, Qi Wang, Tingting Cui, Xiuli Yi, Chunying Li, Shuli Li
Publikováno v:
Frontiers in Immunology, Vol 13 (2022)
Vitiligo is a depigmented skin disorder caused by a variety of factors, including autoimmune, metabolic disturbance or their combined effect, etc. Non-targeted metabolomic analyses have denoted that dysregulated fatty acids metabolic pathways are inv
Autor:
Pan Kang, Jianru Chen, Weigang Zhang, Ningning Guo, Xiuli Yi, Tingting Cui, Jiaxi Chen, Yuqi Yang, Yinghan Wang, Pengran Du, Zhubiao Ye, Baizhang Li, Chunying Li, Shuli Li
Publikováno v:
Cell death discovery. 8(1)
Vitiligo is a cutaneous depigmenting autoimmune disease caused by the extensive destruction of epidermal melanocytes. Convincing data has defined a critical role for oxidative stress in the pathogenesis of vitiligo. Oxeiptosis is a caspase-independen
Folic Acid Protects Melanocytes from Oxidative Stress via Activation of Nrf2 and Inhibition of HMGB1
Autor:
Pengran Du, Shaolong Zhang, Shuli Li, Yuqi Yang, Pan Kang, Jiaxi Chen, Tianwen Gao, Chunying Li, Qian Zhang, Weigang Zhang
Publikováno v:
Oxidative Medicine and Cellular Longevity, Vol 2021 (2021)
Oxidative Medicine and Cellular Longevity
Oxidative Medicine and Cellular Longevity
Vitiligo is a cutaneous depigmentation disease due to loss of epidermal melanocytes. Accumulating evidence has indicated that oxidative stress plays a vital role in vitiligo via directly destructing melanocytes and triggering inflammatory response th
Autor:
Zhe Jian, Shuli Li, Jiaxi Chen, Ling Liu, Xuguang Chen, Yinghan Wang, Chunying Li, Pengran Du, Tongtian Zhuang, Kai Li, Jianru Chen, Baizhang Li, Xiuli Yi, Qingrong Ni, Pan Kang, Zhubiao Ye, Yuqian Chang, Tianwen Gao, Tingting Cui
Publikováno v:
Cell Death & Disease
Cell Death and Disease, Vol 11, Iss 6, Pp 1-14 (2020)
Cell Death and Disease, Vol 11, Iss 6, Pp 1-14 (2020)
Vitiligo is a disfiguring disease featuring chemokines-mediated cutaneous infiltration of autoreactive CD8+ T cells that kill melanocytes. Copious studies have indicated that virus invasion participates in the pathogenesis of vitiligo. IFIH1, encodin