Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Penelope Andrew"'
Autor:
Kurt Schmidt, Penelope Andrew, Bernd Mayer, Georg Kojda, Klaus Groschner, Verena Schmitz, Astrid Schrammel
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 281:H2053-H2061
The neuronal and endothelial isoforms of nitric oxide (NO) synthase (nNOS and eNOS, respectively) both catalyze the production of NO but are regulated differently. Stably transfected HEK 293 cell lines containing nNOS, eNOS, and a soluble mutant of e
Autor:
Antonius C. F. Gorren, Bernd Mayer, Hans jörg Habisch, Ernst R. Werner, Richard C. Venema, Kurt Schmidt, Penelope Andrew, Regina Golser, Andrea Leber
Publikováno v:
Journal of Biological Chemistry. 275:5291-5296
Endothelial nitric-oxide synthase (type III) (eNOS) was reported to form an inhibitory complex with the bradykinin receptor B2 (B2R) from which the enzyme is released in an active form upon receptor activation (Ju, H., Venema, V. J., Marrero, M. B.,
Autor:
Bernd Mayer, Penelope Andrew
Publikováno v:
Naunyn-Schmiedeberg's Archives of Pharmacology. 358:127-133
Biosynthesis of nitric oxide (NO) is performed by the dimeric, heme-containing enzyme nitric oxide synthase, which requires the flavins FAD and FMN, as well as the pteridine cofactor (6R)-5,6,7,8-tetrahydro-L-biopterin (H4biopterin) in order to catal
Autor:
Ivan J. D. Lindley, Rainer de Martin, Elisabeth Foglar, Penelope Andrew, Christian Dittrich, Hanna Harant
Publikováno v:
Journal of Biological Chemistry. 271:26954-26961
Induction of interleukin-8 (IL-8) by IL-1 or tumor necrosis factor (TNF), and repression by interferons or glucocorticoids have been shown to involve sequences between nucleotides -94 and -71 of the 5'-flanking region, and the transcription factors N
Autor:
Robert Maier, Penelope Andrew, Friedrich Brunner, Gerald Wölkart, Bernd Mayer, Rudolf Zechner
Publikováno v:
Cardiovascular research. 57(1)
Objective: The role of nitric oxide (NO) in myocardial ischemia/reperfusion injury remains controversial as both NO donors and NO synthase (NOS) inhibitors have shown to be protective. We generated transgenic (TG) mice that overexpress endothelial NO
Publikováno v:
Circulation. 104(25)
Background — The major source of nitric oxide (NO) in the heart is the constitutive form of NO synthases (eNOS, NOS III) that is expressed in vascular endothelium and cardiac myocytes. NO mediates endothelium-dependent vasodilation and may modulate
Autor:
Regina Golser, Antonius C.F. Gorren, Andrea Leber, Penelope Andrew, Hans-Jörg Habisch, Ernst R. Werner, Kurt Schmidt, Richard C. Venema, Bernd Mayer
Publikováno v:
Journal of Biological Chemistry. 275:16400