Zobrazeno 1 - 10
of 32
pro vyhledávání: '"Pei-Xi Chen"'
Publikováno v:
In Life Sciences 2005 78(1):61-66
Autor:
Cedric Bardy, Michael Navarro, Shawn P. Driscoll, Takahiro Tadokoro, Atsushi Miyanohara, Tomohisa Kato Jr., Silvia Marsala, Joseph D. Ciacci, Eric Curtis, Jack D. Bui, Jozef Kafka, Stefan Juhas, Kazuhiko Yamada, Samuel L. Pfaff, Pei Xi Chen, Andrew J. Todd, Tetsuya Yoshizumi, Jan Strnadel, Kota Kamizato, Jana Juhasova, Michael P. Hefferan, Chak Sum Ho, Andreas Nørgaard Glud, Thomas D. Glenn, Oleksandr Platoshyn, Marian Hruska-Plochan, Cassiano Carromeu, Jiri Klima, Martin Marsala, Taba Kheradmand, Alysson R. Muotri, Dáša Bohačiaková, Fred H. Gage
The use of autologous (or syngeneic) cells derived from induced pluripotent stem cells (iPSCs) holds great promise for future clinical use in a wide range of diseases and injuries. It is expected that cell replacement therapies using autologous cells
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::23e6a2f54557cf79f2a5bfe977276eb1
https://eprints.gla.ac.uk/162268/1/162268.pdf
https://eprints.gla.ac.uk/162268/1/162268.pdf
Autor:
Dongdan Zheng, Liqiu Mo, Runmin Guo, Wenming Xu, Pei-Xi Chen, Keng Wu, Jianqiang Feng, Xiaoxiao Hua, Jian-Cong Lin
Publikováno v:
Molecular Medicine Reports. 8:603-608
A number of studies have demonstrated that inflammation plays a role in doxorubicin (DOX)-induced cardiotoxicity. However, the molecular mechanism by which DOX induces cardiac inflammation has yet to be fully elucidated. The present study aimed to in
Autor:
Xin-xue Liao, Pei-Xi Chen, Liqiu Mo, Jianqiang Feng, Xiuyu Wang, Mei-Fen Zhang, Aiping Lan, Chun-Tao Yang, Fen Hu, Xiao-bian Dong, Dongdan Zheng
Publikováno v:
Molecular and Cellular Biochemistry. 362:149-157
Hydrogen sulfide (H(2)S) has been shown to exert cardioprotective effects. However, the roles of extracellular signal-regulated protein kinases 1/2 (ERK1/2) in H(2)S-induced cardioprotection have not been completely elucidated. In this study, cobalt
Autor:
Aiping Lan, Chun-Mei Zhao, Chun-Tao Yang, Chen-Heng Hu, Jianqiang Feng, Pei-Xi Chen, Yan-fen Dong, Jin-lan Meng, Wei-Yi Mei, Jian-hong Wang
Publikováno v:
Clinical and Experimental Pharmacology and Physiology. 38:42-49
1. Increasing evidence indicates that hydrogen sulphide (H₂S) may serve as an important biological cytoprotective agent. Heat shock protein (Hsp) 90 can attenuate stress-induced injury. However, whether Hsp90 mediates the cytoprotective effect of H
Autor:
Mei Zhang, Liqiu Mo, Yu Cui, Rui-Xian Guo, Wei Li, Xin-xue Liao, Wei Liu, Yan-Li Wang, Pei-Xi Chen, Jun-Li Zhi, Sheng-Nan Sun, Jianqiang Feng
Publikováno v:
Clinical and Experimental Pharmacology and Physiology. 36:304-311
1. Cytoprotection by H(2)O(2) preconditioning against oxidative stress-induced apoptosis of PC12 cells has been demonstrated previously. In the present study, we investigated the effects of H(2)O(2) preconditioning on nuclear factor (NF)-kappaB activ
Autor:
Hui-Min Yu, Yan-Li Wang, Jianqiang Feng, Chu-Huai Wang, Yu Cui, Liqiu Mo, Chun-Mei Zhao, Jun-Li Zhi, Wei Liu, Pei-Xi Chen, Sheng-Nan Sun
Publikováno v:
Neuroscience Letters. 410:174-177
We have demonstrated that the activation of p38 mitogen-activated protein kinase (MAPK) in the spinal microglia played an essential role in the development of morphine antinociceptive tolerance. The aim of this study was to investigate whether inhibi
Publikováno v:
Apoptosis. 11:931-941
The aim of this study was to investigate the role of JAK-STAT pathway in the cytoprotection afforded by preconditioning with H(2)O(2). It was shown that (1) Preconditioning with 100 micromol/L H(2)O(2) can markedly protect PC12 cells against apoptosi
Autor:
Hui-Min Yu, Yu Cui, Pei-Xi Chen, Er-hu Tang, J. Q. Feng, Jian Chen, Sheng-Nan Sun, Jun-Li Zhi, Xiaolan Tang
Publikováno v:
Apoptosis. 11:943-953
The aim of present study is to explore the cytoprotection of curcumin against 1-methyl-4-phenylpridinium ions (MPP(+))-induced apoptosis and the molecular mechanisms underlying in PC12 cells. Our findings indicated that MPP(+) significantly reduced t
Publikováno v:
Brain Research. 1069:235-243
Compelling evidence has suggested that spinal glial cells were activated by chronic morphine treatment and involved in the development of morphine tolerance. However, the mechanisms of glial activation were still largely unknown in morphine tolerance