Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Pegah Karimi Elizee"'
Publikováno v:
International Journal of Preventive Medicine, Vol 4, Iss 8, Pp 863-865 (2013)
Externí odkaz:
https://doaj.org/article/1a52f182e4d34e858af18bcaebf3b2be
Autor:
Heidar Sharafi, Farah Bokharaei-Salim, Shima Salimi, Maryam Keshvari, Seyyed Mohammad Miri, Pegah Karimi Elizee, Hosein Keyvani, Seyed Moayed Alavian, Bita Behnava
Publikováno v:
Iranian Red Crescent Medical Journal. 19
Background: Sustained virologic response (SVR) to pegylated-interferon (PegIFN) and ribavirin (RBV) in hepatitis C virus (HCV)-infected patients could be predicted by detection of serum HCV RNA whereas detection of HCV RNA in other reservoirs such as
Autor:
Seyyed Mohammad Miri, Pegah Karimi Elizee, Bita Behnava, Seyed Moayed Alavian, Ali Pouryasin, Shima Salimi, Heidar Sharafi, Maryam Keshvari
Publikováno v:
Hepatitis Monthly. 17
Background and Aims: The presence of the hepatitis C virus (HCV) in cells of extrahepatic organs like peripheral blood mononuclear cells (PBMCs) have important implications for transmission, disease progression, and effective treatment of HCV-infecte
Publikováno v:
Thrita.
Context: Peripheral blood mononuclear cells (PBMC), as the main extrahepatic reservoir of HCV infection, play an important role in HCV progression. Numerous host microRNAs (miRNAs) are probably involved in HCV entry, the formation of viral infection,
Autor:
Leila Mehrnoush, Bita Behnava, Danesh Kadjbaf, Maryam Keshvari, Pegah Karimi Elizee, Ali Pouryasin, Shima Salimi, Seyed Moayed Alavian, Heidar Sharafi
Publikováno v:
Hepatitis Monthly
Background: Molecular studies have demonstrated that the hepatitis C virus (HCV) genotype and host genetics play predictive roles in the management of patients infected with HCV. Objectives: This study aimed to investigate the HCV genotype, core amin
Autor:
Ahmad Gharehbaghian, Seyed Moayed Alavian, Sedigheh Amini Kafi-Abad, Bashir Hajibeigy, Farhad Zamani, Seyyed Mohammad Miri, Pegah Karimi Elizee, Seyed Vahid Tabatabaei, Bita Behnava, Maryam Keshvari, Kamran Bagheri Lankarani
Publikováno v:
Hepatitis Monthly
Background Treatment guidelines contraindicate ribavirin for treatment of hepatitis C virus (HCV) infection in thalassemia major patients. Nevertheless, the current evidence suggests that ribavirin might be tolerated by these patients. Objectives Des
Autor:
Pegah Karimi Elizee, Kamran Bagheri Lankarani, Seyed Vahid Tabatabaei, Bita Behnava, Maryam Keshvari, Seyyed Mohammad Miri, Seyed Moayed Alavian
Publikováno v:
Liver International. 30:1173-1180
BACKGROUND/AIMS Chronic hepatitis C virus infection (HCV) is a major comorbidity in patients with haemophilia. Peginterferon alpha and ribavirin is current standard anti-HCV therapy but there is little information about safety and efficacy of peginte
Autor:
Seyyed Mohammad Miri, Maryam Keshvari, Bita Behnava, Seyed Moayed Alavian, Kamran Bagheri Lankarani, S. V. Tabatabaei, Pegah Karimi Elizee
Publikováno v:
Transfusion. 49:2195-2199
BACKGROUND: Hepatitis C virus (HCV) infection is the most common transfusion-transmitted disease in multiply transfused patients worldwide. In this study, the aim was to investigate distribution of HCV genotypes in Iranian patients with thalassemia.
Autor:
Seyed Moayed Alavian, Shima Salimi, Heidar Sharafi, Ali Pouryasin, Maryam Keshvari, Mehran Ghazimoghaddam, Bita Behnava, Pegah Karimi Elizee, Leila Mehrnoush
Publikováno v:
Hepatitis Monthly
Background: Hepatitis C Virus (HCV) is the major cause of liver failure in thalassemic patients. In these patients, iron overload and their comorbidities make difficulties during Pegylated-Interferon (PEG-IFN) and Ribavirin (RBV) therapy. Objectives:
Autor:
Seyed Moayed Alavian, Pegah Karimi Elizee, Heidar Sharafi, Bita Behnava, Leila Mehrnoush, Maryam Keshvari, Saleh Sandoughdaran, Shima Salimi
Publikováno v:
Hepatitis Monthly
Background: Most thalassemic patients with chronic hepatitis C virus (HCV) infection do not respond to therapy with pegylated interferon (Peg-IFN) plus ribavirin (RBV) due to hepatic siderosis and RBV dose reduction caused by RBV-induced anemia. Obje