Zobrazeno 1 - 10
of 33
pro vyhledávání: '"Paula Sundstrom"'
Autor:
John E. Beute, Alex Y. Kim, Jamie J. Park, Allen Yang, Keshia Torres-Shafer, David W. Mullins, Paula Sundstrom
Publikováno v:
Frontiers in Cellular and Infection Microbiology, Vol 12 (2022)
Pseudomembranous candidiasis (thrush), erythematous candidiasis, and fungal esophagitis are infections of the barrier mucosa of the upper gastrointestinal tract. The majority of these infections are caused by Candida albicans, an opportunistic fungal
Externí odkaz:
https://doaj.org/article/d6719bd17be04bf8b4768ca1e2183fec
Autor:
Samin Kim, Quoc Bao Nguyen, Michael J Wolyniak, Gregory Frechette, Christian R Lehman, Brandon K Fox, Paula Sundstrom
Publikováno v:
PLoS ONE, Vol 13, Iss 2, p e0192260 (2018)
The mechanisms that fungi use to co-regulate subsets of genes specifically associated with morphogenic states represent a basic unsolved problem in fungal biology. Candida albicans is an important model of fungal differentiation both for rapid interc
Externí odkaz:
https://doaj.org/article/b32b56d8e6994a92a7ba27fecdebc103
Autor:
Quoc Bao Nguyen, Brandon K Fox, Gregory Frechette, Samin Kim, Christian R. Lehman, Michael J. Wolyniak, Paula Sundstrom
Publikováno v:
PLoS ONE
PLoS ONE, Vol 13, Iss 2, p e0192260 (2018)
PLoS ONE, Vol 13, Iss 2, p e0192260 (2018)
The mechanisms that fungi use to co-regulate subsets of genes specifically associated with morphogenic states represent a basic unsolved problem in fungal biology. Candida albicans is an important model of fungal differentiation both for rapid interc
Autor:
Janet F. Staab, Paula Sundstrom, Courtney A. Lyman, Laura J. Gordon, Matthew A. Molenda, Yong Sun Bahn
Publikováno v:
Eukaryotic Cell. 6:2376-2390
Candida albicans is an opportunistic human fungal pathogen that causes systemic candidiasis as well as superficial mucosal candidiasis. In response to the host environment, C. albicans transitions between yeast and hyphal forms. In particular, hyphal
Publikováno v:
Eukaryotic Cell. 6:693-709
To elucidate the molecular mechanisms controlling the expression of the hypha-specific adhesin gene HWP1 of Candida albicans , its promoter was dissected and analyzed using a green fluorescent protein reporter gene. A 368-bp region, the HWP1 control
Publikováno v:
Molecular Microbiology. 50:391-409
Summary Frequent interconversion between yeasts, pseudohyphae and true hyphae is a hallmark of Candida albicans growth in mammalian tissues. The requirement for transient CAP1-dependent pulses of cAMP for generating true hyphae, Hwp1 and virulence ra
Autor:
Paula Sundstrom
Publikováno v:
Cellular Microbiology. 4:461-469
Summary Microbial adherence is one of the most important determinants of pathogenesis, yet very few adhesins have been identified from fungal pathogens. Four structurally related adhesins, Hwp1, Ala1p/Als5p, Als1p, from Candida albicans and Epa1p fro
Publikováno v:
The Journal of Infectious Diseases. 185:521-530
Oroesophageal candidiasis is caused by the combined action of fungal virulence factors and host inflammatory responses when protective immunity is absent. Hyphal wall protein 1 (Hwp1) on germ tubes and true hyphae of Candida albicans forms covalent c
Autor:
William J. Buesching, Susan L. Koletar, Christel Haberland‐Carrodeguas, F. Michael Beck, Paula Sundstrom, Carl M. Allen
Publikováno v:
Journal of Oral Pathology & Medicine. 31:99-105
Background: In contrast to the immunosupressed patient population, the prevalence of fluconazole-resistant strains of Candida albicans among healthy individuals has not been extensively studied. Methods: Candida species were cultured form 50 healthy
Autor:
Paula Sundstrom
Publikováno v:
Molecular Principles of Fungal Pathogenesis
Current research shows that major changes in gene expression accompany morphogenesis and suggest that fungal attributes necessary for proliferation and survival in the host are present in both yeast and hyphal growth forms. Manipulation of host-fungu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2346b341623c8d141a4e0be0d0d43639
https://doi.org/10.1128/9781555815776.ch4
https://doi.org/10.1128/9781555815776.ch4