Zobrazeno 1 - 10 of 10 pro vyhledávání: '"Paul Plested"'
1
Evidence for Rosettes as an Unrecognized Stage in the Life Cycle of Leishmania Parasites
Autor: Gregory R. Moe, David M. Iovannisci, C Paul Plested
Publikováno v: Journal of Eukaryotic Microbiology. 57:405-414
Leishmania parasites, which afflict 12 million people in 88 countries, exist as promastigotes transmitted by insect vectors and as amastigotes residing in mammalian macrophages. Promastigote cells arranged in rosettes have also been described but uni
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::17788dd1e3f50f91e8b7fe78d79f2357
https://doi.org/10.1111/j.1550-7408.2010.00496.x
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2
Antibodies in Myasthenia Gravis and Related Disorders
Autor: John McConville, Nick Willcox, Werner Hoch, Angela Vincent, John Newsom-Davis, David Beeson, Leslie Jacobson, Maria Elena Farrugia, Agata Polizzi, Gary Sims, Paola Dalton, Amelia Evoli, Paul Plested, Ian Matthews, Frans Blaes, Bethan Lang, Teresa Tang, John Bowen
Publikováno v: Annals of the New York Academy of Sciences. 998:324-335
Acetylcholine receptor (AChR) antibodies are present in around 85% of patients with myasthenia gravis (MG) as measured by the conventional radioimmunoprecipitation assay. Antibodies that block the fetal form of the AChR are occasionally present in mo
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::af4a3d0d6fdad125f2ce83913c117557
https://doi.org/10.1196/annals.1254.036
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3
IgG from ?seronegative? myasthenia gravis patients binds to a muscle cell line, TE671, but not to human acetylcholine receptor
Autor: Bethan Lang, Franz Blaes, Paul Plested, David Beeson, Angela Vincent
Publikováno v: Annals of Neurology. 47:504-510
Antibodies to acetylcholine receptor (AChR) are found in 85% of patients with myasthenia gravis (seropositive MG [SPMG]) and are thought to be pathogenic; but in 15% of MG patients, the standard immunoprecipitation test for anti-AChR is negative (ser
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f19e638fbf4c982c76e29791400fee1d
https://doi.org/10.1002/1531-8249(200004)47:4<504::aid-ana14>3.0.co
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5
Antibodies Affecting Ion Channel Function in Acquired Neuromyotonia, in Seropositive and Seronegative Myasthenia Gravis, and in Antibody-mediated Arthrogryposis Multiplex Congenita
Autor: Nicholas Willcox, Angela Vincent, Sara Ghorazian, Leslie Jacobson, Paul Plested, John Newsdom-Davis, Cal Maclenan, Jeremy Farrar, Agata Polizzi, Teresa Tang, Claire Newland, David Beeson, Sietske Riemersma
Publikováno v: Annals of the New York Academy of Sciences. 841:482-496
A new autoimmune disease affecting the neuromuscular junction has been defined. Acquired neuromyotonia is associated with antibodies to voltage-gated potassium channels that act, at least in part, by reducing potassium channel function with resulting
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::daf32fa01a11365bc9149657bb3a31e0
https://doi.org/10.1111/j.1749-6632.1998.tb10968.x
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6
Interleukin-1 stimulates diglyceride accumulation in the absence of protein kinase C activation
Autor: Kanwar Virdee, C. Paul Plested, Henrik Skjodt, Barry L. Brown, R. Graham G. Russell, Alison D. Short, Pauline R.M. Dobson
Publikováno v: Regulatory Peptides. 29:109-116
Despite advances in the knowledge of the intracellular signalling in response to extracellular messengers, the mechanism of action of interleukin-1 (IL-1) has remained an enigma. In the present study, we have employed human dermal fibroblasts (Detroi
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::307962d959a63dfdf435a5ee7ae3b05a
https://doi.org/10.1016/0167-0115(90)90074-7
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8
Desensitisation of the oxytocin receptor and other G-protein coupled receptors in the human myometrium
Autor: C. Paul Plested, Andrés López Bernal
Publikováno v: Experimental physiology. 86(2)
G-protein coupled receptors (GPCRs) are key to maintaining uterine quiescence and inducing phasic contractions at term. However, the biochemical mechanisms whereby uterine GPCRs are desensitised and re-sensitised during these physiological conditions
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dc7c8f0b8c20761b39fcedae1140dedc
https://pubmed.ncbi.nlm.nih.gov/11429648
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9
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