Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Patti Polinsky"'
Autor:
Stephen M. Schwartz, Bardia Askari, Rebecca M. Varon, Lawrence Chan, Shelley Barnhart, Kazuhiro Oka, Erin D. MacDougall, Farah Kramer, Fredrik Johansson, Karin E. Bornfeldt, Patti Polinsky, Michael E. Rosenfeld
Publikováno v:
The American Journal of Pathology. 168:2064-2073
Very low-density lipoprotein (VLDL) and LDL plasma levels are associated with cardiovascular mortality. Whereas VLDL/LDL lowering causes regression of early atherosclerotic lesions, less is known about the effects of aggressive lipid lowering on regr
Publikováno v:
Cardiovascular Pathology. 14:120-125
Objective The present study describes the distribution of atherosclerotic lesions in the coronary arteries of chow-fed 60-week-old male ApoE−/−, 17-β-estradiol-treated ApoE−/−, and wild-type mice. Methods and results The histologic examinati
Autor:
Stephen M. Schwartz, Patti Polinsky, Richard J. Johnson, Donna Lombardi, Shinichi Suga, Katherine L. Gordon
Publikováno v:
Hypertension. 33:1013-1019
Abstract —We hypothesized that short-term exposure to angiotensin II (Ang II) could result in structural and functional changes in the kidney that would favor sodium retention and the development of sustained hypertension. To test this hypothesis,
Autor:
Patti Polinsky, Katalin Kauser, Michael E. Rosenfeld, Gabor M. Rubanyi, Renu Virmani, Stephen M. Schwartz
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology. 20(12)
Abstract —Most previous studies of atherosclerosis in hyperlipidemic mouse models have focused their investigations on lesions within the aorta or aortic sinus in young animals. None of these studies has demonstrated clinically significant advanced
Autor:
Hong Seog Seo, Lyn Powell-Braxton, Stuart Bunting, Donna Lombardi, Stephen M. Schwartz, Michael E. Rosenfeld, Patti Polinsky
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology. 17(12)
Abstract A systematic analysis of the distribution of advanced atherosclerotic lesions was undertaken in chow-fed, 9-month-old apolipoprotein (apo) E-deficient mice to identify sites amenable for study of mechanisms of formation of stenotic lesions.