Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Patrizia Fabbi"'
Autor:
Paola Altieri, Maria Bertolotto, Patrizia Fabbi, Elena Sportelli, Manrico Balbi, Francesco Santini, Claudio Brunelli, Marco Canepa, Fabrizio Montecucco, Pietro Ameri
Publikováno v:
Data in Brief, Vol 19, Iss , Pp 925-931 (2018)
The data presented here are related to the research paper entitled “Thrombin induces protease-activated receptor 1 signaling and activation of human atrial fibroblasts and dabigatran prevents these effects” (Altieri et al., 2018) [1]. Data show t
Externí odkaz:
https://doaj.org/article/2748a3cfd678496093a7e8237118676a
Autor:
Barbara Salani, Silvia Ravera, Patrizia Fabbi, Silvano Garibaldi, Mario Passalacqua, Claudio Brunelli, Davide Maggi, Renzo Cordera, Pietro Ameri
Publikováno v:
Cellular Physiology and Biochemistry, Vol 43, Iss 3, Pp 879-890 (2017)
Background: Sulfonylureas, such as glibenclamide, are antidiabetic drugs that stimulate beta-cell insulin secretion by binding to the sulfonylureas receptors (SURs) of adenosine triphosphate-sensitive potassium channels (KATP). Glibenclamide may be a
Externí odkaz:
https://doaj.org/article/9d00b87e3b3a4e23a5e68872d1a8de7e
Autor:
Chiara Barisione, Silvano Garibaldi, Anna Lisa Furfaro, Mariapaola Nitti, Daniela Palmieri, Mario Passalacqua, Anna Garuti, Daniela Verzola, Alessia Parodi, Pietro Ameri, Paola Altieri, Patrizia Fabbi, Pier Francesco Ferrar, Claudio Brunelli, Violeta Arsenescu, Manrico Balbi, Domenico Palombo, Giorgio Ghigliotti
Publikováno v:
PLoS ONE, Vol 11, Iss 2, p e0149276 (2016)
OBJECTIVE:The uremic toxin Indoxyl-3-sulphate (IS), a ligand of Aryl hydrocarbon Receptor (AhR), raises in blood during early renal dysfunction as a consequence of tubular damage, which may be present even when eGFR is normal or only moderately reduc
Externí odkaz:
https://doaj.org/article/df9b062c73184a7380123cf8e5afe9e1
Autor:
Patrizia Fabbi, Paolo Spallarossa, Silvano Garibaldi, Chiara Barisione, Marzia Mura, Paola Altieri, Barbara Rebesco, Maria Gaia Monti, Marco Canepa, Giorgio Ghigliotti, Claudio Brunelli, Pietro Ameri
Publikováno v:
PLoS ONE, Vol 10, Iss 5, p e0124643 (2015)
Insulin-like growth factor-1 (IGF-1) promotes the survival of cardiomyocytes by activating type 1 IGF receptor (IGF-1R). Within the myocardium, IGF-1 action is modulated by IGF binding protein-3 (IGFBP-3), which sequesters IGF-1 away from IGF-1R. Sin
Externí odkaz:
https://doaj.org/article/7873e7f011504a0fbd4313266562c921
Autor:
Paola Altieri, Paolo Spallarossa, Chiara Barisione, Silvano Garibaldi, Anna Garuti, Patrizia Fabbi, Giorgio Ghigliotti, Claudio Brunelli
Publikováno v:
PLoS ONE, Vol 7, Iss 9, p e46126 (2012)
Senescence and apoptosis are two distinct cellular programs that are activated in response to a variety of stresses. Low or high doses of the same stressor, i.e., the anticancer drug doxorubicin, may either induce apoptosis or senescence, respectivel
Externí odkaz:
https://doaj.org/article/7fd83d493d4a468a9b55668dd81322a2
Publikováno v:
Heart International, Vol 2, Iss 3-4 (2006)
Acute myocardial infarction (AMI) occurs when the atheromatous process prevents total blood flow through the coronary artery. It was previously thought that progressive luminal narrowing from the continued growth of smooth muscle cells (SMCs) in the
Externí odkaz:
https://doaj.org/article/cacb4c878eb44c80a521a2f441dc5ee6
Autor:
Andrea Armirotti, Paola Altieri, Roberto Murialdo, Silvia Borile, Federico Carbone, Chiara Barisione, Clarissa Ruggeri, Silvano Garibaldi, Pietro Ameri, Alberto Ballestrero, Marco Canepa, Patrizia Fabbi, Edoardo Lazzarini, Fabrizio Montecucco, Paolo Spallarossa, Claudio Brunelli
Publikováno v:
British Journal of Pharmacology. 174:3713-3726
Background and Purpose 5-fluorouracil (5FU) and its prodrug, capecitabine, can damage endothelial cells, whilst endothelial integrity is preserved by glucagon-like peptide 1 (GLP-1). Here, we studied the effect of 5FU on endothelial senescence and wh
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::85fb7a5619ee06bb3bf1bf88ad9a3add
https://doi.org/10.1111/bph.13725
https://doi.org/10.1111/bph.13725
Autor:
Patrizia Fabbi, Francesco Indiveri, Silvano Garibaldi, Giorgio Ghigliotti, Gian Paolo Bezante, Claudio Brunelli, Antonio Barsotti, Massimiliano Fedele, Domenico Risso, Manrico Balbi
Publikováno v:
Clinical Biochemistry. 44:605-611
Objectives To identify systemically detectable vascular inflammation associated to redox system unbalance, advanced oxidation protein products (AOPP), formed by HClO reaction with proteins, Thiol levels, and their ratio (AOPP/Thiol ratio) were measur
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d8bbf9fee6803faf61a91f0bf21c69f8
https://doi.org/10.1016/j.clinbiochem.2011.03.134
https://doi.org/10.1016/j.clinbiochem.2011.03.134
Autor:
Barbara Salani, Chiara Barisione, Paola Altieri, Paolo Spallarossa, Mario Passalacqua, Marzia Mura, Silvano Garibaldi, Patrizia Fabbi, Claudio Brunelli, Concetta Aloi, Giorgio Ghigliotti
Publikováno v:
Biochemical and Biophysical Research Communications. 388:523-528
Cell attachment is provided by cell-matrix and cell-cell bonds, and acts as a regulator of vascular smooth muscle cell (VSMC) survival, activity and homeostasis, as well as of VSMCs response to pathogenic stimuli. In this work we elicited an exclusiv
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::705584c7d381adb437c60bb4278d1c74
https://doi.org/10.1016/j.bbrc.2009.08.031
https://doi.org/10.1016/j.bbrc.2009.08.031
Autor:
Silvano Garibaldi, Alberto Ballestrero, Patrizia Fabbi, Valeria Manca, Antonio Barsotti, Chiara Barisione, Paola Altieri, Paolo Spallarossa, Giorgio Ghigliotti, Claudio Brunelli
Publikováno v:
Cardiovascular Research. 69:736-745
Objective: Dysregulation of myocardial metalloproteinases (MMPs) is now regarded as an early contributory mechanism for the initiation and progression of heart failure. Doxorubicin is a strongly cardiotoxic anticancer drug. This study investigates th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::337f45e9f2a1db236db535da84b34103
https://doi.org/10.1016/j.cardiores.2005.08.009
https://doi.org/10.1016/j.cardiores.2005.08.009