Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Patrick B. Reeves"'
Autor:
Finnian R. Mc Causland, João Sérgio Neves, Patrick B. Reeves, Sushrut S. Waikar, Simon Correa, Katherine Scovner Ravi, Suraj Sarvode Mothi
Publikováno v:
Hemodialysis international. International Symposium on Home HemodialysisREFERENCES. 26(1)
INTRODUCTION Intradialytic hypotension (IDH) is a common complication of hemodialysis (HD) and is associated with excess morbidity and mortality. Higher serum phosphate is associated with adverse cardiovascular outcomes in maintenance HD patients; ho
Publikováno v:
Clinical journal of the American Society of Nephrology : CJASN. 13(8)
Individuals with ESKD requiring maintenance hemodialysis face a unique hemodynamic challenge, typically on a thrice-weekly basis. In an effort to achieve some degree of euvolemia, ultrafiltration goals often involve removal of the equivalent of an en
Autor:
Marian DiFiglia, Xueyi Li, Kimberly B Kegel, Matthew P Frosch, Nicholas Masso, Kwadwo A Ansong, Jonathan Alexander, Patrick B Reeves, Hollis McClory, Jeffrey S Kimm, Ellen Sapp, Antonio Valencia
Publikováno v:
Human molecular genetics. 26(21)
A mutation in the huntingtin (Htt) gene produces mutant Htt and Huntington's disease (HD), a neurodegenerative disorder. HD patients have oxidative damage in the brain, but the causes are unclear. Compared with controls, we found brain levels of NADP
Autor:
Jonathan Alexander, Ellen Sapp, Marian DiFiglia, Nicholas Masso, Antonio Valencia, Xueyi Li, Patrick B. Reeves, Kimberly B. Kegel
Publikováno v:
NeuroReport. 23:10-15
Patients with Huntington's disease suffer severe neuronal loss and signs of oxidative damage in the brain. Previously we found that primary neurons from embryonic cortex of mice bearing the Huntington's disease mutation (140 glutamines inserted into
Autor:
Neil Aronin, Ellen Sapp, Xueyi Li, Kimberly B. Kegel, Jonathan Alexander, Kathryn Chase, Antonio Valencia, Patrick B. Reeves, Marian DiFiglia
Publikováno v:
Journal of Neuroscience Research. 88:179-190
Patients with Huntington's disease have an expanded polyglutamine tract in huntingtin and suffer severe brain atrophy and neurodegeneration. Because membrane dysfunction can occur in Huntington's disease, we addressed whether mutant huntingtin in bra
Autor:
Edith L. Pfister, Neil Aronin, Phillip D. Zamore, Ellen Sapp, Kallanthottathil G. Rajeev, Marian DiFiglia, Meghan B. Sass, Jennifer Yoder, Rajendra K. Pandey, Miguel Sena-Esteves, Dinah W.Y. Sah, Patrick B. Reeves, Muthiah Manoharan, Kathryn Chase
Publikováno v:
Proceedings of the National Academy of Sciences. 104:17204-17209
Huntington's disease (HD) is an autosomal dominant disease caused by a CAG repeat expansion in the Htt gene (1). Mutant Htt causes neuronal death, dementia, and movement dysfunction; there is no effective treatment. In an inducible transgenic mouse m
Autor:
Jonathan Alexander, Ellen Sapp, Antonio Valencia, Kimberly B. Kegel, Jeffrey S. Kimm, Hollis McClory, Matthew P. Frosch, Nicholas Masso, Kwadwo A. Ansong, Patrick B. Reeves, Marian DiFiglia, Xueyi Li
Publikováno v:
Hum Mol Genet
A mutation in the huntingtin (Htt) gene produces mutant Htt and Huntington's disease (HD), a neurodegenerative disorder. HD patients have oxidative damage in the brain, but the causes are unclear. Compared with controls, we found brain levels of NADP
Autor:
Antonio, Valencia, Patrick B, Reeves, Ellen, Sapp, Xueyi, Li, Jonathan, Alexander, Kimberly B, Kegel, Kathryn, Chase, Neil, Aronin, Marian, DiFiglia
Publikováno v:
Journal of neuroscience research. 88(1)
Patients with Huntington's disease have an expanded polyglutamine tract in huntingtin and suffer severe brain atrophy and neurodegeneration. Because membrane dysfunction can occur in Huntington's disease, we addressed whether mutant huntingtin in bra