Zobrazeno 1 - 10
of 26
pro vyhledávání: '"Patricia A. Cragg"'
Autor:
Cecilia Camargo, Luis Nicanor Pezo Lanfranco, Patricia Landa Cragg, María del Carmen Vega, Julio Phan
Publikováno v:
Boletín de Arqueología PUCP. :135-143
En el año 2018 se hallaron tres entierros de inmigrantes chinos de fines del siglo XIX y comienzos del siglo XX en el distrito limeño de Carabayllo, durante los trabajos de monitoreo arqueológico de la empresa Cálidda. En esta nota se presenta la
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e09668849a144804fee5b55f16e8fa34
https://doi.org/10.18800/boletindearqueologiapucp.202101.007
https://doi.org/10.18800/boletindearqueologiapucp.202101.007
Publikováno v:
Bulletin de l'Institut Français d'Études Andines, Vol 33, Pp 815-860 (2004)
This article documents the unwrapping of a coastal fardo dating to the Inca period. The fardo, excavated at Rinconada Alta in the Rimac Valley, is particularly rich in garments and textile accessories, and provides the opportunity for examining the s
Externí odkaz:
https://doaj.org/article/dd25f8ec814b431a8eddb2a5bc186f78
Autor:
Carol T. Bussey, Gillian Hughes, Andrew Bahn, Patricia A. Cragg, Regis R. Lamberts, Ingrid C. Fomison-Nurse, Rosalind F. Cook
Publikováno v:
Experimental Physiology. 104:808-818
NEW FINDINGS What is the central question of this study? Are there specific contributions of β1 - and β2 -adrenoceptor subtypes to the impaired β-adrenoceptor responsiveness of the type 2 diabetic heart? What is the main finding and its importance
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7ef565c05c3ae6f8e47859b9ce62b985
https://doi.org/10.1113/ep087437
https://doi.org/10.1113/ep087437
Publikováno v:
Experimental Physiology. 102:911-923
New Findings What is the central question of the study? The sympathetic system regulates heart rate via β-adrenoceptors; this is impaired during diabetes. However, the specific β-adrenoceptor subtype contributions in heart rate regulation in diabet
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a38498ea6cc65f3ea57a1f39d6f5959b
https://doi.org/10.1113/ep086293
https://doi.org/10.1113/ep086293
Autor:
Rosalind F, Cook, Carol T, Bussey, Ingrid C, Fomison-Nurse, Gillian, Hughes, Andrew, Bahn, Patricia A, Cragg, Regis R, Lamberts
Publikováno v:
Experimental physiology. 104(6)
What is the central question of this study? Are there specific contributions of βImpaired β-adrenoceptor (β-AR) responsiveness causes cardiac vulnerability in patients with type 2 diabetes, but the independent contributions of β
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c6e116e137f016d074ed45013b174233
https://pubmed.ncbi.nlm.nih.gov/30889297
https://pubmed.ncbi.nlm.nih.gov/30889297
Publikováno v:
Experimental physiology. 102(8)
What is the central question of the study? The sympathetic system regulates heart rate via β-adrenoceptors; this is impaired during diabetes. However, the specific β-adrenoceptor subtype contributions in heart rate regulation in diabetes in vivo ar
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::dddee358288af4cede5354cc296243d1
https://pubmed.ncbi.nlm.nih.gov/28543947
https://pubmed.ncbi.nlm.nih.gov/28543947
Autor:
Daryl O. Schwenke, Kenji Kangawa, Mikiyasu Shirai, Takeshi Horio, Patricia A. Cragg, Ichiro Kishimoto, Takeshi Tokudome
Publikováno v:
Endocrinology. 153:2436-2443
Acute myocardial infarction (MI) increases sympathetic nerve activity (SNA) to the heart, which exacerbates chronic cardiac deterioration. The hormone ghrelin, if administered soon after an MI, prevents the increase in cardiac SNA and improves early
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5ee352d311c3de9f49f6c7bb97bedfbb
https://doi.org/10.1210/en.2011-2057
https://doi.org/10.1210/en.2011-2057
Publikováno v:
Acta Physiologica. 200:279-287
Aim: Ghrelin has been implicated as a modulator of numerous physiologicalpathways. To date, there have not been any studies describing the role ofghrelin in modulating the chemoreflex control of pulmonary ventilation. Yetthe respiratory system impac
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ecfd80383cc6e1b659d3a2bbd04854de
https://doi.org/10.1111/j.1748-1716.2010.02142.x
https://doi.org/10.1111/j.1748-1716.2010.02142.x
Autor:
Daryl O. Schwenke, Takeshi Tokudome, Ichiro Kishimoto, Patricia A. Cragg, Mikiyasu Shirai, Takeshi Horio, Kenji Kangawa
Publikováno v:
Endocrinology. 149:5172-5176
Acute myocardial infarction (MI) initiates an increase in cardiac sympathetic nerve activity (CSNA), which ultimately exacerbates chronic cardiac dysfunction. Ghrelin (Ghr), a GH-releasing peptide, is an effective treatment for improving cardiac func
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c1c96e93a671bd2864c3a8d0219e9ce3
https://doi.org/10.1210/en.2008-0472
https://doi.org/10.1210/en.2008-0472
Publikováno v:
Acta physiologica (Oxford, England). 214(3)
Aim The peripheral chemoreflex is augmented in heart failure, and it may contribute to sympathoexcitation. This study aimed to investigate both the chemoreflex and the cardiac sympathetic nerve activity in the acute-stage post-myocardial infarction.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8eb4f3f50be47cb7a378145cecaa6ac1
https://pubmed.ncbi.nlm.nih.gov/25980319
https://pubmed.ncbi.nlm.nih.gov/25980319