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pro vyhledávání: '"Parkinson's disease1"'
Autor:
Tenghong Lian, Weijiao Zhang, Danning Li, Peng Guo, Mingyue He, Yanan Zhang, Jinghui Li, Huiying Guan, Wenjing Zhang, Dongmei Luo, Weijia Zhang, Xiaomin Wang, Wei Zhang
Publikováno v:
BMC Geriatrics, Vol 24, Iss 1, Pp 1-12 (2024)
Abstract Objective This study was performed to explore the differences in the clinical characteristics and oxidative stress indicators, inflammatory factors, and pathological proteins in serum between Parkinson’s disease (PD) with anxiety (PD-A) an
Externí odkaz:
https://doaj.org/article/11b65d8f87624130b224453db034d96e
Akademický článek
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Autor:
Alexandre N. Rcom-H’cheo-Gauthier, Samantha L. Osborne, Adrian C.B. Meedneniya, Dean Louis Pountney
Publikováno v:
Frontiers in Neuroscience, Vol 10 (2016)
Aggregation of the pre-synaptic protein, α-synuclein (α-syn), is the key aetiological factor in Parkinson’s disease (PD) and other alpha-synucleinopathies, such as multiple system atrophy and Dementia with Lewy bodies. Various triggers for pathol
Externí odkaz:
https://doaj.org/article/351dd0240ea3480c9c5cbe71f340332a
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 10 (2016)
Transplantation of fetal human ventral mesencephalic dopaminergic neurons into the striatum is a promising strategy to compensate for the characteristic dopamine deficit observed in Parkinson’s disease. This therapeutic approach, however, is curren
Externí odkaz:
https://doaj.org/article/f297d80358564e7696e97d8920a49ce7
Publikováno v:
Frontiers in Cellular Neuroscience
Frontiers in Cellular Neuroscience, Vol 10 (2016)
Front. Cell. Neurosci
Seiler, Stefanie; Di Santo, Stefano; Widmer, Hans Rudolf (2016). Nogo-A Neutralization Improves Graft Function in a Rat Model of Parkinson's Disease. Frontiers in cellular neuroscience, 10, p. 87. Frontiers Research Foundation 10.3389/fncel.2016.00087
Frontiers in Cellular Neuroscience, Vol 10 (2016)
Front. Cell. Neurosci
Seiler, Stefanie; Di Santo, Stefano; Widmer, Hans Rudolf (2016). Nogo-A Neutralization Improves Graft Function in a Rat Model of Parkinson's Disease. Frontiers in cellular neuroscience, 10, p. 87. Frontiers Research Foundation 10.3389/fncel.2016.00087
Transplantation of fetal human ventral mesencephalic dopaminergic neurons into the striatum is a promising strategy to compensate for the characteristic dopamine deficit observed in Parkinson’s disease. This therapeutic approach, however, is curren