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of 7
pro vyhledávání: '"Parisa Iloun"'
Autor:
Felix Michaud, Ruggiero Francavilla, Dimitry Topolnik, Parisa Iloun, Suhel Tamboli, Frederic Calon, Lisa Topolnik
Publikováno v:
eLife, Vol 13 (2024)
Alzheimer’s disease (AD) leads to progressive memory decline, and alterations in hippocampal function are among the earliest pathological features observed in human and animal studies. GABAergic interneurons (INs) within the hippocampus coordinate
Externí odkaz:
https://doaj.org/article/d9e5304ffee34b25896f700dee2e3aff
Autor:
Abolhassan Ahmadiani, Sevda Gheibi, Parisa Iloun, Rasoul Ghasemi, Etrat Hooshmandi, Khosrow Kashfi
Publikováno v:
Cellular and Molecular Neurobiology. 41:1497-1507
Alzheimer's disease (AD) is characterized with increased formation of amyloid-β (Aβ) in the brain. Aβ peptide toxicity is associated with disturbances of several intracellular signaling pathways such as mitogen activated protein kinases (MAPKs). T
Autor:
Étienne Gervais, Parisa Iloun, Ekaterina Martianova, Ana Claudia Gonçalves Bessa, Serge Rivest, Lisa Topolnik
Publikováno v:
The Journal of comparative neurologyREFERENCES. 530(9)
Microglia can interact with glutamatergic neurons and, through control of synaptic elements, regulate their physiological function. Much less is known about the partnership between microglia and GABAergic inhibitory interneurons. Here, we compared th
Publikováno v:
Molecular Biology Reports. 46:489-496
β-Amyloid peptide (Aβ), the major element of senile plaques in Alzheimer’s disease (AD), has been found to accumulate in brain regions critical for memory and cognition. Deposits of Aβ trigger neurotoxic events which lead to neural apoptotic dea
Autor:
Parisa, Iloun, Etrat, Hooshmandi, Sevda, Gheibi, Khosrow, Kashfi, Rasoul, Ghasemi, Abolhassan, Ahmadiani
Publikováno v:
Cellular and molecular neurobiology. 41(7)
Alzheimer's disease (AD) is characterized with increased formation of amyloid-β (Aβ) in the brain. Aβ peptide toxicity is associated with disturbances of several intracellular signaling pathways such as mitogen activated protein kinases (MAPKs). T
Publikováno v:
Molecular biology reports. 46(1)
β-Amyloid peptide (Aβ), the major element of senile plaques in Alzheimer's disease (AD), has been found to accumulate in brain regions critical for memory and cognition. Deposits of Aβ trigger neurotoxic events which lead to neural apoptotic death
Despite the consensus that neuro-inflammation and insulin resistance (IR) are two hallmarks of Alzheimer disease (AD), the molecular mechanisms responsible for the development of IR remain uncharacterized. MAPKs are signaling molecules that are impli
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6141bead001be545d52eeee16b795bdb