A peptide containing residues 26-44 of tau protein impairs mithocondrial oxidative phosphorylation acting

Autor: Atlante A a, Amadoro G b, Bobba A a, de Bari L a, Corsetti V b,c, Pappalardo G d, Marra E a, Calissano P b,c, Passarella Se

Publikováno v: Biochimica et biophysica acta. Bioenergetics 1777 (2008): 1289–1300. doi:10.1016/j.bbabio.2008.07.004
info:cnr-pdr/source/autori:Atlante A a; Amadoro G b; Bobba A a; de Bari L a; Corsetti V b,c; Pappalardo G d; Marra E a; Calissano P b,c; Passarella Se/titolo:A peptide containing residues 26-44 of tau protein impairs mithocondrial oxidative phosphorylation acting .../doi:10.1016%2Fj.bbabio.2008.07.004/rivista:Biochimica et biophysica acta. Bioenergetics/anno:2008/pagina_da:1289/pagina_a:1300/intervallo_pagine:1289–1300/volume:1777

Having confirmed that adenovirus-mediated overexpression of NH(2)-tau fragment lacking the first 25 aminoacids evokes a potent neurotoxic effect, sustained by protracted stimulation of NMDA receptors, in primary neuronal cultures we investigated whet

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