Zobrazeno 1 - 10
of 115
pro vyhledávání: '"Paola Altieri"'
Autor:
Emma L. Robinson, Pietro Ameri, Leen Delrue, Marc Vanderheyden, Jozef Bartunek, Paola Altieri, Stephane Heymans, Ward A. Heggermont
Publikováno v:
Frontiers in Cardiovascular Medicine, Vol 10 (2023)
BackgroundAnthracycline-induced cardiotoxicity is a well-known serious clinical entity. However, detailed mechanistic insights on how short-term administration leads to late and long-lasting cardiotoxicity, are still largely undiscovered. We hypothes
Externí odkaz:
https://doaj.org/article/e102d1878e214670940c50ef20f1b115
Autor:
Gemma Marcucci, Paola Altieri, Salvatore Benvenga, Marta Bondanelli, Valentina Camozzi, Filomena Cetani, Luisella Cianferotti, Mirko Duradoni, Caterina Fossi, Ettore degli Uberti, Fausto Famà, Giovanna Mantovani, Claudio Marcocci, Laura Masi, Uberto Pagotto, Andrea Palermo, Simone Parri, Rosaria Maddalena Ruggeri, Maria Chiara Zatelli, Maria Luisa Brandi, On behalf of “Hypoparathyroidism Working Group” affiliated to the Italian Society of Endocrinology (S.I.E.)
Publikováno v:
Orphanet Journal of Rare Diseases, Vol 16, Iss 1, Pp 1-11 (2021)
Abstract Background Hypoparathyroidism (HypoPT) or pseudo-hypoparathyroidism (pseudo-HypoPT) during pregnancy may cause maternal and fetal/neonatal complications. In this regard, only a few case reports or case series of pregnant or lactating women h
Externí odkaz:
https://doaj.org/article/8e962859912f412abf830c3b95403a25
Autor:
Chiara Brullo, Eleonora Russo, Silvano Garibaldi, Paola Altieri, Pietro Ameri, Silvia Ravera, Maria Grazia Signorello
Publikováno v:
Antioxidants, Vol 12, Iss 2, p 216 (2023)
In the effort to obtain multitarget compound interfering with inflammation, oxidative stress, and tumorigenesis, we synthesized a small library of pyrazole compounds, selecting 4a, 4f, and 4g as the most noteworthy being IC50 against platelet ROS pro
Externí odkaz:
https://doaj.org/article/b3f40636ad0048e58e1db1923554c88a
Autor:
Paola Altieri, Maria Bertolotto, Patrizia Fabbi, Elena Sportelli, Manrico Balbi, Francesco Santini, Claudio Brunelli, Marco Canepa, Fabrizio Montecucco, Pietro Ameri
Publikováno v:
Data in Brief, Vol 19, Iss , Pp 925-931 (2018)
The data presented here are related to the research paper entitled “Thrombin induces protease-activated receptor 1 signaling and activation of human atrial fibroblasts and dabigatran prevents these effects” (Altieri et al., 2018) [1]. Data show t
Externí odkaz:
https://doaj.org/article/2748a3cfd678496093a7e8237118676a
Autor:
Guido Zavatta, Paola Altieri, Giulia Vandi, Valentina Vicennati, Uberto Pagotto, Fabio Vescini
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 23, p 12975 (2021)
The advent of new insights into phosphate metabolism must urge the endocrinologist to rethink the pathophysiology of widespread disorders, such as primary hyperparathyroidism, and also of rarer endocrine metabolic bone diseases, such as hypoparathyro
Externí odkaz:
https://doaj.org/article/59be5a3bbce14cd1a69c9d524f79ffef
Autor:
Federico Villa, Silvia Bruno, Ambra Costa, Mingchuan Li, Michele Russo, James Cimino, Paola Altieri, Clarissa Ruggeri, Cansu Gorgun, Pierangela De Biasio, Dario Paladini, Domenico Coviello, Rodolfo Quarto, Pietro Ameri, Alessandra Ghigo, Silvia Ravera, Roberta Tasso, Sveva Bollini
Publikováno v:
Cancers, Vol 13, Iss 15, p 3729 (2021)
Cardiovascular side effects are major shortcomings of cancer treatments causing cardiotoxicity and late-onset cardiomyopathy. While doxorubicin (Dox) has been reported as an effective chemotherapy agent, unspecific impairment in cardiomyocyte mitocho
Externí odkaz:
https://doaj.org/article/b0549992d8d6404a8101784f818e8057
Autor:
Chiara Barisione, Silvano Garibaldi, Anna Lisa Furfaro, Mariapaola Nitti, Daniela Palmieri, Mario Passalacqua, Anna Garuti, Daniela Verzola, Alessia Parodi, Pietro Ameri, Paola Altieri, Patrizia Fabbi, Pier Francesco Ferrar, Claudio Brunelli, Violeta Arsenescu, Manrico Balbi, Domenico Palombo, Giorgio Ghigliotti
Publikováno v:
PLoS ONE, Vol 11, Iss 2, p e0149276 (2016)
OBJECTIVE:The uremic toxin Indoxyl-3-sulphate (IS), a ligand of Aryl hydrocarbon Receptor (AhR), raises in blood during early renal dysfunction as a consequence of tubular damage, which may be present even when eGFR is normal or only moderately reduc
Externí odkaz:
https://doaj.org/article/df9b062c73184a7380123cf8e5afe9e1
Autor:
Paola Altieri, Chiara Barisione, Edoardo Lazzarini, Anna Garuti, Gian Paolo Bezante, Marco Canepa, Paolo Spallarossa, Carlo Gabriele Tocchetti, Sveva Bollini, Claudio Brunelli, Pietro Ameri
Publikováno v:
Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, Vol 5, Iss 1 (2016)
BackgroundChronic cardiotoxicity is less common in male than in female patients receiving doxorubicin and other anthracyclines at puberty and adolescence. We hypothesized that this sex difference might be secondary to distinct activities of sex hormo
Externí odkaz:
https://doaj.org/article/67d0fb22b0b8492b8204b08f6a22c4bb
Autor:
Patrizia Fabbi, Paolo Spallarossa, Silvano Garibaldi, Chiara Barisione, Marzia Mura, Paola Altieri, Barbara Rebesco, Maria Gaia Monti, Marco Canepa, Giorgio Ghigliotti, Claudio Brunelli, Pietro Ameri
Publikováno v:
PLoS ONE, Vol 10, Iss 5, p e0124643 (2015)
Insulin-like growth factor-1 (IGF-1) promotes the survival of cardiomyocytes by activating type 1 IGF receptor (IGF-1R). Within the myocardium, IGF-1 action is modulated by IGF binding protein-3 (IGFBP-3), which sequesters IGF-1 away from IGF-1R. Sin
Externí odkaz:
https://doaj.org/article/7873e7f011504a0fbd4313266562c921
Autor:
Paola Altieri, Paolo Spallarossa, Chiara Barisione, Silvano Garibaldi, Anna Garuti, Patrizia Fabbi, Giorgio Ghigliotti, Claudio Brunelli
Publikováno v:
PLoS ONE, Vol 7, Iss 9, p e46126 (2012)
Senescence and apoptosis are two distinct cellular programs that are activated in response to a variety of stresses. Low or high doses of the same stressor, i.e., the anticancer drug doxorubicin, may either induce apoptosis or senescence, respectivel
Externí odkaz:
https://doaj.org/article/7fd83d493d4a468a9b55668dd81322a2