Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Panayota Kolypetri"'
Autor:
Panayota Kolypetri, Shirong Liu, Laura M. Cox, Mai Fujiwara, Radhika Raheja, Dvora Ghitza, Anya Song, Dominique Daatselaar, Valerie Willocq, Howard L. Weiner
Publikováno v:
iScience, Vol 24, Iss 4, Pp 102356- (2021)
Summary: Splenic Ly6Chigh monocytes are innate immune cells involved in the regulation of central nervous system-related diseases. Recent studies have reported the shaping of peripheral immune responses by the gut microbiome via mostly unexplored pat
Externí odkaz:
https://doaj.org/article/bd95a85eaa3743389ae962d12f675f6a
Autor:
Paris E. Georghiou, Vassiliki Magafa, Shofiur Rahman, Paul Cordopatis, Karen A. Carayanniotis, Panayota Kolypetri, George Carayanniotis
Publikováno v:
The Journal of Immunology. 193:96-101
Enhanced iodide ingestion is known to accelerate the incidence and severity of spontaneous autoimmune thyroiditis [iodide-accelerated spontaneous autoimmune thyroiditis (ISAT)] in NOD.H2h4 mice. CD4+ cells are required for the development and mainten
Publikováno v:
Thyroid. 24:1170-1178
Enhanced iodide intake in NOD.H2(h4) mice accelerates the incidence and severity of spontaneous autoimmune thyroiditis (SAT) via an unknown mechanism. A plausible hypothesis is that iodide-induced apoptosis of thyrocytes can create imbalances in anti
Publikováno v:
The Journal of Immunology. 190:1466-1471
Experimental autoimmune thyroiditis, induced in mice after challenge with thyroglobulin (Tg), is known to be under the genetic control of the H2Ak locus. Because cathepsins are known to influence proteolytic processing of Tg in vivo, we examined in t
Publikováno v:
HORMONES. 9:151-160
OBJECTIVE: In NOD.H2h4 mice, high dietary iodine intake has been known to cause Iodineaccelerated Spontaneous Autoimmune Thyroiditis (ISAT) via an unknown mechanism. The aim of the study was to examine whether the NOD.H2h4 genetic background predispo
Publikováno v:
International reviews of immunology. 34(6)
In the field of autoimmune thyroiditis, NOD.H2(h4) mice have attracted significant and increasing attention since they not only develop spontaneous disease but they present thyroiditis with accelerated incidence and severity if they ingest iodide thr
Publikováno v:
International reviews of immunology.
In the field of autoimmune thyroiditis, NOD.H2
Publikováno v:
Clinical and experimental immunology. 176(3)
Summary Recent studies have shown that high salt (HS) intake exacerbates experimental autoimmune encephalomyelitis and have raised the possibility that a HS diet may comprise a risk factor for autoimmune diseases in general. In this report, we have e
Publikováno v:
The Journal of Immunology. 190:172.1-172.1
Enhanced iodide intake in NOD.H2h4 mice accelerates the onset and severity of spontaneous autoimmune thyroiditis (ISAT) via an unknown mechanism. In this study, we have tested two hypotheses: 1) that NOD.H2h4 thyrocytes exhibit increased susceptibili