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pro vyhledávání: '"Pablo SÁNCHEZ MARTÍN"'
Autor:
Pablo SÁNCHEZ MARTÍN
Publikováno v:
Ars Iuris Salmanticensis, Vol 1, Iss 1, Pp 13-19 (2013)
Externí odkaz:
https://doaj.org/article/9e925ddf601c45c79ad91eb6c1f04ee7
Autor:
Pablo Sánchez-Martín, Claudine Kraft
Publikováno v:
Autophagy Reports, Vol 2, Iss 1 (2023)
YKT6 is a SNARE (Soluble N-ethylmaleimide-Sensitive Fusion Protein Attachment Protein Receptor) protein governing membrane fusion events of several cellular organelles. In autophagy, YKT6 is involved in early phagophore formation as well as directly
Externí odkaz:
https://doaj.org/article/8131d651e60a4dfdbaf1e4b9897c66ff
Autor:
Pablo Sánchez-Martín, Franziska Kriegenburg, Ludovico Alves, Julius Adam, Jana Elsaesser, Riccardo Babic, Hector Mancilla, Mariya Licheva, Georg Tascher, Christian Münch, Stefan Eimer, Claudine Kraft
Publikováno v:
Journal of Cell Science. 136
Autophagy is a catabolic process during which cytosolic material is enwrapped in a newly formed double-membrane structure called the autophagosome, and subsequently targeted for degradation in the lytic compartment of the cell. The fusion of autophag
Publikováno v:
Information Sciences. 637:118928
Publikováno v:
Nature Communications
Autor:
Masaaki Komatsu, Pablo Sánchez-Martín
Publikováno v:
The Journal of Cell Biology
Sánchez-Martín and Komatsu preview work from Itakura and colleagues that describes a mechanism that mediates degradation of misfolded extracellular proteins.
The mechanisms of quality control for extracellular proteins are still poorly underst
The mechanisms of quality control for extracellular proteins are still poorly underst
Autor:
Masaaki Komatsu, Masato Koike, Pablo Sánchez-Martín, Satoshi Waguri, Yu-shin Sou, Shun Kageyama
Publikováno v:
EMBO Rep
p62/SQSTM1 is a multivalent protein that has the ability to cause liquid–liquid phase separation and serves as a receptor protein that participates in cargo isolation during selective autophagy. This protein is also involved in the non‐canonical
Publikováno v:
Digital.CSIC. Repositorio Institucional del CSIC
instname
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH
r-CIPF. Repositorio Institucional Producción Científica del Centro de Investigación Principe Felipe (CIPF)
Biochim Biophys Acta Mol Cell Res
r-CIPF: Repositorio Institucional Producción Científica del Centro de Investigación Principe Felipe (CIPF)
Centro de Investigación Principe Felipe (CIPF)
instname
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH
r-CIPF. Repositorio Institucional Producción Científica del Centro de Investigación Principe Felipe (CIPF)
Biochim Biophys Acta Mol Cell Res
r-CIPF: Repositorio Institucional Producción Científica del Centro de Investigación Principe Felipe (CIPF)
Centro de Investigación Principe Felipe (CIPF)
15 páginas, 10 figuras. The Transparency document associated this article can be found, in online version. Appendix A. Supplementary data: https://doi.org/10.1016/j.bbamcr.2019.118613.
Lafora progressive myoclonus epilepsy is a fatal rare neuro
Lafora progressive myoclonus epilepsy is a fatal rare neuro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c6e0e96b95e57801eda260ffc9580be4
http://hdl.handle.net/10261/202051
http://hdl.handle.net/10261/202051
Publikováno v:
Biochimica et Biophysica Acta (BBA)-Molecular Cell Research
Biochimica et Biophysica Acta. Molecular Cell Research
Biochimica et Biophysica Acta. Molecular Cell Research
Autophagy is a degradative pathway during which autophagosomes are formed that enwrap cytosolic material destined for turnover within the lytic compartment. Autophagosome biogenesis requires controlled lipid and membrane rearrangements to allow the f
p62/SQSTM1 is a multivalent protein that has an ability to cause a liquid-liquid phase separation and serves as a receptor protein that participates in cargo isolation during selective autophagy. This protein is also involved in the non-canonical act
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ef1bca3706a299eb552c49e12e990303
https://doi.org/10.1101/709105
https://doi.org/10.1101/709105