Zobrazeno 1 - 10
of 211
pro vyhledávání: '"P.C. van Rijen"'
Autor:
E. Aronica, K. Boer, K.J. Doorn, E. Zurolo, W.G.M. Spliet, P.C. van Rijen, J.C. Baayen, J.A. Gorter, A. Jeromin
Publikováno v:
Neurobiology of Disease, Vol 36, Iss 1, Pp 81-95 (2009)
An increasing number of observations suggest an important role for voltage-gated potassium (Kv) channels in epilepsy. We studied the cell-specific distribution of Kv4.2, phosphorylated (p) Kv4.2 and the Kv4.2 interacting protein NCS-1 using immunocyt
Externí odkaz:
https://doaj.org/article/a4ae868312e64252a084b09150436d34
Autor:
E. Aronica, K. Boer, E.A. van Vliet, S. Redeker, J.C. Baayen, W.G.M. Spliet, P.C. van Rijen, D. Troost, F.H. Lopes da Silva, W.J. Wadman, J.A. Gorter
Publikováno v:
Neurobiology of Disease, Vol 26, Iss 3, Pp 497-511 (2007)
We investigated the involvement of the complement cascade during epileptogenesis in a rat model of temporal lobe epilepsy (TLE), and in the chronic epileptic phase in both experimental as well as human TLE. Previous rat gene expression analysis using
Externí odkaz:
https://doaj.org/article/7d7a593e04904c54b77c3ddcff98c96b
Autor:
T. Ravizza, K. Boer, S. Redeker, W.G.M. Spliet, P.C. van Rijen, D. Troost, A. Vezzani, E. Aronica
Publikováno v:
Neurobiology of Disease, Vol 24, Iss 1, Pp 128-143 (2006)
Focal cortical dysplasia (FCD) and glioneuronal tumors (GNT) are recognized causes of chronic intractable epilepsy. The cellular mechanism(s) underlying their epileptogenicity remain largely unknown. Compelling evidence in experimental models of seiz
Externí odkaz:
https://doaj.org/article/1b277f85b7c548e6a8abc12367736c41
Akademický článek
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Autor:
null A. Korotkov, null N.S. Sim, null M.J. Luinenburg, null J.J. Anink, null J. van Scheppingen, null T.S. Zimmer, null A. Bongaarts, null D.W.M. Broekaart, null C. Mijnsbergen, null F.E. Jansen, null W. Van Hecke, null W.G.M. Spliet, null P.C. van Rijen, null M. Feucht, null J.A. Hainfellner, null P. Krsek, null J. Zamecnik, null P.B. Crino, null K. Kotulska, null L. Lagae, null A.C. Jansen, null D.J. Kwiatkowski, null S. Jozwiak, null P. Curatolo, null A. Mühlebner, null J.H. Lee, null J.D. Mills, null E.A. van Vliet, null E. Aronica
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::35be2f51daaffec1f650a003b9770f59
https://doi.org/10.1111/nan.12717/v2/response1
https://doi.org/10.1111/nan.12717/v2/response1
Autor:
G. Ciriminna, James D. Mills, Nicholas Rensing, Eleonora Aronica, Wim G.M. Spliet, Andrea Arena, Anatoly Korotkov, Johannes C. Baayen, Sander Idema, Till S. Zimmer, E.A. van Vliet, W. Van Hecke, Jasper J. Anink, Floor E. Jansen, Michael Wong, P.C. van Rijen
Publikováno v:
Zimmer, T S, Ciriminna, G, Arena, A, Anink, J J, Korotkov, A, Jansen, F E, van Hecke, W, Spliet, W G, van Rijen, P C, Baayen, J C, Idema, S, Rensing, N R, Wong, M, Mills, J D, van Vliet, E A & Aronica, E 2020, ' Chronic activation of anti-oxidant pathways and iron accumulation in epileptogenic malformations ', Neuropathology and Applied Neurobiology, vol. 46, no. 6, pp. 546-563 . https://doi.org/10.1111/nan.12596
Neuropathology and Applied Neurobiology
Neuropathology and Applied Neurobiology, 46(6), 546-563. Wiley-Blackwell
Neuropathology and applied neurobiology, 46(6), 546-563. Wiley-Blackwell
Neuropathology and Applied Neurobiology
Neuropathology and Applied Neurobiology, 46(6), 546-563. Wiley-Blackwell
Neuropathology and applied neurobiology, 46(6), 546-563. Wiley-Blackwell
AIMS: Oxidative stress is evident in resected epileptogenic brain tissue of patients with developmental brain malformations related to mammalian target of rapamycin activation: tuberous sclerosis complex (TSC) and focal cortical dysplasia type IIb (F
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ad51eabd472603fc51128c93fccd4ec0
https://pubmed.ncbi.nlm.nih.gov/32155661
https://pubmed.ncbi.nlm.nih.gov/32155661
Autor:
Eleonora Aronica, Jasper J. Anink, Laura Wierts, E.A. van Vliet, P.C. van Rijen, H.E. de Vries, W. W. Kamphuis, J. van Scheppingen, Wim G.M. Spliet, Diede W. M. Broekaart, B. van het Hof, Floor E. Jansen
Publikováno v:
Neuropathology and Applied Neurobiology
Neuropathology and Applied Neurobiology, 46(2), 142-159. Wiley-Blackwell
Broekaart, D W M, van Scheppingen, J, Anink, J J, Wierts, L, van het Hof, B, Jansen, F E, Spliet, W G, van Rijen, P C, Kamphuis, W W, de Vries, H E, Aronica, E & van Vliet, E A 2020, ' Increased matrix metalloproteinases expression in tuberous sclerosis complex: modulation by microRNA 146a and 147b in vitro ', Neuropathology and Applied Neurobiology, vol. 46, no. 2, pp. 142-159 . https://doi.org/10.1111/nan.12572
Neuropathology and applied neurobiology, 46(2), 142-159. Wiley-Blackwell
Neuropathology and Applied Neurobiology, 46(2), 142-159. Wiley-Blackwell
Broekaart, D W M, van Scheppingen, J, Anink, J J, Wierts, L, van het Hof, B, Jansen, F E, Spliet, W G, van Rijen, P C, Kamphuis, W W, de Vries, H E, Aronica, E & van Vliet, E A 2020, ' Increased matrix metalloproteinases expression in tuberous sclerosis complex: modulation by microRNA 146a and 147b in vitro ', Neuropathology and Applied Neurobiology, vol. 46, no. 2, pp. 142-159 . https://doi.org/10.1111/nan.12572
Neuropathology and applied neurobiology, 46(2), 142-159. Wiley-Blackwell
AIM Matrix metalloproteinases (MMPs) and their endogenous tissue inhibitors (TIMPs) control proteolysis within the extracellular matrix (ECM) of the brain. Dysfunction of this enzymatic system due to brain inflammation can disrupt the blood-brain bar
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4016a114c367b0de118ad7740fe0df3a
https://pubmed.ncbi.nlm.nih.gov/31183875
https://pubmed.ncbi.nlm.nih.gov/31183875
Autor:
Cyrille H. Ferrier, Tim J. Veersema, P.C. van Rijen, Eleonora Aronica, Avanita S. Prabowo, Wim G.M. Spliet, Jasper J. Anink, Maria Thom, Anand Iyer, A. Y. N. Schouten-van Meeteren
Publikováno v:
Neuropathology and Applied Neurobiology. 41:e1-e15
Aims Recent evidence supports the activation of mechanisms underlying cellular ageing and neurodegeneration in developmental lesions associated with epilepsy. The present study examined the ongoing cell injury and vulnerability to neuronal degenerati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a245e1d4d1bbfbcff058273a81714dea
https://doi.org/10.1111/nan.12143
https://doi.org/10.1111/nan.12143
Autor:
Johannes A. Hainfellner, P.C. van Rijen, Martha Feucht, Diede W. M. Broekaart, J. van Scheppingen, Eleonora Aronica, E.A. van Vliet, Thomas Czech, Theresa Scholl, Angelika Mühlebner, Jasper J. Anink, Wim G.M. Spliet, Mark R. J. Zuidberg
Publikováno v:
Journal of neuroinflammation, 13(1). BioMed Central
Journal of Neuroinflammation
Journal of Neuroinflammation [E], 13(1). BioMed Central
Journal of neuroinflammation, 13:202. BioMed Central
Journal of Neuroinflammation
Journal of Neuroinflammation [E], 13(1). BioMed Central
Journal of neuroinflammation, 13:202. BioMed Central
Background The proteasome is a multisubunit enzyme complex involved in protein degradation, which is essential for many cellular processes. During inflammation, the constitutive subunits are replaced by their inducible counterparts, resulting in the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3206a53e35d2d18d71d299f638ebcc26
https://doi.org/10.1186/s12974-016-0662-z
https://doi.org/10.1186/s12974-016-0662-z
Autor:
P.C. van Rijen, O. van Nieuwenhuizen, Peter H. Gosselaar, G.A.P. de Kort, M.J. van Schooneveld, Kim Boshuisen, K.P.J. Braun, Frans S. S. Leijten
Publikováno v:
Neurology. 75:1623-1630
Objective: To explore whether EEG and MRI abnormalities in the “healthy” hemisphere influence seizure and cognitive outcome after functional hemispherectomy. Methods: This is a retrospective consecutive cohort study of 43 children who underwent f
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f95e3d89afa098094cd9ef3b4e446bb2
https://doi.org/10.1212/wnl.0b013e3181fb4400
https://doi.org/10.1212/wnl.0b013e3181fb4400